Treatment of skin diseases with calcium salts

2021 ◽  
Vol 20 (6) ◽  
pp. 648-648
Author(s):  
V. G.
Keyword(s):  
Herpes Zoster ◽  
Calcium Chloride ◽  
Skin Diseases ◽  
Dermatitis Herpetiformis ◽  
Pruritus Ani ◽  
Calcium Salts ◽  
Intravenous Injections ◽  
Pruritus Vulvae

Rosentul (Med. Ob. Iizhn. Pov., 1924, No. 5) saw very good results from the use of calcium salts, in the form of intravenous injections of a 10% solution of calcium chloride, 10 cubic meters each. with. PA injection, with the latter being repeated daily, for a number of skin diseases, namely, for acute eczema, urticaria, herpes zoster, lichen chronicus Vidaln, pruritus vulvae, pruritus ani, dermatitis herpetiformis Dhringa, etc.

Treatment of sepsis with calcium salts. Professor Petrov (V. Chir. and Pogr. Obl., Vol. I, No. 2), Chistyakov (Nov. Chir. Arch., Vol. II, No. 1)

1923 ◽  
Vol 19 (2) ◽  
pp. 100-100
Author(s):  
V. Bogolyubov
Keyword(s):  
Calcium Chloride ◽  
Chloride Solution ◽  
Calcium Chloride Solution ◽  
Calcium Salts ◽  
Intravenous Infusions ◽  
Surgical Methods

The author warmly recommends treatment of sepsis with intravenous infusions of 1% calcium chloride solution (the method first proposed by Prof. Aleksinski). Although this method does not seem sufficiently theoretically justified, nevertheless P., on the basis of his observations, advises to resort to intravenous infusions of 1% solution of crystalline calcium chloride at 250-400 cfu for acute and subacute forms of sepsis where the well-known surgical methods of dissection and removal of infected nidi have been carried to completion and have not put the patient on the path to recovery.

scholarly journals Kestabilan Model Matematika Infeksi Primer Penyakit Varicella Dan Infeksi Rekuren Penyakit Herpes Zoster Oleh Virus Varicella Zoster

2020 ◽  
Vol 17 (1) ◽  
pp. 82-91
Author(s):  
Hardiyanti ◽  
R Ratianingsih ◽  
Hajar
Keyword(s):  
Herpes Zoster ◽  
Stability Analysis ◽  
Varicella Zoster Virus ◽  
Critical Point ◽  
Postherpetic Neuralgia ◽  
Primary Infection ◽  
Skin Diseases ◽  
Varicella Zoster ◽  
Stable Conditions ◽  
Disease Free

Varicella and herpes zoster are two infectious skin diseases of human that caused by varicella zoster virus, where varicella disease is a primary infection that often infected younger people while herpes zoster disease is a recurrent disease that often infected older people because of reactivation of latent varicella-zoster virus. If the pain caused by herpes zoster after recurrent phase is a appeared then the condition is known as postherpetic neuralgia. This study builds a mathematical model of primary infection (varicella disease) and recurrent infection (herpes zoster disease) developed from the SIR model (Susceptible, Infected, Recovered). The human population is divided into seven subpopulations, namely susceptible, infection, recovered of varicella, herpes zoster and postherpetic neuralgia subpopulation. Stability analysis at the critical point by linearization method gives a critical point 𝑇1 that guaranted to exist and unstable if 𝛼 𝜇(𝛽1+𝜇) 𝐴 , while the critical point 𝑇1 does not have any reqruitment. Stability analysis at the endemic disease-free critical point is represented 𝑇1 that will be unstable if 𝑇2 exist and stable 𝑇1 if 𝑇2 exist. Numerical simulations by simulated to describe such temporary disease-free conditions and an endemic stable conditions.

Treatment of acute gonorrheal adnexitis

1927 ◽  
Vol 23 (5) ◽  
pp. 593-593
Keyword(s):  
Calcium Chloride ◽  
Chloride Solution ◽  
Calcium Chloride Solution ◽  
Intravenous Injections ◽  
Anti Inflammatory

The author recommends for this purpose intravenous injections of afenil (10% calcium chloride solution by Knoll) in a dose of 10 cc. Performing these injections once a day, it is possible, in acute and subacute gonorrhoids salpingo-oophoritis, in 8 - 12 days to obtain such results, such with conventional anti-inflammatory treatment are obtained only in 3-4 weeks, and doing injections on 2 times a day, it is possible already in 2-3 days to stop "acute adnexitis.

scholarly journals RIGOR MORTIS AND THE INFLUENCE OF CALCIUM AND MAGNESIUM SALTS UPON ITS DEVELOPMENT

1908 ◽  
Vol 10 (1) ◽  
pp. 45-77 ◽  
Author(s):  
S. J. Meltzer ◽  
John Auer
Keyword(s):  
The Other ◽  
Original Position ◽  
Rigor Mortis ◽  
Calcium Salts ◽  
Intravenous Injections ◽  
Magnesium Salts ◽  
Calcium And Magnesium ◽  
Ion Effects ◽  
The One ◽  
The Right

Calcium salts hasten and magnesium salts retard the development of rigor mortis, that is, when these salts are administered subcutaneously or intravenously. When injected intra-arterially, concentrated solutions of both kinds of salts cause nearly an immediate onset of a strong stiffness of the muscles which is apparently a contraction, brought on by a stimulation caused by these salts and due to osmosis. This contraction, if strong, passes over without a relaxation into a real rigor. This form of rigor may be classed as work-rigor (Arbeitsstarre). In animals, at least in frogs, with intact cords, the early contraction and the following rigor are stronger than in animals with destroyed cord. If M/8 solutions—nearly equimolecular to "physiological" solutions of sodium chloride—are used, even when injected intra-arterially, calcium salts hasten and magnesium salts retard the onset of rigor. The hastening and retardation in this case as well as in the cases of subcutaneous and intravenous injections, are ion effects and essentially due to the cations, calcium and magnesium. In the rigor hastened by calcium the effects of the extensor muscles mostly prevail; in the rigor following magnesium injection, on the other hand, either the flexor muscles prevail or the muscles become stiff in the original position of the animal at death. There seems to be no difference in the degree of stiffness in the final rigor, only the onset and development of the rigor is hastened in the case of the one salt and retarded in the other. Calcium hastens also the development of heat rigor. No positive facts were obtained with regard to the effect of magnesium upon heat vigor. Calcium also hastens and magnesium retards the onset of rigor in the left ventricle of the heart. No definite data were gathered with regard to the effects of these salts upon the right ventricle.

The Role of Apoptosis in the Pathogenesis of Dermatitis Herpetiformis

2005 ◽  
Vol 18 (4) ◽  
pp. 691-699 ◽  
Author(s):  
M. Caproni ◽  
D. Torchia ◽  
E. Antiga ◽  
D. Degl'Innocenti ◽  
E. Barletta ◽  
...  
Keyword(s):  
Fas Ligand ◽  
Skin Diseases ◽  
Dermatitis Herpetiformis ◽  
Basal Layer ◽  
Immunohistochemical Analysis ◽  
Skin Lesions ◽  
Terminal Deoxynucleotidyl Transferase ◽  
Cutaneous Lesions ◽  
Malignant Skin ◽  
Perilesional Skin

Apoptosis is a form of cell death that is claimed to be involved in a number of chronic inflammatory and malignant skin diseases. The aim of this study was to investigate whether apoptosis may contribute to the pathogenesis of epidermal changes in dermatitis herpetiformis (DH) and, in particular, whether certain apoptosis-related markers such as Bax, Bcl-2, Fas and Fas ligand (FasL) take part in this process. For the detection of apoptotic nuclei, terminal deoxynucleotidyl transferase-mediated deoxyuridine triphosphate nick end labelling technique (TUNEL) was employed on cryostat sections. Skin lesions from six and perilesional skin from four DH patients were stained with monoclonal antibodies to Bax, Bcl-2, Fas and FasL. The same evaluation was also performed on three patients affected by bullous pemphigoid (BP) and in two healthy donors. Using TUNEL technique, a remarkable increase in the apoptotic rate within the epidermal compartment was observed in DH and BP patients in comparison with normal controls. In our immunohistochemical analysis, Bax/Bcl-2 ratio was almost the same in the epidermis of perilesional/lesional DH, BP and healthy skin specimens. In DH and BP specimens both Bax and Bcl-2 proteins were increased in the dermal perivascular compartment. Fas showed a prevalently epidermal staining, both in DH and BP lesions, while FasL was distributed in perivascular and subjunctional dermis; some FasL+ cells infiltrated the DEJ and the basal layer of epidermis. This study allowed us to highlight conspicuous apoptotic phenomena in basal and suprabasal keratinocytes within lesional and perilesional skin of DH. We conclude that in DH, as well as in BP, apoptosis plays a role in the pathogenesis of cutaneous lesions in concert with other pathogenetic mechanisms.

Cytological studies of early stages of powdery mildew in barley and wheat. V. Effects of calcium on the infection of coleoptiles of barley by Erysiphe graminis hordei

1978 ◽  
Vol 56 (20) ◽  
pp. 2544-2549 ◽  
Author(s):  
Susumu Takamatsu ◽  
Hiroshi Ishizaki ◽  
Hitoshi Kunoh
Keyword(s):  
Powdery Mildew ◽  
Calcium Chloride ◽  
Calcium Nitrate ◽  
Infection Process ◽  
Erysiphe Graminis ◽  
Calcium Salts ◽  
Early Stages ◽  
Erysiphe Graminis Hordei ◽  
The Times ◽  
Chloride Treatment

Some effects of calcium salts on the infection process of Erysiphe graminis hordei in coleoptiles of barley were investigated. Calcium chloride enhanced the incidence of haustoria but depressed that of papillae. Calcium bromide and calcium nitrate likewise enhanced the incidence of haustoria. Calcium chloride treatment at varied times and with varied durations during incubation after inoculation showed that events occurring in coleoptiles between 9 and 13 h after inoculation were most affected by the treatment. This period included the times that appressoria matured, cytoplasm aggregated, papillae were produced, and haustoria were formed, and the effect of calcium might be associated with one or more of these events.

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