scholarly journals Towards the modern doctrine of Landry paralysis

2020 ◽  
Vol VIII (1) ◽  
pp. 97-109
Author(s):  
A. E. Yanishevsky
Keyword(s):  
Nervous System ◽  
Clinical Picture ◽  
First Principle ◽  
Pathological Anatomy ◽  
Normal Anatomy ◽  
Nervous Disease ◽  
Nervous Systems ◽  
Anatomy And Physiology ◽  
Topical Diagnosis ◽  
Symptom Complex

Pri opredѣleni kazhdago nervnago zabolѣvanya, kak" otdѣl'noy nozologicheskoy yedinitsy, sovremennaya nevropatologya kladet" v" osnovu tri glavnykh" printsipa: anatomo-fizologichesky, patologo-anatomichesky i etologichesky. Na osnovani pervago printsipa nevropatolog", pol'zuyas' faktami normal'noy anatomi i fizologi nervnoy sistemy, opredѣlyayet" v" klinicheskoy kartinѣ zabolѣvanya uklonenya v" proyavleni dѣyatel'nosti otdѣl'nykh" nervnykh" sistem", nervnykh" yedinits"nevronov". Opredѣlyaya, kakya sistemy i kakya chasti ikh" porazheny, on" stavit" topicheskuyu dagnostiku zabolѣvanya. Na osnovani dannykh" patologicheskoy anatomi nevropatolog" opredѣlyayet" kharakter" porazhenya, lezhashchago v" osnovѣ zabolѣvanya, i nakonets" opredѣlyayet" prichinu, vyzvavshuyu dannoye zabolѣvane, obuslovivshuyu bystrotu ili medlennost' yego razvitya, pol'zuyas' v" ostrykh" sluchayakh" dannymi bakterologi i toksikologi. No ne smotrya na uspѣkhi nevrapotologi, sushchestvuyet" mnogo zabolѣvany, gdѣ vsѣ tri printsipa ne mogut" byt' strogo provedeny, tak" kak" predstavlyayut' mnogo yeshche spornago. Eti zabolѣvanya predstavlyayutsya simptomokompleksom", v" osnovѣ kotorago mogut" lezhat' porazhenya, chrezvychayno raznoobraznyya po svoyey lokalizatsi, po kharakteru patologo-anatomicheskago protsessa i po etologi. K" chislu podobnago roda zabolѣvany prinadlezhit" bolѣzn', izvѣstnaya pod" imenem" ostrago voskhodyashchago paralicha Landry. Развернуть 1371/5000 When defining each nervous disease, as a separate nosological unit, modern neuropathology lays in the basis of three main principles: anatomical-physiological, pathological-anatomical and ethiological. On the basis of the first principle, the neuropathologist, using the facts of normal anatomy and physiology of the nervous system, determines in the clinical picture of the disease, the evasion in the manifestation of the activity of individual nervous systems, nervous units - neurons. Determining which systems and which parts of them are affected, he sets the topical diagnosis of the disease. Based on the data of the pathological anatomy, the neuropathologist determines the nature of the lesion underlying the disease, and finally determines the cause that caused this disease, which caused the rapidity or slowness of its development, using the data of toxicology and toxicology in acute cases. But in spite of the successes of neuropathology, there are many diseases where all three principles cannot be strictly followed, since they represent a lot more controversial. These diseases are presented as a symptom complex, in the basis of which there may be lesions, extremely diverse in their localization, in the nature of the pathological-anatomical process and in ethiology. To the number of such kind of diseases belongs the disease, known under the name of acute ascending paralysis of Landry.

Theoretical and practical data on the use of mud in conjunction with some types of electrotherapy

1929 ◽  
Vol 25 (5) ◽  
pp. 574-574
Author(s):  
L. N. Klyachkin
Keyword(s):  
Spinal Cord ◽  
Nervous System ◽  
Peripheral Nervous System ◽  
Clinical Picture ◽  
Therapeutic Success ◽  
Radicular Symptom ◽  
Combined Use ◽  
Symptom Complex

SD Bassel (Kl. Med., 6, 1929) believes that the therapeutic success of mud cakes can be increased by the combined use of certain types of electrotherapy, which is feasible in an out-of-resort setting. Favorable results are obtained with skin scars and diseases of individual joints, as well as with forms of diseases of the spinal cord, where the meningeal-radicular symptom complex predominates in the clinical picture. Favorable results in a number of diseases of the peripheral nervous system

Vascular System

2017 ◽  
pp. 435-470
Author(s):  
Eduardo E. Benarroch ◽  
Jeremy K. Cutsforth-Gregory ◽  
Kelly D. Flemming
Keyword(s):  
Nervous System ◽  
Vascular System ◽  
Clinical Manifestations ◽  
Neural Tissue ◽  
Vascular Supply ◽  
Neural Function ◽  
Normal Anatomy ◽  
Anatomy And Physiology ◽  
System P ◽  
Pathologic Processes

The blood vessels to an organ provide it with a relatively constant supply of oxygen and other nutrients and a means for removal of metabolic waste. Failure to meet these vital requirements results in disease in that organ. Because of the unique structure and organization of the nervous system, localized abnormalities in its blood supply may produce devastating alterations in neural function. This chapter describes the normal anatomy and physiology of the vascular supply to neural tissue and the clinical manifestations of pathologic processes affecting this system.

scholarly journals RESPIRATORY AND NERVOUS SYSTEMS CENTRAL NERVOUS SYSTEM LESIONS DUE TO STREPTOCOCCUS PNEUMONIAE INFECTION IN HIV-POSITIVE PATIENTS

2013 ◽  
Vol 18 (4) ◽  
pp. 23-27
Author(s):  
V. V. Nikolenko ◽  
N. N. Vorobiova ◽  
L. M. Naumova ◽  
E. A Solodnikova ◽  
V. V. Bondarenko ◽  
...  
Keyword(s):  
Central Nervous System ◽  
Nervous System ◽  
Respiratory Failure ◽  
Pneumococcal Infection ◽  
Community Acquired Pneumonia ◽  
Hiv Positive ◽  
Laboratory Examination ◽  
Nervous Systems ◽  
Purulent Meningitis ◽  
Symptom Complex

Currently registration of community-acquired pneumonia and purulent meningitis caused by S. rneumoniae patients in HIV-positive patientsis is not carried out, there is absent the description of the clinical symptom complex. In this regard, the purpose of the work was the detection of the features of the course of pneumococcal infection with the lesion of the respiratory and nervous systems in HIV infection. A clinical and laboratory examination of 110 HIV-positive patients with community-acquired bacterialpneumonias andpurulent meningitises was performed. Community-acquiredpneumonia fulminates with the massive damage of the lung tissue, the presence ofpleural effusion, the early development of respiratory failure, decreased SaO2

scholarly journals Synapsin I (protein I), a nerve terminal-specific phosphoprotein. I. Its general distribution in synapses of the central and peripheral nervous system demonstrated by immunofluorescence in frozen and plastic sections.

1983 ◽  
Vol 96 (5) ◽  
pp. 1337-1354 ◽  
Author(s):  
P De Camilli ◽  
R Cameron ◽  
P Greengard
Keyword(s):  
Nervous System ◽  
Detailed Examination ◽  
Specific Protein ◽  
Nerve Cells ◽  
General Distribution ◽  
Synapsin I ◽  
Nervous Systems ◽  
Synaptic Region ◽  
Specific Localization ◽  
Peripheral Nervous Systems

Synapsin I (formerly referred to as protein I) is the collective name for two almost identical phosphoproteins, synapsin Ia and synapsin Ib (protein Ia and protein Ib), present in the nervous system. Synapsin I has previously been shown by immunoperoxidase studies (De Camilli, P., T. Ueda, F. E. Bloom, E. Battenberg, and P. Greengard, 1979, Proc. Natl. Acad. Sci. USA, 76:5977-5981; Bloom, F. E., T. Ueda, E. Battenberg, and P. Greengard, 1979, Proc. Natl. Acad. Sci. USA 76:5982-5986) to be a neuron-specific protein, present in both the central and peripheral nervous systems and concentrated in the synaptic region of nerve cells. In those preliminary studies, the occurrence of synapsin I could be demonstrated in only a portion of synapses. We have now carried out a detailed examination of the distribution of synapsin I immunoreactivity in the central and peripheral nervous systems. In this study we have attempted to maximize the level of resolution of immunohistochemical light microscopy images in order to estimate the proportion of immunoreactive synapses and to establish their precise distribution. Optimal results were obtained by the use of immunofluorescence in semithin sections (approximately 1 micron) prepared from Epon-embedded nonosmicated tissues after the Epon had been removed. Our results confirm the previous observations on the specific localization of synapsin I in nerve cells and synapses. In addition, the results strongly suggest that, with a few possible exceptions involving highly specialized neurons, all synapses contain synapsin I. Finally, immunocytochemical experiments indicate that synapsin I appearance in the various regions of the developing nervous system correlates topographically and temporally with the appearance of synapses. In two accompanying papers (De Camilli, P., S. M. Harris, Jr., W. B. Huttner, and P. Greengard, and Huttner, W. B., W. Schiebler, P. Greengard, and P. De Camilli, 1983, J. Cell Biol. 96:1355-1373 and 1374-1388, respectively), evidence is presented that synapsin I is specifically associated with synaptic vesicles in nerve endings.

scholarly journals Drosophila Stathmin: A Microtubule-destabilizing Factor Involved in Nervous System Formation

2002 ◽  
Vol 13 (2) ◽  
pp. 698-710 ◽  
Author(s):  
Sylvie Ozon ◽  
Antoine Guichet ◽  
Olivier Gavet ◽  
Siegfried Roth ◽  
André Sobel
Keyword(s):  
Nervous System ◽  
System Development ◽  
Nervous System Development ◽  
Microtubule Assembly ◽  
Nervous Systems ◽  
Germ Cell Migration ◽  
Numerous Cell ◽  
First Time

Stathmin is a ubiquitous regulatory phosphoprotein, the generic element of a family of neural phosphoproteins in vertebrates that possess the capacity to bind tubulin and interfere with microtubule dynamics. Although stathmin and the other proteins of the family have been associated with numerous cell regulations, their biological roles remain elusive, as in particular inactivation of the stathmin gene in the mouse resulted in no clear deleterious phenotype. We identified stathmin phosphoproteins inDrosophila, encoded by a unique gene sharing the intron/exon structure of the vertebrate stathmin andstathmin family genes. They interfere with microtubule assembly in vitro, and in vivo when expressed in HeLa cells. Drosophila stathmin expression is regulated during embryogenesis: it is high in the migrating germ cells and in the central and peripheral nervous systems, a pattern resembling that of mammalian stathmin. Furthermore, RNA interference inactivation ofDrosophila stathmin expression resulted in germ cell migration arrest at stage 14. It also induced important anomalies in nervous system development, such as loss of commissures and longitudinal connectives in the ventral cord, or abnormal chordotonal neuron organization. In conclusion, a single Drosophilagene encodes phosphoproteins homologous to the entire vertebrate stathmin family. We demonstrate for the first time their direct involvement in major biological processes such as development of the reproductive and nervous systems.

Sign in / Sign up

Export Citation Format

Share Document