Investigation of Soot Formation Near Flame-Wall Interaction Region in Rich Ethylene/Air Flames

2018 ◽  
Author(s):  
Ayush Jain ◽  
Yejun Wang ◽  
Waruna D. Kulatilaka
Keyword(s):  
Soot Formation ◽  
Interaction Region ◽  
Wall Interaction

Flame–Wall Interaction Effects on Diesel Post-injection Combustion and Soot Formation Processes

2019 ◽  
Vol 33 (8) ◽  
pp. 7759-7769 ◽  
Author(s):  
H. L. Yip ◽  
I. M. Rizwanul Fattah ◽  
A.C.Y. Yuen ◽  
W. Yang ◽  
P. R. Medwell ◽  
...  
Keyword(s):  
Soot Formation ◽  
Interaction Effects ◽  
Post Injection ◽  
Formation Processes ◽  
Wall Interaction

Effect of Spray/Wall Interaction on Diesel Combustion and Soot Formation in Two-Dimensional Piston Cavity

2013 ◽  
Author(s):  
Kuichun Li ◽  
Masaki Ido ◽  
Yoichi Ogata ◽  
Keiya Nishida ◽  
Baolu Shi ◽  
...  
Keyword(s):  
Soot Formation ◽  
Diesel Combustion ◽  
Two Dimensional ◽  
Wall Interaction

Multidimensional Modeling of Impinging Sprays on the Wall in Diesel Engines

1999 ◽  
Vol 52 (4) ◽  
pp. 119-138 ◽  
Author(s):  
J. Senda ◽  
H. G. Fujimoto
Keyword(s):  
Diesel Engines ◽  
Formation Process ◽  
Fuel Injection ◽  
Soot Formation ◽  
Film Formation ◽  
Combustion Process ◽  
Interaction Process ◽  
Droplet Breakup ◽  
Wall Surface ◽  
Wall Interaction

This article summarizes model analysis of the dispersion process of a Diesel spray on the wall surface in order to simulate the spray-wall interaction process in Diesel engines. The mixture formation process near the wall of the piston cavity affects the combustion process and the hydrocarbon or soot formation process through the quenching of the mixture and flame at the wall surface. In particular, mixture burning occurs mainly near the cavity wall through the whole combustion period in the case of high pressure fuel injection. In this article, representative modeling approaches on spray-wall interaction process including the film flow formation are summarized briefly. Then, our models of spray impingement for low/high-temperature models including the process of fuel film formation, film breakup, wall-drop/film heat transfer, and droplet breakup owing to the solid-liquid interface boiling are introduced with the comparison of experimental results. This review article includes 83 references.

Evidence for an Increased Generation of Prostacyclin in the Microvasculature and an Impairment of the Platelet α-Granule Release in Chronic Renal Failure

1988 ◽  
Vol 60 (02) ◽  
pp. 205-208 ◽  
Author(s):  
Paul A Kyrle ◽  
Felix Stockenhuber ◽  
Brigitte Brenner ◽  
Heinz Gössinger ◽  
Christian Korninger ◽  
...  
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Blood Clotting ◽  
Normal Subjects ◽  
Chronic Uraemia ◽  
Wall Interaction ◽  
End Stage ◽  
Granule Release

SummaryThe formation of prostacyclin (PGI2) and thromboxane A2 and the release of beta-thromboglobulin (beta-TG) at the site of platelet-vessel wall interaction, i.e. in blood emerging from a standardized injury of the micro vasculature made to determine bleeding time, was studied in patients with end-stage chronic renal failure undergoing regular haemodialysis and in normal subjects. In the uraemic patients, levels of 6-keto-prostaglandin F1α (6-keto-PGF1α) were 1.3-fold to 6.3-fold higher than the corresponding values in the control subjects indicating an increased PGI2 formation in chronic uraemia. Formation of thromboxane B2 (TxB2) at the site of plug formation in vivo and during whole blood clotting in vitro was similar in the uraemic subjects and in the normals excluding a major defect in platelet prostaglandin metabolism in chronic renal failure. Significantly smaller amounts of beta-TG were found in blood obtained from the site of vascular injury as well as after in vitro blood clotting in patients with chronic renal failure indicating an impairment of the a-granule release in chronic uraemia. We therefore conclude that the haemorrhagic diathesis commonly seen in patients with chronic renal failure is - at least partially - due to an acquired defect of the platelet a-granule release and an increased generation of PGI2 in the micro vasculature.

Further Evidence that Glycoprotein IIb-IIIa Mediates Platelet Spreading on Subendothelium

1991 ◽  
Vol 65 (02) ◽  
pp. 202-205 ◽  
Author(s):  
Harvey J Weiss ◽  
Vincet T Turitto ◽  
Hans R Baumgartner
Keyword(s):  
Platelet Adhesion ◽  
Surface Coverage ◽  
Rabbit Aorta ◽  
Conclusive Evidence ◽  
Initial Contact ◽  
Single Exposure ◽  
Vessel Segment ◽  
Multiple Exposures ◽  
Wall Interaction ◽  
Platelet Spreading

SummaryIn order to explore further the mechanism by which glycoprotein GPIIb-IIIa promotes platelet vessel wall interaction, platelet adhesion to subendothelium was studied in an annular chamber in which subendothelium from rabbit aorta was exposed at a shear rate of 2,600 s−1 to blood from patients with thrombasthenia. Perfusions were conducted for each of 5 exposure times (1 ,2,3, 5 and 10 min), and the percent surface coverage of the vessel segment with platelets in the contact (C) and spread (S) stage was determined. Increased values of platelet contact (C) were obtained in thrombasthenia at all exposure times; this finding is consistent with a defect in platelet spreadirg, based on a previously described kinetic model of platelet attachment to subendothelium. According to this model of attachment, increased values of platelet contact (C) at a single exposure time may be indicative of either a defect in spreading (S) or initial contact (C), but multiple exposures will result in increased contact only for defects which are related to defectiye platelet spreading (s).The results obtained over a broad range of exposure times provide more conclusive evidence that GPIIb-IIIa mediates platelet spreading than those previously obtained at single exposure times.

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