Pathophysiology and Prevalence of Detrusor-Sphincter Dyssynergia

2020 ◽  
pp. 27-30
Author(s):  
Marcio Augusto Averbeck ◽  
Helmut Madersbacher
Keyword(s):  
Detrusor Sphincter Dyssynergia

Role of Uttarabasti in the management of Functional Bladder Outlet Obstruction: A Case Study

2017 ◽  
Vol 2 (3) ◽  
Author(s):  
Jaimin R. Patel
Keyword(s):  
Bladder Outlet Obstruction ◽  
Outlet Obstruction ◽  
Urine Flow ◽  
Complete Relief ◽  
Urinary Flow ◽  
Detrusor Sphincter Dyssynergia ◽  
Symptom Appraisal ◽  
Prostatic Enlargement ◽  
Normal Urine ◽  
Outflow Channel

Bladder outlet obstruction (BOO) produces compression or resistance upon the bladder outflow channel at any location from the bladder neck to urethral meatus. It may be induced by specific functional and anatomic causes. Functional obstruction may be caused by detrusor-sphincter dyssynergia (DSD) and anatomic obstruction most commonly from benign prostatic enlargement (BPH) or urethral stricture. Obstructive symptoms include hesitancy, sensation of incomplete bladder emptying, diminished urinary stream. The combination of PVR, urinary flow measures, and symptom appraisal has been generally accepted as the initial screening and evaluation paradigm for BOO. In, Ayurveda, BOO is similar to Mutraghata means obstruction in the urine flow. Uttarbasti is the prime treatment of Mutraghata. Present case is diagnosed as a functional bladder outlet obstruction (BOO) on the basis of symptoms, normal reports of USG and ascending urethrogram and diminished flow of urine in Uroflowmetry. Total 7 Uttarbasti with 50ml Sahcharadi Tailam was given along with Rasayana and Mutraghatahara medicine. Patient has complete relief in his obstructive urine complains and has normal urine flow without taking Tab. AFDURA after 7 years. And also improvement appear in Uroflowmetry.

Neurogenic urinary disorders in patients with tuberculous spondylitis before and after surgical treatment

2021 ◽  
Vol 11 (1) ◽  
pp. 27-32
Author(s):  
Aleksandr I. Gorbunov ◽  
Aleksandr N. Murav’ev ◽  
Evgenij G. Sokolovich ◽  
Petr K. Yablonsky
Keyword(s):  
Urinary Tract ◽  
Detrusor Overactivity ◽  
Lower Urinary Tract ◽  
De Novo ◽  
Spinal Tuberculosis ◽  
Neurological Status ◽  
Lower Urinary Tract Dysfunction ◽  
Detrusor Sphincter Dyssynergia ◽  
Before And After ◽  
Lower Urinary

ABSTRACT: Tuberculosis inflammation of vertebral column (spondylitis) can lead to neurogenic lower urinary tract dysfunction. There is lack of available publications for neurogenic lower urinary tract dysfunction in spinal tuberculosis. OBJECTIVE: To evaluate urodynamic disturbances in spinal tuberculosis before and after surgery for spondylitis. MATERIALS AND METHODS: We observed 19 patients with spinal tuberculosis, who had symptoms of micturitions impairment. 14 patients (73,6%) were male and 5 (26,4%) were female, average age was 43,7 7,9 years (2766). Control evaluation was performed after surgery on day 2128. RESULTS: Before surgery we found detrusor overactivity in 11 (57,9%) patients and 2 of those with detrusor overactivity had detrusor-sphincter dyssynergia. Detrusor hypo-/acontractility was diagnosed in 8 (42,1%). After surgery 5 patients (26,3%) exhibited improvement, in one case urodynamic disturbances were resolved. One patient developed detrusor overactivity and incontinence de novo and one patient had worsening neurological status, loss of sensitivity and acontractile bladder. CONCLUSION: Variable lower urinary tract dysfunction can be diagnosed in spinal tuberculosis. Only 26,3% of patients have improvement after surgery. New conditions or worsening of previous neurogenic lower urinary tract dysfunctions can be observed.

Urethral Stents for Detrusor Sphincter Dyssynergia

2006 ◽  
Vol 175 (4) ◽  
pp. 1396-1396
Author(s):  
T. Nambirajan ◽  
S. Woolsey ◽  
V. Mahendra ◽  
A.R. Stone ◽  
I.K. Walsh
Keyword(s):  
Detrusor Sphincter Dyssynergia

scholarly journals Possible Role of Large Fluid Intake in Delaying Formation of Encrustations and, thereby, Prolonging Working Life of Memokath Stent for Nearly 14 Years in a Spinal Cord Injury Patient

2007 ◽  
Vol 7 ◽  
pp. 1663-1669 ◽  
Author(s):  
Subramanian Vaidyanathan ◽  
Bakul M. Soni ◽  
Peter L. Hughes ◽  
Gupreet Singh
Keyword(s):  
Spinal Cord ◽  
Spinal Cord Injury ◽  
Fluid Intake ◽  
Stone Formation ◽  
Outlet Obstruction ◽  
Spinal Cord Injury Patient ◽  
Autonomic Dysreflexia ◽  
Detrusor Sphincter Dyssynergia ◽  
Cord Injury ◽  
Injury Patient

The Memokath stent has been used in spinal cord injury patients as a reversible alternative to external urethral sphincterotomy, but the stent has a finite lifetime of <2 years before failure in the majority of patients. We report an unusual case of a spinal cord injury patient in whom memokath stent was functioning for almost 14 years. The long life span of the Memokath in this patient was probably due to this person's habit of drinking around 5 l of fluids a day. Large fluid intake resulted in high urine output and, consequently, deceased the risk of urine infections and delayed formation of encrustations around the stent. Although this case represents an unusual length of time for a Memokath stent to have been in place and functioning, caution should be exercised against the long-term use of Memokath stents. Memokath stents do not get absorbed into the mucosa unlike urolume stents and, therefore, are prone to stone formation. Further, Memokath stents have not yet been approved in the U.S. either for bladder outlet obstruction or detrusor-sphincter dyssynergia. This case is also a reminder to health professionals that if a tetraplegic patient, in whom a Memokath stent has been deployed for treatment of detrusor-sphincter dyssynergia, presents with autonomic dysreflexia, encrustations blocking the lumen of the stent or calculus formation around the stent should be considered as possible reasons for autonomic dysreflexia.

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