scholarly journals Clonidine-induced dilutional hyponatraemia

1979 ◽  
Vol 55 (639) ◽  
pp. 42-43 ◽  
Author(s):  
A. W. Burrows ◽  
B. Gribbin
2015 ◽  
Vol 130 (2) ◽  
pp. 117-124 ◽  
Author(s):  
Giovanni Sansoè ◽  
Manuela Aragno ◽  
Raffaella Mastrocola ◽  
Maurizio Parola

Adrenergic hyper-function reduces renal excretion of water. In advanced cirrhosis, hypersecretion of vasopressin (antidiuretic hormone or ADH) is considered the cause of dilutional hyponatraemia. We show that in experimental cirrhosis sympatholytic agents (α2A-adrenoceptor agonists) are as effective as V2 antagonists to blunt water retention.


2018 ◽  
pp. bcr-2018-226154
Author(s):  
Simon Mifsud ◽  
Maria Alessandra Zammit ◽  
Ramon Casha ◽  
Claudia Fsadni

The syndrome of inappropriate antidiuretic hormone (SIADH) secretion is a frequent cause of hyponatraemia. It is a dilutional hyponatraemia secondary to impaired urinary dilution in the absence of renal disease or any identifiable non-osmotic stimulus known to induce antidiuretic hormone secretion. SIADH can arise secondary to various respiratory tract infections; however, the association between SIADH and influenza A infection is described in only a few cases in the literature. The authors present a case report of influenza A that may have caused a profound SIADH-related hyponatraemia.


BMJ ◽  
1966 ◽  
Vol 2 (5529) ◽  
pp. 1573-1574 ◽  
Author(s):  
J. F. Hallpike ◽  
J. A. Morgan-Hughes

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