A comparative study of potassium ion transport in liver mitochondria of laboratory rats, pigeons (Columba livia) and guineafowl (Numida meleagris)

2017 ◽  
Vol 53 (4) ◽  
pp. 343-345 ◽  
Author(s):  
M. V. Dubinin ◽  
A. A. Vedernikov ◽  
E. I. Khoroshavina ◽  
A. E. Smirnova ◽  
V. N. Samartsev
Keyword(s):  
Comparative Study ◽  
Ion Transport ◽  
Columba Livia ◽  
Liver Mitochondria ◽  
Potassium Ion ◽  
Laboratory Rats ◽  
Numida Meleagris ◽  
Potassium Ion Transport

Potassium Ion Transport by Valinomycin across a Hg-Supported Lipid Bilayer

2005 ◽  
Vol 127 (38) ◽  
pp. 13316-13323 ◽  
Author(s):  
Lucia Becucci ◽  
Maria Rosa Moncelli ◽  
Renate Naumann ◽  
Rolando Guidelli
Keyword(s):  
Ion Transport ◽  
Lipid Bilayer ◽  
Potassium Ion ◽  
Supported Lipid Bilayer ◽  
Potassium Ion Transport

KCNQ5 Participates in Form Deprivation Myopia by Regulating Potassium Ion Transport Pathway

2021 ◽  
Author(s):  
Qin Yang ◽  
Qingqing Tan ◽  
Changjun Lan ◽  
Bozhen Lv ◽  
Guimei Zhou ◽  
...  
Keyword(s):  
Ion Transport ◽  
Potassium Ion ◽  
Transport Pathway ◽  
Potassium Ion Transport

scholarly journals Inhibition of Glycolysis Alters Potassium Ion Transport and Mitochondrial Redox Activity in Rat Brain

1988 ◽  
Vol 8 (6) ◽  
pp. 857-865 ◽  
Author(s):  
Cesar N. Raffin ◽  
Thomas J. Sick ◽  
Myron Rosenthal
Keyword(s):  
Ion Transport ◽  
Iodoacetic Acid ◽  
Potassium Ion ◽  
Redox Activity ◽  
Extracellular Potassium ◽  
Potassium Ion Transport ◽  
Ion Activity ◽  
Inhibition Of Glycolysis ◽  
Glycolytic Inhibitor ◽  
Active Processes

To examine the relationships between brain glycolysis, ion transport, and mitochondrial reduction/oxidation (redox) activity, extracellular potassium ion activity (K+0) and redox shifts of cytochrome oxidase (cytochrome a,a3) were recorded previous to and during superfusion of rat cerebral cortex with the glycolytic inhibitor iodoacetic acid (IAA). IAA produced oxidation of cytochrome a,a3, increased local oxygenation, increased K+0, and, in response to neuronal activation, slowed rates of K+0 reaccumulation. Rates of rereduction of cytochrome a,a3, after the oxidation of this cytochrome by stimulation, were also slowed by IAA. These effects of IAA demonstrate the dependence of K+0 reaccumulation on the integrity of glycolysis, support the concept that active processes are involved in brain ion transport, and suggest a link between ATP supplied by glycolysis and ion transport activity. These data are also compatible with the suggestion that residual dysfunctions after brain ischemia result from derangements in glycolytic functioning rather than from limitations in oxygen availability or oxidative metabolic activity.

scholarly journals Energetics of Potassium Ion Transport in Aplysia Gut

2002 ◽  
Vol 19 (6) ◽  
pp. 629-632 ◽  
Author(s):  
George A. Gerencser ◽  
Stanley Y. Loo ◽  
Kuuleialoha M. Cornette ◽  
Jianliang Zhang
Keyword(s):  
Ion Transport ◽  
Potassium Ion ◽  
Potassium Ion Transport

Potassium ion transport by gramicidin and valinomycin across a Ag(111)-supported tethered bilayer lipid membrane

2008 ◽  
Vol 53 (22) ◽  
pp. 6372-6379 ◽  
Author(s):  
Lucia Becucci ◽  
Massimo Innocenti ◽  
Emanuele Salvietti ◽  
Alessio Rindi ◽  
Irene Pasquini ◽  
...  
Keyword(s):  
Ion Transport ◽  
Lipid Membrane ◽  
Bilayer Lipid Membrane ◽  
Potassium Ion ◽  
Bilayer Lipid ◽  
Potassium Ion Transport
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