Augmented Plasma Protein C Activity after Coronary Thrombolysis with Urokinase in Patients with Acute Myocardial Infarction

Cardiology ◽  
1992 ◽  
Vol 80 (3-4) ◽  
pp. 252-256 ◽  
Author(s):  
Shinya Goto ◽  
Shunnosuke Handa ◽  
Yohko Kawai ◽  
Kiyoaki Watanabe ◽  
Sumihisa Abe ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Plasma Protein ◽  
Protein C ◽  
Coronary Thrombolysis ◽  
Protein C Activity

High Levels of Plasma Protein C in Nephrotic Syndrome

1985 ◽  
Vol 53 (01) ◽  
pp. 005-007 ◽  
Author(s):  
I Pabinger-Fasching ◽  
K Lechner ◽  
H Niessner ◽  
P Schmidt ◽  
E Balzar ◽  
...  
Keyword(s):  
Nephrotic Syndrome ◽  
Plasma Protein ◽  
Protein C ◽  
Inverse Correlation ◽  
Elevated Plasma ◽  
Significant Inverse Correlation ◽  
At Iii ◽  
Thrombotic Tendency ◽  
Protein C Activity

SummaryIn patients with severe nephrotic syndrome determinations of plasma protein C : Ag levels (8 patients: 5 adults, 3 children) and protein C activity (3 out of 8 patients) revealed significantly elevated plasma protein C concentrations. Furthermore we observed a significant inverse correlation of protein C : Ag to AT III : Ag levels. No protein C : Ag could be detected in the urine of two patients studied. We conclude from our data, that changes of plasma protein C do not contribute to the high thrombotic tendency in nephrotic syndrome.

scholarly journals Protein C Deficiency in a Patient of Acute Myocardial Infarction

1970 ◽  
Vol 3 (1) ◽  
pp. 34-35 ◽  
Author(s):  
Tamzeed Ahmed ◽  
Mahbub Mansur
Keyword(s):  
Risk Factors ◽  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Protein C ◽  
Low Density Lipoprotein ◽  
Density Lipoprotein ◽  
Oxidase Inhibitor ◽  
Coronary Angiogram ◽  
Protein C Deficiency ◽  
Xanthine Oxidase Inhibitor

A 42-year old male presented with acute myocardial infarction with no discernable risk factors; he never smoked; did not suffer from diabetes and had a well controlled blood pressure with single medication; plasma concentration of total cholesterol was on the upper normal limit, high and low density lipoprotein, cholesterol and triglyceride being normal. In addition to a single antihypertensive he received Allupurinol (Xanthine Oxidase inhibitor) for hyperuricaemia. Coronary angiogram revealed ectatic epicardial coronary arteries. The patient developed deep vein thrombosis of right leg after four days of the coronary angiogram. Coagulation analysis revealed protein C deficiency. The recognition of protein C deficiency as a risk factor for myocardial infarction is important as anticoagulators prevent further thrombotic events whereas inhibitors of platelet aggregation are ineffective. Ibrahim Med. Coll. J. 2009; 3(1): 34-35 Key words: myocardial infarction, risk factors, protein C deficiency, anticoagulation.   doi: 10.3329/imcj.v3i1.2918

Coronary thrombolysis by intravenous streptokinase in acute myocardial infarction: Acute and follow-up studies

1984 ◽  
Vol 53 (6) ◽  
pp. 655-661 ◽  
Author(s):  
James F. Spann ◽  
Sol Sherry ◽  
Blase A. Carabello ◽  
Barry S. Denenberg ◽  
Richard H. Mann ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Intravenous Streptokinase ◽  
Coronary Thrombolysis ◽  
Follow Up Studies

scholarly journals Short and Long-term Effects of Coronary Thrombolysis in Acute Myocardial Infarction : The treatments for myocardial infarction in acute phase

1993 ◽  
Vol 56 (SupplementV) ◽  
pp. 1409-1413
Author(s):  
Takeshi Motomiya ◽  
Yoshiki Tokuyasu ◽  
Harumizu Sakurada ◽  
Osamu Yanase ◽  
Shuzo Nomura ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Acute Phase ◽  
Long Term Effects ◽  
Coronary Thrombolysis ◽  
Short And Long Term
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