scholarly journals Serum amyloid A and Janus kinase 2 in a mouse model of diabetic kidney disease

PLoS ONE ◽  
2019 ◽  
Vol 14 (2) ◽  
pp. e0211555 ◽  
Author(s):  
Brad P. Dieter ◽  
Rick L. Meek ◽  
Robert J. Anderberg ◽  
Sheryl K. Cooney ◽  
Jen L. Bergin ◽  
...  
2014 ◽  
Vol 95 (3) ◽  
pp. 250-262 ◽  
Author(s):  
Robert J Anderberg ◽  
Rick L Meek ◽  
Kelly L Hudkins ◽  
Sheryl K Cooney ◽  
Charles E Alpers ◽  
...  

2015 ◽  
Vol 95 (6) ◽  
pp. 697-697 ◽  
Author(s):  
Robert J Anderberg ◽  
Rick L Meek ◽  
Kelly L Hudkins ◽  
Sheryl K Cooney ◽  
Charles E Alpers ◽  
...  

2016 ◽  
Vol 30 (8) ◽  
pp. 1467-1472 ◽  
Author(s):  
Brad P. Dieter ◽  
Sterling M. McPherson ◽  
Maryam Afkarian ◽  
Ian H. de Boer ◽  
Rajnish Mehrotra ◽  
...  

2020 ◽  
Vol 158 (6) ◽  
pp. S-278
Author(s):  
Tanja Davis ◽  
Daleen Conradie ◽  
Preetha Shridas ◽  
Marcielle C. de Beer ◽  
Frederick c. de Beer ◽  
...  

2011 ◽  
Vol 21 (5) ◽  
pp. 285-291 ◽  
Author(s):  
Ariel Troib ◽  
Daniel Landau ◽  
Jack F. Youngren ◽  
Leonid Kachko ◽  
Ralph Rabkin ◽  
...  

2003 ◽  
Vol 284 (6) ◽  
pp. F1138-F1144 ◽  
Author(s):  
Kumar Sharma ◽  
Peter McCue ◽  
Stephen R. Dunn

Diabetic nephropathy is increasing in incidence and is now the number one cause of end-stage renal disease in the industrialized world. To gain insight into the genetic susceptibility and pathophysiology of diabetic nephropathy, an appropriate mouse model of diabetic nephropathy would be critical. A large number of mouse models of diabetes have been identified and their kidney disease characterized to various degrees. Perhaps the best characterized and most intensively investigated model is the db/ db mouse. Because this model appears to exhibit the most consistent and robust increase in albuminuria and mesangial matrix expansion, it has been used as a model of progressive diabetic renal disease. In this review, we present the findings from various studies on the renal pathology of the db/ db mouse model of diabetes in the context of human diabetic nephropathy. Furthermore, we discuss shortfalls of assessing functional renal disease in mouse models of diabetic kidney disease.


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