In vitro cultivation of shrimp Taura syndrome virus (TSV) in a C6/36 mosquito cell line

2011 ◽  
Vol 34 (10) ◽  
pp. 805-810 ◽  
Author(s):  
N Arunrut ◽  
J Phromjai ◽  
W Gangnonngiw ◽  
N Kanthong ◽  
S Sriurairatana ◽  
...  
2021 ◽  
Author(s):  
S. Santhosh Kumar ◽  
S. Sivakumar ◽  
S. Abdul Majeed ◽  
S. Vimal ◽  
G. Taju ◽  
...  

Author(s):  
M.G.R. Varma ◽  
Mary Pudney ◽  
C.J. Leake ◽  
Christine Wallace

PLoS ONE ◽  
2014 ◽  
Vol 9 (4) ◽  
pp. e96107 ◽  
Author(s):  
Jaime G. Mayoral ◽  
Kayvan Etebari ◽  
Mazhar Hussain ◽  
Alexander A. Khromykh ◽  
Sassan Asgari

2007 ◽  
Vol 77 (2) ◽  
pp. 283-290 ◽  
Author(s):  
Juan Salas-Benito ◽  
Jorge Reyes-Del Valle ◽  
Ivonne Ceballos-Olvera ◽  
Rosa M. del Angel ◽  
Mariana Salas-Benito ◽  
...  

Author(s):  
Mary Pudney ◽  
Z. Marhoul ◽  
M.G.R. Varma ◽  
C.J. Leake

2006 ◽  
Vol 72 (2) ◽  
pp. 1705-1707 ◽  
Author(s):  
Maurício C. Horta ◽  
Marcelo B. Labruna ◽  
Edison L. Durigon ◽  
Teresinha T. S. Schumaker

ABSTRACT We report the isolation and establishment of Rickettsia felis in the C6/36 cell line. Rickettsial growth was intense, always with 90 to 100% of cells being infected after few weeks. The rickettsial isolate was confirmed by testing infected cells by PCR and sequencing fragments of three major Rickettsia genes (gltA, ompB, and the 17-kDa protein gene).


1973 ◽  
Vol 138 (2) ◽  
pp. 364-372 ◽  
Author(s):  
M. Hatanaka ◽  
R. Klein ◽  
C. W. Long ◽  
R. Gilden

Tumorigenic and nontumorigenic mutants induced by a single 5'-bromodeoxyuridine (BrdU) treatment of a nonproducer (NP) tumorigenic cell line were isolated and characterized. Among the cloned derivatives were examples of virus-free and sarcoma virus-producing cell lines. Oncogenicity did not correlate with production of virus or ease of rescue of the sarcoma genome. All lines, including nononcogenic derivatives, retained the sarcoma genome. Phenotypic reversion of some cell mutants was observed after in vivo inoculation or long term in vitro cultivation. The M-50T cell line, obtained from a tumor induced by M-50 cells, had a sarcoma genome rescuable by direct superinfection; this was only achieved with parental M-50 cells by a cell fusion rescue technique. The M-43-2T cell, obtained from a single small static tumor induced by otherwise nononcogenic M-43-2 cells, shed sarcoma virus and became tumorigenic. M-58-4-48 became tumorigenic after passage 48 of the M-58-4 line, which was originally nontumorigenic. These observations of phenotypic reversion demonstrate that the presence of the sarcoma gene in cells is an essential but not sufficient condition of tumorigenesis.


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