Early diagnosis and prompt treatment of severe thyrotoxicosis in patients with congestive cardiac failure is critical: lessons from a case of type 1 amiodarone induced severe thyrotoxicosis

2019 ◽  
Author(s):  
Rana Siddique ◽  
Tauni Rahat Ai
Keyword(s):  
Early Diagnosis ◽  
Cardiac Failure ◽  
Congestive Cardiac Failure ◽  
Prompt Treatment

An intrathoracic arteriovenous malformation discovered as an extremely uncommon reason of neonatal congestive cardiac failure

2009 ◽  
Vol 19 (5) ◽  
pp. 530-533 ◽  
Author(s):  
Sigrun R. Hofmann ◽  
Matthias Weise ◽  
Katharina I. Nitzsche
Keyword(s):  
Arteriovenous Malformation ◽  
Cardiac Failure ◽  
Arteriovenous Malformations ◽  
Neonatal Period ◽  
High Flow ◽  
Congestive Cardiac Failure ◽  
Vein Of Galen ◽  
Prenatal Detection ◽  
First Case

AbstractCongenital arteriovenous malformations are rare causes of congestive cardiac failure in neonates. The most common sites are in the head and liver, but other sites include the thorax, the abdomen and the limbs. The onset of failure is usually not in the immediate neonatal period, but later on in life, albeit that lesions such as the arteriovenous malformation of the vein of Galen, and other arteriovenous malformations in different locations which produce high flow can present early. We describe here the first case, to the best of our knowledge, of prenatal detection of an intrathoracic arteriovenous malformation producing neonatal cardiac failure, which was successfully treated by surgery postnatally.

MULTINODULAR HEMANGIOMATOSIS OF THE LIVER IN INFANCY

PEDIATRICS ◽  
1972 ◽  
Vol 49 (4) ◽  
pp. 563-573
Author(s):  
Robert H. McLean ◽  
James H. Moller ◽  
Warren J. Warwick ◽  
Leon Satran ◽  
Russell V. Lucas
Keyword(s):  
Cardiac Failure ◽  
Intensive Treatment ◽  
Combined Effect ◽  
Arteriovenous Shunt ◽  
Congestive Cardiac Failure ◽  
Treatment Modalities ◽  
Arteriovenous Fistulae ◽  
Classical Triad ◽  
Hepatic Hemangiomas ◽  
High Output

Multinodular hemangiomatosis of the liver (MHL) is characterized pathologically by multiple hemangiomas, primarily in the liver but also in other organs. The hepatic hemangiomas act as small arteriovenous fistulae and their combined effect may result in a massive peripheral arteriovenous shunt and high output congestive cardiac failure. Patients with MHL may be recognized clinically by the classical triad of congestive cardiac failure, hepatomegaly, and cutaneous hemangiomas. All but 2 of 31 cases of MHL herein reviewed had the onset of symptoms prior to 6 months of age. High output cardiac failure were severe and resulted in a 70% mortality. Hepatomegaly was massive and out of proportion to the degree of cardiac failure. Therapy includes intensive treatment of congestive cardiac failure and judicious transfusion in anemic patients. Irradiation of the liver and corticosteroids have been utilized in attempts to speed evolution of the hepatic arteriovenous fistulae. The data are not sufficient to establish the efficacy of either of these treatment modalities.

POTT’S PUFFY TUMOR: A CASE REPORT

2020 ◽  
Vol 18 (Suppl.1) ◽  
pp. 93-96
Author(s):  
V. Stoyanov ◽  
D. Petkov ◽  
P. Bozdukova
Keyword(s):  
Case Report ◽  
Early Diagnosis ◽  
Rare Complication ◽  
Frontal Bone ◽  
Frontal Sinusitis ◽  
Subperiosteal Abscess ◽  
Prompt Treatment ◽  
Pott’S Puffy Tumor ◽  
Intracranial Complications ◽  
Life Threatening

Pott’s puffy tumor (PPT) is a rare complication of sinusitis characterized by osteomyelitis of the frontal bone with subperiosteal abscess presenting as frontal swelling. It was first described by Sir Percival Pott in 1768 in relation to frontal head trauma. Later, it was established that this entity is more common in relation to frontal sinusitis (1). In this article we report a case of PPT in a 17-year-old boy. CT scan confirmed subperiosteal abscess. At surgery, the subperiosteal abscess was drained and sequestrectomy of the affected frontal bone was done. Broad-spectrum antibiotics were given for 4 weeks. The patient recovered without residual problems and has remained well. PPT is now relatively uncommon and early diagnosis and prompt treatment is necessary to avoid further intracranial complications, which can be life-threatening.

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