Flood Schemes, Flood Hazards, and Awareness in New Zealand

2019 ◽  
Author(s):  
Patrick Walsh ◽  
Ryan Paulik ◽  
Thomas Robertson
Keyword(s):  
Geosciences ◽  
2020 ◽  
Vol 10 (3) ◽  
pp. 114 ◽  
Author(s):  
Mark Quigley ◽  
Brendan Duffy

Earthquakes can influence flood hazards by altering the flux, volumes, and distributions of surface and/or subsurface waters and causing physical changes to natural and engineered environments (e.g., elevation, topographic relief, permeability) that affect surface and subsurface hydrologic regimes. This paper analyzes how earthquakes increased flood hazards in Christchurch, New Zealand, using empirical observations and seismological data. Between 4 September 2010 and 4 December 2017, this region hosted one moment magnitude (Mw) 7.1 earthquake, 3 earthquakes with Mw ≥ 6, and 31 earthquakes with local magnitude (ML) ≥ 5. Flooding related to liquefaction-induced groundwater pore-water fluid pressure perturbations and groundwater expulsion occurred in at least six earthquakes. Flooding related to shaking-induced ground deformations (e.g., subsidence) occurred in at least four earthquakes. Flooding related to tectonic deformations of the land surface (fault surface rupture and/or folding) occurred in at least two earthquakes. At least eight earthquakes caused damage to surface (e.g., buildings, bridges, roads) and subsurface (e.g., pipelines) infrastructure in areas of liquefaction and/or flooding. Severe liquefaction and associated groundwater-expulsion flooding in vulnerable sediments occurred at peak ground accelerations as low as 0.15 to 0.18 g (proportion of gravity). Expected return times of liquefaction-induced flooding in vulnerable sediments were estimated to be 100 to 500 years using the Christchurch seismic hazard curve, which is consistent with emerging evidence from paleo-liquefaction studies. Liquefaction-induced subsidence of 100 to 250 mm was estimated for 100-year peak ground acceleration return periods in parts of Christchurch.


1999 ◽  
Vol 190 ◽  
pp. 563-566
Author(s):  
J. D. Pritchard ◽  
W. Tobin ◽  
J. V. Clausen ◽  
E. F. Guinan ◽  
E. L. Fitzpatrick ◽  
...  

Our collaboration involves groups in Denmark, the U.S.A. Spain and of course New Zealand. Combining ground-based and satellite (IUEandHST) observations we aim to determine accurate and precise stellar fundamental parameters for the components of Magellanic Cloud Eclipsing Binaries as well as the distances to these systems and hence the parent galaxies themselves. This poster presents our latest progress.


Author(s):  
Ronald S. Weinstein ◽  
N. Scott McNutt

The Type I simple cold block device was described by Bullivant and Ames in 1966 and represented the product of the first successful effort to simplify the equipment required to do sophisticated freeze-cleave techniques. Bullivant, Weinstein and Someda described the Type II device which is a modification of the Type I device and was developed as a collaborative effort at the Massachusetts General Hospital and the University of Auckland, New Zealand. The modifications reduced specimen contamination and provided controlled specimen warming for heat-etching of fracture faces. We have now tested the Mass. General Hospital version of the Type II device (called the “Type II-MGH device”) on a wide variety of biological specimens and have established temperature and pressure curves for routine heat-etching with the device.


Author(s):  
Sidney D. Kobernick ◽  
Edna A. Elfont ◽  
Neddra L. Brooks

This cytochemical study was designed to investigate early metabolic changes in the aortic wall that might lead to or accompany development of atherosclerotic plaques in rabbits. The hypothesis that the primary cellular alteration leading to plaque formation might be due to changes in either carbohydrate or lipid metabolism led to histochemical studies that showed elevation of G-6-Pase in atherosclerotic plaques of rabbit aorta. This observation initiated the present investigation to determine how early in plaque formation and in which cells this change could be observed.Male New Zealand white rabbits of approximately 2000 kg consumed normal diets or diets containing 0.25 or 1.0 gm of cholesterol per day for 10, 50 and 90 days. Aortas were injected jin situ with glutaraldehyde fixative and dissected out. The plaques were identified, isolated, minced and fixed for not more than 10 minutes. Incubation and postfixation proceeded as described by Leskes and co-workers.


1998 ◽  
Vol 36 (5) ◽  
pp. 255-262
Author(s):  
SIMPANYA ◽  
JARVIS ◽  
BAXTER

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