Decrease of Immunoglobulin G-Fc Receptor-Bearing T Lymphocytes in Graves' Disease*

1982 ◽  
Vol 55 (3) ◽  
pp. 399-402 ◽  
Author(s):  
HIDEMITSU MORI ◽  
NOBUYUKI AMINO ◽  
YOSHINORI IWATANI ◽  
SEISHI ASARI ◽  
YASUYUKI IZUMIGUCHI ◽  
...  
Keyword(s):  
T Lymphocytes ◽  
Immunoglobulin G ◽  
Fc Receptor ◽  
Graves Disease

scholarly journals Expansion of Fc receptor-bearing T lymphocytes in patients with immunoglobulin G and immunoglobulin A myeloma.

1981 ◽  
Vol 67 (1) ◽  
pp. 308-311 ◽  
Author(s):  
R G Hoover ◽  
S Hickman ◽  
H M Gebel ◽  
N Rebbe ◽  
R G Lynch
Keyword(s):  
T Lymphocytes ◽  
Immunoglobulin G ◽  
Immunoglobulin A ◽  
Fc Receptor

scholarly journals Structural basis for pH-insensitive inhibition of immunoglobulin G recycling by an anti-neonatal Fc receptor antibody

2017 ◽  
Vol 292 (42) ◽  
pp. 17449-17460 ◽  
Author(s):  
Jon A. Kenniston ◽  
Brandy M. Taylor ◽  
Gregory P. Conley ◽  
Janja Cosic ◽  
Kris J. Kopacz ◽  
...  
Keyword(s):  
Immunoglobulin G ◽  
Fc Receptor ◽  
Structural Basis ◽  
Neonatal Fc Receptor ◽  
Receptor Antibody

scholarly journals Augmentation of macrophage complement receptor function in vitro. I. Characterization of the cellular interactions required for the generation of a T-lymphocyte product that enhances macrophage complement receptor function.

1979 ◽  
Vol 150 (3) ◽  
pp. 653-675 ◽  
Author(s):  
J A Griffin ◽  
F M Griffin
Keyword(s):  
T Lymphocytes ◽  
Complex Disease ◽  
Peritoneal Macrophages ◽  
Fc Receptor ◽  
Microbial Pathogens ◽  
Complement Receptor ◽  
Complement Receptors ◽  
Cellular Interactions ◽  
Receptor Function

The function of complement receptors of mouse peritoneal macrophages was converted in vitro from mediating only attachment of macrophage complement receptor function was achieved by treating freshly explanted macrophages with supernates from cultures containing T lymphocytes and appropriately triggered macrophages. Fc receptor-mediated phagocyctosis by macrophages was required for the production of active supernates, for neither ingestion via the cells' complement receptors nor ingestion via nonimmunologic means was a sufficient stimulus for the macrophages' participation in the generation of supernatant activity. Fc receptor-triggered macrophages interacted by a contact dependent, but histocompatibility independent, mechanism with T lymphocytes, thereby signalling the lymphocytes to elaborate the active product. The possible significance of enhanced macrophage complement receptor function in inflammation, host defense against microbial pathogens, immune complex disease, and neoplasia is discussed.

Metal-catalyzed oxidation of immunoglobulin G impairs Fc receptor-mediated binding to macrophages

1998 ◽  
Vol 25 (7) ◽  
pp. 780-785 ◽  
Author(s):  
Linda Margiloff ◽  
Larissa Chaplia ◽  
Andrew Chow ◽  
Pravin C. Singhal ◽  
Joseph Mattana
Keyword(s):  
Immunoglobulin G ◽  
Fc Receptor ◽  
Metal Catalyzed Oxidation ◽  
Metal Catalyzed ◽  
Catalyzed Oxidation

TSAb AND T LYMPHOCYTES SUBSET CHANGES IN PATIENTS WITH GRAVES' DISEASE (GD) TREATED BY CARBIMAZOLE (C)

1985 ◽  
pp. 419-420 ◽  
Author(s):  
A.M. Madec ◽  
J. Orgiazzi ◽  
A. Stefanutti ◽  
N. Genetet ◽  
B. Genetet ◽  
...  
Keyword(s):  
T Lymphocytes ◽  
Graves Disease
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