Forest inventory with optimal two-phase two-stage sampling schemes based on the anticipated variance

1999 ◽  
Vol 29 (11) ◽  
pp. 1691-1708 ◽  
Author(s):  
Daniel Mandallaz ◽  
Ronghua Ye

This work presents optimal sampling schemes for forest inventory. The sampling procedures are optimal in the sense that they minimize the anticipated variance for given costs or conversely, the anticipated variance is the average of the design-based variance under a local Poisson model for the spatial distribution of the trees. The resulting optimal inclusion rules are either probability proportional to size, in one-stage procedures, or a combination of probability proportional to prediction and probability proportional to error, in two-stage procedures. Best feasible approximations of the exact optimal sampling schemes are also given.

2001 ◽  
Vol 31 (10) ◽  
pp. 1845-1853 ◽  
Author(s):  
Daniel Mandallaz ◽  
Adrian Lanz

This work presents optimal allocation rules for two-phase, two-stage sampling schemes in which the sampling density and the costs of the second phase can vary over domains. The optimality criterion is based on the anticipated variance. It also gives an improved version of discrete approximation for the resulting inclusion probabilities. An example illustrates the theory.


2003 ◽  
Vol 154 (3-4) ◽  
pp. 117-121 ◽  
Author(s):  
Daniel Mandallaz

This paper gives a non-mathematical review of the concept of anticipated variance which allows to solve entirely the optimisation problem for two-phase two-stage forest inventories with cluster or simple random sampling, in the sense that the anticipated variance is minimised for given costs. The anticipated variance is the average of the design-based variance under a local Poisson-model for the spatial distribution of the trees. The resulting sampling rules have a clear intuitive background and require only simple algebra to be implemented. The required parameters can be estimated from any pre-existing two-phase inventory. An example based on the Swiss National Inventory illustrates the method.


2002 ◽  
Vol 32 (12) ◽  
pp. 2236-2243 ◽  
Author(s):  
D Mandallaz

This note presents an important improvement for optimal sampling schemes based on the anticipated variance. The anticipated variance is defined as the average of the design-based variance under a simple stochastic model in which the trees are assumed to be uniformly and independently distributed within a given number of so-called Poisson strata. We consider two-phase two-stage cluster sampling schemes in which costs and terrestrial second-phase sampling density can vary over domains. The estimation procedure is based on post-stratification with respect to so-called working strata that do not need to be identical with the Poisson strata, usually unknown, which induces a lack of fit. It is then possible to derive analytically the optimal sampling schemes. Data from the Swiss National Inventory illustrates the method.


2009 ◽  
Vol 66 (1) ◽  
pp. 90-99 ◽  
Author(s):  
Paulo Justiniano Ribeiro Jr ◽  
Denise Nunes Viola ◽  
Clarice Garcia Borges Demétrio ◽  
Bryan F. Manly ◽  
Odair Aparecido Fernandes

Onion (Allium cepa) is one of the most cultivated and consumed vegetables in Brazil and its importance is due to the large laborforce involved. One of the main pests that affect this crop is the Onion Thrips (Thrips tabaci), but the spatial distribution of this insect, although important, has not been considered in crop management recommendations, experimental planning or sampling procedures. Our purpose here is to consider statistical tools to detect and model spatial patterns of the occurrence of the onion thrips. In order to characterize the spatial distribution pattern of the Onion Thrips a survey was carried out to record the number of insects in each development phase on onion plant leaves, on different dates and sample locations, in four rural properties with neighboring farms under different infestation levels and planting methods. The Mantel randomization test proved to be a useful tool to test for spatial correlation which, when detected, was described by a mixed spatial Poisson model with a geostatistical random component and parameters allowing for a characterization of the spatial pattern, as well as the production of prediction maps of susceptibility to levels of infestation throughout the area.


2010 ◽  
Vol 30 (S 01) ◽  
pp. S153-S155
Author(s):  
D. Delev ◽  
S. Pahl ◽  
J. Driesen ◽  
H. Brondke ◽  
J. Oldenburg ◽  
...  

1993 ◽  
Vol 69 (02) ◽  
pp. 124-129 ◽  
Author(s):  
Susan Solymoss ◽  
Kim Thi Phu Nguyen

SummaryActivated protein C (APC) is a vitamin K dependent anticoagulant which catalyzes the inactivation of factor Va and VIIIa, in a reaction modulated by phospholipid membrane surface, or blood platelets. APC prevents thrombin generation at a much lower concentration when added to recalcified plasma and phospholipid vesicles, than recalcified plasma and platelets. This observation was attributed to a platelet associated APC inhibitor. We have performed serial thrombin, factor V one stage and two stage assays and Western blotting of dilute recalcified plasma containing either phospholipid vesicles or platelets and APC. More thrombin was formed at a given APC concentration with platelets than phospholipid. One stage factor V values increased to higher levels with platelets and APC than phospholipid and APC. Two stage factor V values decreased substantially with platelets and 5 nM APC but remained unchanged with phospholipid and 5 nM APC. Western blotting of plasma factor V confirmed factor V activation in the presence of platelets and APC, but lack of factor V activation with phospholipid and APC. Inclusion of platelets or platelet membrane with phospholipid enhanced rather than inhibited APC catalyzed plasma factor V inactivation. Platelet activation further enhanced factor V activation and inactivation at any given APC concentration.Plasma thrombin generation in the presence of platelets and APC is related to ongoing factor V activation. No inhibition of APC inactivation of FVa occurs in the presence of platelets.


1967 ◽  
Vol 18 (01/02) ◽  
pp. 198-210 ◽  
Author(s):  
Ronald S Reno ◽  
Walter H Seegers

SummaryA two-stage assay procedure was developed for the determination of the autoprothrombin C titre which can be developed from prothrombin or autoprothrombin III containing solutions. The proenzyme is activated by Russell’s viper venom and the autoprothrombin C activity that appears is measured by its ability to shorten the partial thromboplastin time of bovine plasma.Using the assay, the autoprothrombin C titre was determined in the plasma of several species, as well as the percentage of it remaining in the serum from blood clotted in glass test tubes. Much autoprothrombin III remains in human serum. With sufficient thromboplastin it was completely utilized. Plasma from selected patients with coagulation disorders was assayed and only Stuart plasma was abnormal. In so-called factor VII, IX, and P.T.A. deficiency the autoprothrombin C titre and thrombin titre that could be developed was normal. In one case (prethrombin irregularity) practically no thrombin titre developed but the amount of autoprothrombin C which generated was in the normal range.Dogs were treated with Dicumarol and the autoprothrombin C titre that could be developed from their plasmas decreased until only traces could be detected. This coincided with a lowering of the thrombin titre that could be developed and a prolongation of the one-stage prothrombin time. While the Dicumarol was acting, the dogs were given an infusion of purified bovine prothrombin and the levels of autoprothrombin C, thrombin and one-stage prothrombin time were followed for several hours. The tests became normal immediately after the infusion and then went back to preinfusion levels over a period of 24 hrs.In other dogs the effect of Dicumarol was reversed by giving vitamin K1 intravenously. The effect of the vitamin was noticed as early as 20 min after administration.In response to vitamin K the most pronounced increase was with that portion of the prothrombin molecule which yields thrombin. The proportion of that protein with respect to the precursor of autoprothrombin C increased during the first hour and then started to go down and after 3 hrs was equal to the proportion normally found in plasma.


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