scholarly journals Rocking dizziness and headache: A two-way street

Cephalalgia ◽  
2013 ◽  
Vol 33 (14) ◽  
pp. 1160-1169 ◽  
Author(s):  
Yoon-Hee Cha ◽  
Yongyan Cui
Keyword(s):  
Migraine Headache ◽  
Prolonged Exposure ◽  
Age Of Onset ◽  
Interview Study ◽  
Vestibular Migraine ◽  
Passive Motion ◽  
Chronic Dizziness ◽  
Mal De Debarquement Syndrome ◽  
New Onset

Background Chronic rocking dizziness, often described as the feeling of being on a boat, is classically triggered by prolonged exposure to passive motion. Patients with this motion-triggered sensation of rocking, which is also known as mal de debarquement syndrome, often develop new onset headaches along with the dizziness. Chronic rocking dizziness has also been noted in vestibular migraine, occurring without a motion trigger. We sought to clarify the association between both motion-triggered (MT) and non-motion-triggered (non-MT) chronic rocking dizziness and headache history. Methods Our methods included questionnaire and interview study of subjects with either MT or non-MT chronic rocking dizziness. Results Onset of headaches was earlier in patients with non-MT rocking dizziness (median 26 years: MT; 16 years: non-MT). In MT subjects, there was a bimodal peak of age of onset of headache (20–29 years and 40–49 years). Most headache met criteria for migraine in both groups. By the time that chronic dizziness occurred, both groups had a comparable prevalence of migraine headache (41%: MT; 46%: non-MT). Pre-existing headache usually worsened after the onset of dizziness. Discussion Though rocking dizziness does not meet current criteria for vestibular migraine, migraine physiology may predispose to, develop in, or worsen with the onset of chronic rocking dizziness.

Age-related features in vestibular migraine onset: A multiparametric analysis

Cephalalgia ◽  
2020 ◽  
Vol 40 (14) ◽  
pp. 1605-1613 ◽  
Author(s):  
Michele Ori ◽  
Gabriele Arra ◽  
Michela Caricato ◽  
Rocco Freccia ◽  
Francesco Frati ◽  
...  
Keyword(s):  
Migraine Headache ◽  
Age Of Onset ◽  
Vestibular Migraine ◽  
Vestibular Disorders ◽  
Positional Vertigo ◽  
Multiparametric Analysis ◽  
Age Related ◽  
Vestibular Symptoms ◽  
Clinical Records ◽  
Migraine Onset

Background Clinical heterogeneity is a peculiarity of vestibular migraine, in contrast to other vestibular disorders that have a more stereotypical expression. Migraine presents a range of variability in symptoms depending on the age of the patient. Supposing that migraine headache and vestibular migraine share the same pathogenetic mechanisms, a multiparametric analysis was performed to verify the hypotheses of an age-related influence on the clinical features of vestibular migraine at the onset. Methods In this retrospective study, we analysed the clinical records of 72 consecutive patients affected by vestibular migraine from June 2012 to November 2018: 64 females and eight males; mean age 38.2 ± 9.6. We considered only patients that reported onset of vestibular symptoms within 12 months preceding inclusion into the study. Results Statistical analysis shows a significant increase in the diagnosis of probable vestibular migraine with increasing age and a decrease in vestibular migraine diagnosis ( p = 0.034). The incidence of spontaneous dizziness increases with age ( p = 0.012); by contrast, external spontaneous vertigo, and visually induced vertigo decrease after 40 years of age ( p = 0.018), clinically characterising the onset of juvenile forms. Spontaneous vertigo, head motion-induced vertigo/dizziness, and positional vertigo did not show significant variations with age. Conclusion Our data show that the type of vestibular symptoms in vestibular migraine varies according to the age of onset.

scholarly journals Sudden Onset of Vestibular Migraine Complicated with BPPV and Mal de Debarquement Syndrome – a Diagnostic Dilemma

2021 ◽  
Vol 13 (6) ◽  
pp. 139-141
Author(s):  
R. Perkoviс ◽  
S. Maslovara ◽  
K. Kovaсeviс′ Stranski ◽  
K. Buljan ◽  
S. Tomiс′ ◽  
...  
Keyword(s):  
Migraine Patient ◽  
Mthfr C677t ◽  
Sudden Onset ◽  
Vestibular Migraine ◽  
Diagnostic Dilemma ◽  
High Association ◽  
Detailed Medical History ◽  
Mal De Debarquement Syndrome ◽  
History Of ◽  
Vestibular Symptoms

Vestibular migraine is combination of migraine and vestibular symptoms. In clinical examination it can be replaced with benign paroxysmal positional vertigo (BPPV) cupulolithiasis, but also BPPV is common comorbidity in migraine patients. There is also high association between vestibular migraine and Mal de Debarquement syndrome. Patient came to hospital with vertigo that was diagnosed as left PC-BPPV canalolithiasis. After first Epley's maneuver symptoms didn't resolved. Week after, at second Epley's maneuver performed patient developed left PC-BPPV cupulolithiasis. Month after, at third Epley's maneuver BPPV resolved but patient developed Mal de Debarquement syndrome. Laboratory testing showed hyperhomocisteinemia and homozygous MTHFR C677T and PAI, with low vitamin D. After reviewing the vestibular symptoms in the first attack which was misdiagnosed as BPPV canalolithiasis, and history of migraine, patient was diagnosed with vestibular migraine. Patient well responded to migraine diet and supplementation with B complex. Vestibular disorders are similar to each other and they can overlap. More attention in taking detailed medical history should be given to patients with vertigo or dizziness.

Migraine and vertigo

2020 ◽  
pp. 128-137
Author(s):  
Yoon-Hee Cha
Keyword(s):  
Migraine Headache ◽  
Clinical Characteristics ◽  
Vestibular Migraine ◽  
Headache Disorders ◽  
Temporal Profile ◽  
Growing Body ◽  
Vestibular Symptoms ◽  
The Common ◽  
Treatment Responses

The phenomena of migraine headache and vertigo share many epidemiological, anatomical, and clinical characteristics. The historically parallel development of the neuroscience of each field has formally intersected in the development of consensus criteria for vestibular migraine and the inclusion of vestibular migraine in the International Classification of Headache Disorders. Differences exist in the temporal profile of head pain and vertigo as manifestations of migraine, which can obscure the association. However, the growing body of evidence on the common demographic, neurochemical signature, and treatment responses of pain and vestibular symptoms indicate that they exist as symptoms of a common syndrome, one which can only be fully understood by recognizing the significance of each kind of manifestation.

STROKE DUE TO MIGRAINE HEADACHE PRESENTING AS NEW ONSET PSYCHOSIS

1998 ◽  
Vol 91 (Supplement) ◽  
pp. S52
Author(s):  
Alan Jacovich ◽  
George Moses ◽  
Jan Carlton
Keyword(s):  
Migraine Headache ◽  
New Onset

Prolonged exposure to damp and moldy workplaces and new-onset asthma

2011 ◽  
Vol 84 (7) ◽  
pp. 713-721 ◽  
Author(s):  
Kirsi Karvala ◽  
Elina Toskala ◽  
Ritva Luukkonen ◽  
Jukka Uitti ◽  
Sanna Lappalainen ◽  
...  
Keyword(s):  
Prolonged Exposure ◽  
New Onset

scholarly journals A Case Report of Cortisol-Secreating Adrenal Adenoma Causing Cushing Syndrome

2019 ◽  
Vol 3 (1) ◽  
pp. 01-08
Author(s):  
Husain Radhi
Keyword(s):  
Adrenal Glands ◽  
Prolonged Exposure ◽  
Cushing Syndrome ◽  
Adrenal Adenoma ◽  
Laboratory Data ◽  
Clinical Manifestations ◽  
Endogenous Production ◽  
Morning Cortisol ◽  
Free Cortisol ◽  
New Onset

Cushing syndrome is a state of prolonged exposure to excess cortisol production. Causes can be varied ranging from exogenous intake or endogenous production from a pituitary source due to overproduction of adrenocorticotropic hormone (ACTH) or due to over secretion from unilateral or bilateral adrenal glands. Cushing syndrome in an adult can present with proximal muscle weakness, facial plethora, wasting of the extremities with increased fat in the abdomen and face, wide purplish striae, bruising with no obvious trauma, and supraclavicular fat pads. Here, we report a 38 year old male who presented with suspicion of having Cushing syndrome due to his clinical manifestations and high cortisol levels. He presented to the clinic with new onset diabetes, severe hypertension, chronic abdominal pain and distension which was being investigating by different specialties. The patient underwent CT scan of the abdomen which show right adrenal adenoma and on examination, he was found to have proximal weakness and purple striae. Laboratory data showed high morning cortisol, 24 urine free cortisol and ACTH levels which were strongly suggestive of ACTH independent Cushing syndrome.

Clozapine efficiency in tardive syndromes induced by anti-psychotic treatment

2017 ◽  
Vol 41 (S1) ◽  
pp. S754-S755
Author(s):  
L. Jouini ◽  
U. Ouali ◽  
R. Zaouche ◽  
R. Jomli ◽  
Y. Zgueb ◽  
...  
Keyword(s):  
Prolonged Exposure ◽  
Clinical Signs ◽  
Anti Psychotic ◽  
Adverse Side Effects ◽  
2Nd Generation ◽  
Segmental Dystonia ◽  
Competing Interest ◽  
Low Prevalence ◽  
Cholinergic Effects ◽  
New Onset

IntroductionTardive syndromes (TS) resulting from prolonged exposure to dopamine receptor blocking agents are frequent. Clozapine is considered to have a low risk of causing new onset TS and accounts therefore as an interesting option in patients with invalidating TS.ObjectivesOur study aims to describe clozapine indications in patients experiencing TS.MethodsPresentation of the clinical cases of five patients, who experienced different kinds of TS secondary to 1st and 2nd generation anti-psychotic treatment.ResultsWe present the cases of AB aged 41, MJ aged 40, HM aged 31 and AS aged 30, diagnosed with schizophrenia; and FB aged 24,diagnosed with schizoaffective disorder. Adverse side effects to conventional anti-psychotics such as limb and trunk tremors were described for AB, choreic limb movements, axial and segmental dystonia for MJ, AS, FB and oculogyration for FB. All patients were switched to atypical anti-psychotics without improvement of the TS. The switch to clozapine, associated with abotulinum injection for MJ, led to regression of the TS and improvement of clinical signs. In fact, according to several studies, clozapine seems to be an interesting option when invalidating TS occurs. The low prevalence of TS under clozapine can be explained by its low affinity for striatal-D2 receptors, its anti-serotonin and anti-cholinergic effects.ConclusionsClozapine should be considered in symptomatic patients who develop TS while receiving other anti-psychotics. Further research on mechanism of TS and clozapine effect on TS is needed.Disclosure of interestThe authors have not supplied their declaration of competing interest.

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