On the treatment of facial nerve paralysis by ionization through Elton mud

1929 ◽  
Vol 25 (5) ◽  
pp. 510-515
Author(s):  
A. M. Kozlova
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Qualitative Change ◽  
Term Treatment ◽  
Facial Nerve Paralysis ◽  
Electrical Excitability ◽  
Nerve Paralysis ◽  
The Third ◽  
Long Term Treatment

Facial paralysis can be divided into three groups on the basis of electrodiagnostic studies. The first group without a rebirth reaction, amenable to healing in 4-6 weeks. The second group of paralysis of the facial nerve gives, from the second week of the disease, according to Waller's law, a qualitative change in the electrical excitability of the muscles and nerve; these cases can be cured no earlier than three, four months, or even longer. The third group of paralysis with a complete rebirth reaction requires long-term treatment, more than a year; complete cure in these cases does not occur

scholarly journals On the treatment of facial nerve paralysis by ionization through Elton mud

1929 ◽  
Vol 25 (5) ◽  
pp. 510-515
Author(s):  
A. М. Kozlova
Keyword(s):  
Facial Nerve ◽  
Qualitative Change ◽  
Term Treatment ◽  
Facial Nerve Paralysis ◽  
Electrical Excitability ◽  
Nerve Paralysis ◽  
Complete Cure ◽  
The Third ◽  
Long Term Treatment

Paralysis of the facial nerve, based on electrodiagnostic studies, can be divided into three groups. The first group without a rebirth reaction, amenable to healing in 4-6 weeks. The second group of paralysis of the facial nerve gives, from the second week of the disease, according to Waller's law, a qualitative change in electrical excitability from muscles and nerves; these cases can be cured no earlier than three, four months, or even longer. The third group of paralysis with a complete rebirth reaction requires long-term treatment, more than a year; complete cure in these cases does not occur.

Facial Nerve Paralysis Induced by Herpes Simplex Virus in Mice: An Animal Model of Acute and Transient Facial Paralysis

1995 ◽  
Vol 104 (7) ◽  
pp. 574-581 ◽  
Author(s):  
Toshiaki Sugita ◽  
Yasuo Fujiwara ◽  
Shingo Murakami ◽  
Yoshinari Hirata ◽  
Naoaki Yanagihara ◽  
...  
Keyword(s):  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
Facial Nerve ◽  
Facial Paralysis ◽  
Bell’S Palsy ◽  
Facial Nerve Paralysis ◽  
Bell's Palsy ◽  
Vacuolar Degeneration ◽  
Nerve Paralysis ◽  
Simplex Virus

We have been the first to succeed in producing an acute and transient facial paralysis simulating Bell's palsy, by inoculating herpes simplex virus into the auricles or tongues of mice. The KOS strain of the virus was injected into the auricle of 104 mice and the anterior two thirds of the tongue in 30 mice. Facial paralysis developed between 6 and 9 days after virus inoculation, continued for 3 to 7 days, and then recovered spontaneously. The animals were painlessly sacrificed between 6 and 20 days after inoculation for histopathologic and immunocytochemical study. Histopathologically, severe nerve swelling, inflammatory cell infiltration, and vacuolar degeneration were manifested in the affected facial nerve and nuclei. Herpes simplex virus antigens were also detected in the facial nerve, geniculate ganglion, and facial nerve nucleus. The pathophysiologic mechanisms of the facial paralysis are discussed in light of the histopathologic findings, in association with the causation of Bell's palsy.

Facial Nerve Paralysis with Benign Parotid Masses

1989 ◽  
Vol 98 (8) ◽  
pp. 644-647 ◽  
Author(s):  
Howard L. DeLozier ◽  
Martin J. Spinella ◽  
Glenn D. Johnson
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Case Reports ◽  
Facial Nerve Paralysis ◽  
Local Inflammation ◽  
Nerve Paralysis ◽  
Seventh Nerve ◽  
Rapid Enlargement ◽  
Pathologic Findings ◽  
Physical Findings

Facial paralysis in the presence of a parotid mass has been associated classically with a presumed diagnosis of malignancy. However, isolated case reports have documented the occurrence of paresis or paralysis secondary to pathologically benign, nonneurogenic parotid lesions. These previous cases have been reviewed and three additional cases are described. Comparisons are made on age, sex, symptoms, physical findings, pathologic findings, and prognosis. Involvement of the seventh nerve may be explained on the basis of compression, especially in association with local inflammation. Although facial paralysis still should be considered indicative of a malignancy, it also may be caused by benign masses, particularly those associated with rapid enlargement and/or infection.

Long-Term Prognosis of Profound Facial Nerve Paralysis Secondary to Acoustic Neuroma Resection

ORL ◽  
1999 ◽  
Vol 61 (2) ◽  
pp. 98-102 ◽  
Author(s):  
Takanobu Kunihiro ◽  
Jin Kanzaki ◽  
Ryuzo Shiobara ◽  
Yasuhiro Inoue ◽  
Kazuhiro Kurashima
Keyword(s):  
Facial Nerve ◽  
Acoustic Neuroma ◽  
Facial Nerve Paralysis ◽  
Nerve Paralysis ◽  
Long Term Prognosis

S236 – Novel Biomarker Use in the Evaluation of Facial Nerve Palsy

2008 ◽  
Vol 139 (2_suppl) ◽  
pp. P154-P154
Author(s):  
Carrie Flanagan ◽  
Shaw Gerry ◽  
Rick Odland
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Computer Software ◽  
Correlation Coefficients ◽  
Acute Onset ◽  
Facial Nerve Paralysis ◽  
Linear Regression Models ◽  
Serum Samples ◽  
Nerve Paralysis ◽  
Student’S T

Objectives 1) To determine whether a novel biomarker (hyperphosphorlyated neurofilament-heavy subunit) is able to be quantitated in patients with idiopathic facial nerve paralysis. 2) To determine whether correlation exists between biomarker quantitation and clinical parameters in patients with idiopathic facial nerve paralysis. Methods This is a prospective pilot study that assesses the utility of the serum biomarker hyperphosphorylated neurofilament-heavy subunit protein(NF-H) in evaluating patients with idiopathic facial nerve paralysis. From May 2006 to August 2007, 12 patients that presented to the Emergency, Neurology, and Otolaryngology departments at a county hospital with acute onset unilateral facial paralysis were enrolled into the study. Serum samples at the time of presentation were obtained, and the following additional data was recorded and analyzed: age, sex, severity of paralysis at presentation, duration between time of onset of paralysis and presentation, side of paresis, associated comorbidities, time to recovery, and ancillary testing results. Data analysis was performed using Student's T-test and analysis of variance; linear regression models and correlation coefficients were calculated using Microsoft Excel computer software. Results There was no significant relationship between patient age (r2=0.20), sex (p=0.19), and side of paralysis (p=0.49) and biomarker level. The biomarker level increased with increasing facial paresis severity (p=0.002, r2=0.17), and had a more pronounced correlation when patients presented within the first 24 to 48 hours of paralysis (p=0.006, r2=0.59). There was no correlation between biomarker and prognosis or ancillary testing results. Conclusions Hyperphosphorylated neurofilament-heavy subunit levels correlated with initial severity of facial paralysis, but were time-dependent.

S230 – MRI Findings in Delayed Facial Palsy after Stapedectomy

2008 ◽  
Vol 139 (2_suppl) ◽  
pp. P152-P152 ◽  
Author(s):  
Marc Cohen ◽  
Claudia Kirsch ◽  
Abie Mendelsohn ◽  
Akira Ishiyama
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Facial Palsy ◽  
Rare Complication ◽  
Internal Auditory Canal ◽  
Facial Nerve Paralysis ◽  
Mri Findings ◽  
Nerve Paralysis ◽  
Oral Corticosteroids ◽  
Magnetic Resonance Imaging Mri

Objectives 1) To retrospectively review the pathophysiology and radiologic features of delayed facial palsy after stapedectomy. 2) To discuss the management strategy of this unusual problem. Methods 450 stapedectomies performed at our institution were retrospectively reviewed from 1997 to 2007. A total of 2 patients in this series developed a delayed facial paralysis postoperatively. The clinical presentation, radiographic characteristics on magnetic resonance imaging (MRI), and the management of these patients are presented. Results A total of 2 patients out of 450 stapedectomies within a 10-year period developed the rare complication of delayed facial paralysis (postoperative days 12 and 41, respectively). Both patients developed rapidly progressive complete facial nerve paralysis preceded by periauricular pain and dysguesia. No sign of infection was present in the operated ear. MRI with gadolinium of the internal auditory canal demonstrated gadolinium enhancement of the labyrinthine portion of the facial nerve as seen in Bell's palsy. Both patients were treated with oral corticosteroids and antiviral medications. Subsequent imaging revealed decreased enhancement of the facial nerve with complete resolution of facial paralysis. Conclusions In the rare complication of delayed facial paralysis after stapedectomy, MR imaging is a useful diagnostic tool to dictate the treatment strategy. Delayed facial paralysis following stapedectomy can be treated medically when the MRI scan demonstrates enhancement of the facial nerve in the labyrinthine segment.

Acute suppurative parotitis and facial paralysis

1996 ◽  
Vol 110 (1) ◽  
pp. 91-92 ◽  
Author(s):  
Y. T. Pang ◽  
C. H. Raine
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Rare Complication ◽  
Facial Nerve Paralysis ◽  
Nerve Paralysis

AbstractA case of facial nerve paralysis secondary to acute suppurative parotitis is described. This is a rare complication in the absence of malignant processes in the parotid.

Long-term Outcomes After Middle Fossa Approach for Traumatic Facial Nerve Paralysis

2016 ◽  
Vol 37 (6) ◽  
pp. 799-804 ◽  
Author(s):  
Richard B. Cannon ◽  
Rhett S. Thomson ◽  
Clough Shelton ◽  
Richard K. Gurgel
Keyword(s):  
Facial Nerve ◽  
Facial Nerve Paralysis ◽  
Middle Fossa ◽  
Long Term Outcomes ◽  
Nerve Paralysis ◽  
Middle Fossa Approach

scholarly journals Rare Presentation of Rhino-Orbital-Cerebral Zygomycosis: Bilateral Facial Nerve Palsy

2011 ◽  
Vol 2011 ◽  
pp. 1-2 ◽  
Author(s):  
Alireza Mohebbi ◽  
Hesam Jahandideh ◽  
Ali Amini Harandi
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Nerve Palsy ◽  
Facial Nerve Palsy ◽  
Interesting Case ◽  
Facial Nerve Paralysis ◽  
Rare Presentation ◽  
Nerve Paralysis ◽  
Healthy Man ◽  
Immunocompromised Individual

Rhino-orbital-cerebral zygomycosis afflicts primarily diabetics and immunocompromised individual, but can also occur in normal hosts rarely. We here presented an interesting case of facial nerve palsy and multiple cold abscesses of neck due to rhino-orbital-cerebral zygomycosis in an otherwise healthy man. Although some reports of facial nerve paralysis in conjunction with rhino-orbital-cerebral zygomycosis exist, no case of bilateral complete facial paralysis has been reported in the literature to date.

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