scholarly journals Relation of urinary albumin excretion to coronary heart disease and low renal function: Role of blood pressure

2004 ◽  
Vol 65 (6) ◽  
pp. 2290-2297 ◽  
Author(s):  
Massimo Cirillo ◽  
Martino Laurenzi ◽  
Paolo Panarelli ◽  
Mario Mancini ◽  
Alberto Zanchetti ◽  
...  
2014 ◽  
Vol 26 (1) ◽  
pp. 220-229 ◽  
Author(s):  
Juan F. Navarro-González ◽  
Carmen Mora-Fernández ◽  
Mercedes Muros de Fuentes ◽  
Jesús Chahin ◽  
María L. Méndez ◽  
...  

2012 ◽  
Vol 26 (1) ◽  
pp. 3-4 ◽  
Author(s):  
J. Sendra-Lillo ◽  
D. Sabater-Hernandez ◽  
A. de la Sierra ◽  
A. Sendra-Ortola ◽  
A. Denia-Tomas ◽  
...  

2018 ◽  
Vol 2018 ◽  
pp. 1-5
Author(s):  
Hidenori Hirukawa ◽  
Shinji Kamei ◽  
Tomohiko Kimura ◽  
Atsushi Obata ◽  
Kenji Kohara ◽  
...  

It is very important to explore how we can reduce urinary albumin excretion which is an independent risk factor for ischemic heart disease. In this study, we retrospectively evaluated the effects of RAS inhibitor therapy on diabetic nephropathy in Japanese subjects whose urinary albumin levels were within normal range. We enrolled 100 subjects with type 2 diabetes who did not take any renin-angiotensin system (RAS) inhibitor. We defined the subjects taking RAS inhibitor for more than 3 years as RAS inhibitor group. RAS inhibitor exerted protective effect on the progression of urinary albumin excretion in subjects with type 2 diabetes without diabetic nephropathy. In addition, RAS inhibitor exerted more protective effects on renal function especially in subjects with poor glycemic control. In conclusion, RAS inhibitor could protect renal function against the deleterious effect of chronic hyperglycemia in Japanese subjects with type 2 diabetes even before the onset of diabetic nephropathy.


Diabetes ◽  
1990 ◽  
Vol 39 (2) ◽  
pp. 245-249 ◽  
Author(s):  
E. R. Mathiesen ◽  
B. Ronn ◽  
T. Jensen ◽  
B. Storm ◽  
T. Deckert

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