scholarly journals Cholesterol Disrupts H 2 S‐Mediated Vasodilation in Large Arteries

2021 ◽  
Vol 35 (S1) ◽  
Author(s):  
Perenkita Mendiola ◽  
Jay Naik ◽  
Laura Gonzalez Bosc ◽  
Nancy Kanagy
Keyword(s):  
Large Arteries

STRUCTURAL AND FUNCTIONAL VASCULAR CHANGES IN HIGH NORMAL ARTERIAL PRESSURE

2020 ◽  
Vol 23 (1) ◽  
pp. 7-11
Author(s):  
P. Nikolov
Keyword(s):  
Arterial Pressure ◽  
Pulse Wave Velocity ◽  
Wave Velocity ◽  
Pulse Pressure ◽  
Pulse Wave ◽  
Augmentation Index ◽  
Aortic Pressure ◽  
Large Arteries ◽  
Central Aortic Pressure ◽  
Significant Difference

The PURPUSE of the present study is changes in function and structure of large arteries in individuals with High Normal Arterial Pressure (HNAP) to be established. MATERIAL and METHODS: Structural and functional changes in the large arteries were investigated in 80 individuals with HNAP and in 45 with optimal arterial pressure (OAP). In terms of arterial stiffness, pulse wave velocity (PWV), augmentation index (AI), central aortic pressure (CAP), pulse pressure (PP) were followed up in HNAP group. Intima media thickness (IMT), flow-induced vasodilatation (FMD), ankle-brachial index (ABI) were also studied. RESULTS: Significantly increased values of pulse wave velocity, augmentation index, central aortic pressure, pulse pressure are reported in the HNAP group. In terms of IMT and ABI, being in the reference interval, there is no significant difference between HNAP and OAP groups. The calculated cardiovascular risk (CVR) in both groups is low. CONCLUSION: Significantly higher values of pulse wave velocity, augmentation index, central aortic pressure and pulse pressure in the HNAP group are reported.

BmxTyrosine Kinase Is Specifically Expressed in the Endocardium and the Endothelium of Large Arteries

Circulation ◽  
1997 ◽  
Vol 96 (6) ◽  
pp. 1729-1732 ◽  
Author(s):  
Niklas Ekman ◽  
Athina Lymboussaki ◽  
Imre Västrik ◽  
Krista Sarvas ◽  
Arja Kaipainen ◽  
...  
Keyword(s):  
Large Arteries

THE ELASTICITY OF THE LARGE ARTERIES IN HYPERTENSION

1952 ◽  
Vol 30 (2) ◽  
pp. 125-129
Author(s):  
J. P. Adamson ◽  
J. Doupe
Keyword(s):  
Blood Pressure ◽  
Pulse Wave Velocity ◽  
Wave Velocity ◽  
Pulse Wave ◽  
Diastolic Pressure ◽  
Arterial Elasticity ◽  
Large Arteries ◽  
Wave Velocities

Intra-arterial pressures and pulse wave velocities were measured in 18 subjects whose auscultatory diastolic pressures ranged from 45 to 120 mm. Hg. Various methods were used to lower the blood pressure in the hypertensive and to raise it in nonhypertensive subjects so that pulse wave velocities might be compared in all subjects at a common diastolic pressure. The pulse wave velocities were calculated for a diastolic pressure of 80 mm. Hg. No significant differences were found between hypertensive and nonhypertensive subjects. It was concluded that a defect of arterial elasticity as gauged by pulse wave velocity is not a factor in the pathogenesis of hypertension.

scholarly journals Radiative transport in large arteries

2013 ◽  
Vol 5 (1) ◽  
pp. 54 ◽  
Author(s):  
Dominic Ruh ◽  
Sivaraman Subramanian ◽  
Michael Theodor ◽  
Hans Zappe ◽  
Andreas Seifert
Keyword(s):  
Radiative Transport ◽  
Large Arteries

scholarly journals IV. The development of the branchial arterial arches in birds, with special reference to the origin of the subclavians and carotids

1888 ◽  
Vol 179 ◽  
pp. 111-139 ◽  
Keyword(s):  
Special Reference ◽  
Large Arteries

The manner in which the large arteries springing from the aorta in the higher Vertebrates have been developed from a set of arches comparable to the branchial vessels of Fishes has been clearly explained by Rathke, and since his time the views which he first set forward in the year 1857 have been almost universally accepted and taught. I have been led to believe, however, from the observation of certain facts in the anatomy and development of Birds, that the deductions which Rathke made, drawn chiefly from his studies in Mammalian and Reptilian embryology, are not applicable to this class in so far as concerns the origin of the carotid and subclavian arteries. I am inclined to think also that the theories now generally held will be found inadequate to explain the origin of these vessels in the case of Chelonian and Crocodilian Reptiles, and to a less extent probably in Mammals.

scholarly journals Marginal dietary zinc deficiency in vivo induces vascular smooth muscle cell apoptosis in large arteries

2013 ◽  
Vol 99 (3) ◽  
pp. 525-534 ◽  
Author(s):  
Keith Allen-Redpath ◽  
Ou Ou ◽  
John H. Beattie ◽  
In-Sook Kwun ◽  
Jorg Feldmann ◽  
...  
Keyword(s):  
Smooth Muscle ◽  
Vascular Smooth Muscle ◽  
Smooth Muscle Cell ◽  
Muscle Cell ◽  
Zinc Deficiency ◽  
Vascular Smooth Muscle Cell ◽  
Cell Apoptosis ◽  
Large Arteries ◽  
Muscle Cell Apoptosis

scholarly journals Attenuation and Recovery of Brain Stem Autoregulation in Spontaneously Hypertensive Rats

1998 ◽  
Vol 18 (3) ◽  
pp. 305-310 ◽  
Author(s):  
Kazunori Toyoda ◽  
Kenichiro Fujii ◽  
Setsuro Ibayashi ◽  
Takanari Kitazono ◽  
Tetsuhiko Nagao ◽  
...  
Keyword(s):  
Brain Stem ◽  
Basilar Artery ◽  
Antihypertensive Treatment ◽  
Spontaneously Hypertensive Rats ◽  
Hypertensive Rats ◽  
Doppler Flowmetry ◽  
Spontaneously Hypertensive ◽  
Large Arteries ◽  
Cranial Window ◽  
Severe Hypotension

Cerebral large arteries dilate actively around the lower limits of CBF autoregulation, mediated at least partly by nitric oxide, and maintain CBF during severe hypotension. We tested the hypothesis that this autoregulatory response of large arteries, as well as the response of arterioles, is altered in spontaneously hypertensive rats (SHR) and that the altered response reverts to normal during long-term antihypertensive treatment with cilazapril, an angiotensin-converting enzyme inhibitor. In anesthetized 6- to 7-month-old normotensive Wistar-Kyoto rats (WKY), 4- and 6- to 7-month-old SHR without antihypertensive treatment, and 6- to 7-month-old SHR treated with cilazapril for 10 weeks, local CBF to the brain stem was determined with laser—Doppler flowmetry and diameters of the basilar artery and its branches were measured through a cranial window during stepwise hemorrhagic hypotension. The lower limit of CBF autoregulation shifted upward in untreated SHR to 90 to 105 mm Hg from 30 to 45 mm Hg in WKY, and it reverted to 30 to 45 mm Hg in treated SHR. In response to severe hypotension, the basilar artery dilated by 21 ± 6% (mean ± SD) of the baseline internal diameter in WKY. The vasodilation was impaired in untreated SHR (10 ± 8% in 4-mo-old SHR and 4 ± 5% in 6- to 7-month-old SHR), and was restored to 22 ± 10% by treatment with cilazapril ( P < 0.005). Dilator responses of branch arterioles to hypotension showed similar attenuation and recovery as that of the basilar artery. The data indicate that chronic hypertension impairs the autoregulatory dilation of the basilar artery as well as branch arterioles and that antihypertensive treatment with cilazapril restores the diminished dilation toward normal.

A new geometric procedure forIn vivo pulsed doppler evaluation of velocity distribution inside the diametrical section of large arteries in humans

1990 ◽  
Vol 18 (5) ◽  
pp. 519-531 ◽  
Author(s):  
Patrice Flaud ◽  
Aziz Bensalah ◽  
Jean Louis Counord ◽  
Jaime Levenson ◽  
Alain Simon
Keyword(s):  
Velocity Distribution ◽  
Pulsed Doppler ◽  
Large Arteries ◽  
Doppler Evaluation
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