scholarly journals Reliability of venous gas embolism detection in the subclavian area for decompression stress assessment following scuba diving

2018 ◽  
Vol 48 (3) ◽  
pp. 132-140
Author(s):  
Julien Hugon ◽  
◽  
Asya Metelkina ◽  
Axel Barbaud ◽  
Ron Nishi ◽  
...  
2019 ◽  
Vol 49 (1) ◽  
pp. 61-63
Author(s):  
Romain Siaffa ◽  
◽  
Marc Luciani ◽  
Bruno Grandjean ◽  
Mathieu Coulange ◽  
...  

2019 ◽  
Vol 49 (1) ◽  
pp. 61-63 ◽  
Author(s):  
Romain Siaffa ◽  
◽  
Marc Luciani ◽  
Bruno Grandjean ◽  
Mathieu Coulange ◽  
...  

2019 ◽  
pp. 673-683
Author(s):  
Richard E. Moon ◽  

Gas can enter arteries (arterial gas embolism, AGE) due to alveolar-capillary disruption (caused by pulmonary over-pressurization, e.g. breath-hold ascent by divers) or veins (venous gas embolism, VGE) as a result of tissue bubble formation due to decompression (diving, altitude exposure) or during certain surgical procedures where capillary hydrostatic pressure at the incision site is subatmospheric. Both AGE and VGE can be caused by iatrogenic gas injection. AGE usually produces stroke-like manifestations, such as impaired consciousness, confusion, seizures and focal neurological deficits. Small amounts of VGE are often tolerated due to filtration by pulmonary capillaries; however VGE can cause pulmonary edema, cardiac “vapor lock” and AGE due to transpulmonary passage or right-to-left shunt through a patient foramen ovale. Intravascular gas can cause arterial obstruction or endothelial damage and secondary vasospasm and capillary leak. Vascular gas is frequently not visible with radiographic imaging, which should not be used to exclude the diagnosis of AGE. Isolated VGE usually requires no treatment; AGE treatment is similar to decompression sickness (DCS), with first aid oxygen then hyperbaric oxygen. Although cerebral AGE (CAGE) often causes intracranial hypertension, animal studies have failed to demonstrate a benefit of induced hypocapnia. An evidence-based review of adjunctive therapies is presented.


1997 ◽  
Vol 85 (6) ◽  
pp. 1367-1371 ◽  
Author(s):  
Claude Mann ◽  
Gilles Boccara ◽  
Veronique Grevy ◽  
Francis Navarro ◽  
Jean M. Fabre ◽  
...  

2011 ◽  
Vol 112 (2) ◽  
pp. 401-409 ◽  
Author(s):  
A. Møllerløkken ◽  
S. E. Gaustad ◽  
M. B. Havnes ◽  
C. R. Gutvik ◽  
A. Hjelde ◽  
...  

PEDIATRICS ◽  
1990 ◽  
Vol 85 (4) ◽  
pp. 593-594
Author(s):  
WAYNE R. RACKOFF ◽  
DAVID F. MERTON

Gas embolism to the portal venous system is a well-recognized radiographic sign in infants with necrotizing enterocolitis. It also has been seen after colonic irrigation with hydrogen peroxide solution.1,2 We present what we believe is the first reported patient with radiographic evidence of portal venous gas embolism after ingestion of hydrogen peroxide solution. This finding is important because gas embolism to the portal venous system after colonic irrigation with hydrogen peroxide has been associated with gangrenous and perforated bowel.1,2 CASE REPORT A 2-year-old boy ingested an unknown amount of 3% hydrogen peroxide solution. The child was found with foam around his mouth.


Anaesthesia ◽  
1996 ◽  
Vol 51 (11) ◽  
pp. 683-684
Author(s):  
S. P. W. Neff ◽  
L. Zulueta ◽  
R. Miller

2013 ◽  
Vol 114 (5) ◽  
pp. 602-610 ◽  
Author(s):  
Nico A. M. Schellart ◽  
Tjeerd P. van Rees Vellinga ◽  
Rob A. van Hulst

For over a century, studies on body fat (BF) in decompression sickness and venous gas embolism of divers have been inconsistent. A major problem is that age, BF, and maximal oxygen consumption (V̇o2max) show high multicollinearity. Using the Bühlmann model with eight parallel compartments, preceded by a blood compartment in series, nitrogen tensions and loads were calculated with a 40 min/3.1 bar (absolute) profile. Compared with Haldanian models, the new model showed a substantial delay in N2 uptake and (especially) release. One hour after surfacing, an increase of 14–28% in BF resulted in a whole body increase of the N2 load of 51%, but in only 15% in the blood compartment. This would result in an increase in the bubble grade of only 0.01 Kisman-Masurel (KM) units at the scale near KM = I−. This outcome was tested indirectly by a dry dive simulation (air breathing) with 53 male divers with a small range in age and V̇o2max to suppress multicollinearity. BF was determined with the four-skinfold method. Precordial Doppler bubble grades determined at 40, 80, 120, and 160 min after surfacing were used to calculate the Kisman Integrated Severity Score and were also transformed to the logarithm of the number of bubbles/cm2 (logB). The highest of the four scores yielded logB = −1.78, equivalent to KM = I−. All statistical outcomes of partial correlations with BF were nonsignificant. These results support the model outcomes. Although this and our previous study suggest that BF does not influence venous gas embolism (Schellart NAM, van Rees Vellinga TP, van Dijk FH, Sterk W. Aviat Space Environ Med 83: 951–957, 2012), more studies with different profiles under various conditions are needed to establish whether BF remains (together with age and V̇o2max) a basic physical characteristic or will become less important for the medical examination and for risk assessment.


2013 ◽  
Vol 115 (5) ◽  
pp. 716-722 ◽  
Author(s):  
Dennis Madden ◽  
Mislav Lozo ◽  
Zeljko Dujic ◽  
Marko Ljubkovic

Arterialization of gas bubbles after decompression from scuba diving has traditionally been associated with pulmonary barotraumas or cardiac defects, such as the patent foramen ovale. Recent studies have demonstrated the right-to-left passage of bubbles through intrapulmonary arterial-venous anastamoses (IPAVA) that allow blood to bypass the pulmonary microcirculation. These passages open up during exercise, and the aim of this study is to see if exercise in a postdiving period increases the incidence of arterialization. After completing a dive to 18 m for 47 min, patent foramen ovale-negative subjects were monitored via transthoracic echocardiography, within 10 min after surfacing, for bubble score at rest. Subjects then completed an incremental cycle ergometry test to exhaustion under continuous transthoracic echocardiography observation. Exercise was suspended if arterialization was observed and resumed when the arterialization cleared. If arterialization was observed a second time, exercise was terminated, and oxygen was administered. Out of 23 subjects, 3 arterialized at rest, 12 arterialized with exercise, and 8 did not arterialize at all even during maximal exercise. The time for arterialization to clear with oxygen was significantly shorter than without. Exercise after diving increased the incidence of arterialization from 13% at rest to 52%. This study shows that individuals are capable of arterializing through IPAVA, and that the intensity at which these open varies by individual. Basic activities associated with SCUBA diving, such as surface swimming or walking with heavy equipment, may be enough to allow the passage of venous gas emboli through IPAVA.


Sign in / Sign up

Export Citation Format

Share Document