Serial Munchausen Syndrome by Proxy

PEDIATRICS ◽  
1990 ◽  
Vol 86 (4) ◽  
pp. 581-585
Author(s):  
Randell Alexander ◽  
Wilbur Smith ◽  
Richard Stevenson

Five cases of Munchausen syndrome by proxy (MSBP) are presented in which more than one child in the family was victimized. There was a high incidence of maternal psychiatric histories, marital difficulties, and Münchhausen syndrome in the mothers themselves. Seventy-one percent of the children in the families were known to be victims of MSBP; four of these children (31%) died. Multiple-child MSBP may reflect more significant maternal psychopathology than found in other cases of MSBP, or it may indicate the deteriorating consequences to the mother and other children in the family if this syndrome is not identified with the first child and effective interventions made.

PEDIATRICS ◽  
1984 ◽  
Vol 74 (4) ◽  
pp. 554-556
Author(s):  
Roy Meadow ◽  
Thomas Lennert

The terms Munchausen syndrome by proxy and Polle syndrome have both been used to describe the situation in which one person persistently fabricates illness on behalf of another (usually a mother on behalf of her child). However, investigation of the family records of the real life Baron von Munchausen in Germany reveal that Polle syndrome is an inappropriate title originally derived from incorrect information.


2010 ◽  
Vol 30 (6) ◽  
pp. 46-55 ◽  
Author(s):  
Laura Criddle

This syndrome is associated with a high incidence of recidivism, morbidity, and mortality.


2012 ◽  
Author(s):  
Funda Kurt ◽  
Sevgi Baskan ◽  
Ozdecan Bezirci ◽  
Gurol Canturk ◽  
Zarife Kuloglu ◽  
...  

1993 ◽  
Vol 63 (2) ◽  
pp. 318-321 ◽  
Author(s):  
Herbert A. Schreier ◽  
Judith A. Libow

1979 ◽  
Vol 91 (1) ◽  
pp. 70-76 ◽  
Author(s):  
Paul Bratusch-Marrain ◽  
Hannes Haydl ◽  
Werner Waldhäusl ◽  
Robert Dudczak ◽  
Wolfgang Graninger

ABSTRACT A kindred is presented in which 4 members in 3 generations showed absent or reduced serum concentrations of thyroxine-binding globulin (TBG). TBG was undetectable by radioimmunoassay in one male and decreased to varying extent in 3 female patients (4.0, 4.2 and 8.6 μg/ml; normal range 12.5–26.0 μg/ml). Total thyroxine serum concentrations in the affected subjects were well in the hypothyroid range without clinical evidence of hypothyroidism. The mode of transmission of the trait was consistent with X-chromosome linkage. A high incidence of non-toxic goitre was also present in most of the family members examined irrespective of TBG levels. The transmission of the goitre trait was compatible with autosomal dominant inheritance. Thus its association with transmission of TBG deficiency was interpreted as not causal but coincidental.


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