scholarly journals Potrayal of Post-Traumatic Stress Diorder Among Flood Survivors

2021 ◽  
Vol 10 (1) ◽  
pp. 42-50
Author(s):  
Ernita Zakiah ◽  
Irma Rosalinda ◽  
Mauna

This study aims to look at the description of post traumatic stress disorder (PTSD) in flood survivors. PCL-C was used as an instrument to measure post traumatic stress disorder. This study used the PCL-C scale from Weathers and was developed by Solichah. Data analysis was performed used descriptive analysis techniques with the help of SPSS version 22. Based on the results of the analysis, it was found that subjects who experienced symptoms of post-traumatic stress disorder (PTSD) who were in the high category were 24.6%, 61.5% moderate, and low 13,8%. The implication of this study stated that individuals who have symptoms of PTSD after experiencing natural disaster need professional treatment to reduce their symptoms. Intervention is needed to make the individuals can overcome their obstacles and function better

2020 ◽  
Vol 49 (12) ◽  
pp. 785-789
Author(s):  
John Cooper ◽  
Andrea J Phelps ◽  
Chee H Ng ◽  
David Forbes

Background The COVID-19 pandemic has caused unprecedented stress globally, and the associated medical and health-related traumatic experiences pose significant risks for the development of post-traumatic stress disorder (PTSD), and the exacerbation of pre-existing PTSD, among patients, general practitioners (GPs) and healthcare staff. Objective The aim of this article is to provide guidance to GPs and healthcare staff working in Australia about the diagnosis and treatment of both newly developed and pre-existing PTSD in the COVID-19 context. Case studies are presented; the authors discuss whether pandemic-related PTSD is different to PTSD caused by different types of traumatic exposure, and the associated implications for treatment. Discussion The role of GPs in the management of PTSD during the COVID-19 pandemic remains central, involving early detection, assessment and referral. Moreover, health professionals are not immune to the mental health effects of the pandemic and are encouraged to maintain their wellbeing and to seek professional treatment if needed.


2020 ◽  
Author(s):  
Bart C.J. Dirven ◽  
Dewi van der Geugten ◽  
Miranda van Bodegom ◽  
Leonie Madder ◽  
Laura van Agen ◽  
...  

ABSTRACTPost-traumatic stress disorder (PTSD) is a psychiatric disorder vulnerable individuals can develop following a traumatic event, whereas others are resilient. Enhanced insight into the mechanistic underpinnings contributing to these inter-individual differences in PTSD susceptibility is key to improved treatment and prevention. Aberrant function of the hippocampal dentate gyrus (DG) may contribute to its psychopathology, with the dorsal DG potentially encoding trauma memory generalization and the ventral DG anxiety. Using a mouse model, we investigated the association between deviant DG structure and function and susceptibility to develop PTSD-like symptoms following trauma. Mice were exposed to a traumatic event (unpredictable, inescapable foot shocks) and tested for PTSD symptomatology following recovery. In three independent experiments, DG neuronal morphology, synaptic protein gene expression and neuronal activity during trauma encoding and recall were assessed. Behaviorally, PTSD-like animals displayed some increased anxiety-like behavior already prior to trauma, increased novelty-induced freezing, but no clear differences in remote trauma memory recall. Comparison of the ventral DG of PTSD-like vs resilient mice revealed lower spine density, reduced expression of the postsynaptic protein homer 1b/c gene, a larger population of neurons active during trauma encoding and a greater presence of somatostatin neurons to be associated with PTSD susceptibility. In contrast, the dorsal DG of PTSD-like animals did not differ in terms of spine density or gene expression, but displayed more active neurons during trauma encoding and a lower amount of somatostatin neurons. These data propose a critical role for -mainly the ventral-DG in establishing symptomatology addressed in this PTSD model.


Author(s):  
Aaron S. Heller

Post-traumatic stress disorder (PTSD) is associated with a host of neurobiological changes, including abnormalities in subcortical and cortical structure and function. The majority of neuroimaging studies have been motivated by a fear conditioning and extinction perspective to examine neural changes associated with PTSD. Several studies have found alterations in amygdala, hippocampal, and ventromedial prefrontal cortex. However, not all studies have replicated these findings. This suggests that more nuanced models of PTSD may be needed to account for the pathophysiology of the disorder. This chapter reviews neuroimaging findings related to this fear model and discusses additional considerations, including trauma type, age of trauma, and affective neurodynamics, that may help to account for the lack of consistent replications. Explicit consideration of these factors may facilitate greater coherence among studies going forward and advance our understanding of the neurobiological alterations associated with PTSD.


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