Vein of Galen aneurysmal malformations associated with high output cardiac failure: Report of three autopsy cases

2015 ◽  
Vol 09 (02) ◽  
pp. 227-232
Author(s):  
Reenu Malhotra ◽  
Luisa Florez ◽  
Dollett White ◽  
Sozos Papasozomenos ◽  
Michael Covinsky ◽  
...  
2007 ◽  
Vol 21 (5) ◽  
Author(s):  
Reenu Kapur Malhotra ◽  
Luisa Florez ◽  
Dollett White ◽  
Sozos Papasozomenos ◽  
Michael Covinsky ◽  
...  

2018 ◽  
Vol 05 (03) ◽  
pp. 198-200
Author(s):  
Ashutosh Kumar ◽  
Neeraja Ajayan ◽  
Manikandan Sethuraman ◽  
Ajay Prasad Hrishi

AbstractWeaning of patients with vein of Galen malformations (VOGM) from mechanical ventilation can be challenging in the postprocedure period due to underlying high-output cardiac failure and the fluid overload caused by the neurointervention procedure. We present the case of a neonate with VOGM who was refractory to multiple weaning attempts from mechanical ventilation and was successfully managed with noninvasive ventilation (NIV). NIV can be safely used as a method of weaning, post mechanical ventilation in this subset of patients. The use of NIV will facilitate successful weaning and reduce the incidence of extubation failure in neonates with VOGM in cardiac failure.


2016 ◽  
Vol 125 (3) ◽  
pp. 597-597 ◽  
Author(s):  
Ashley Smith ◽  
Todd Abruzzo ◽  
Mohamed Mahmoud

Author(s):  
Reenu Kapur Malhotra ◽  
Luisa Florez ◽  
Dollett White ◽  
Sozos Papasozomenos ◽  
Michael Covinsky ◽  
...  

Author(s):  
D. Abrahan ◽  
N.K. Yeaney ◽  
M.A. Hamzah ◽  
M.D. Bain ◽  
H.K. Najm ◽  
...  

BACKGROUND: The vein of Galen aneurysm (VGAM) is the most common type of arteriovenous malformation in the neonate. These neonates commonly present with high output cardiac failure that may be associated with pulmonary hypertension. The medical management and stabilization of these neonates can be challenging before staged transarterial embolization of the aneurysm is undertaken. CASE: A 2.34 kilogram neonate, antenatally diagnosed to have VGAM, was born at 36 weeks of gestation for fetal distress. The neonate failed to respond to medical management including inotropes, high frequency mechanical ventilation and inhaled nitric oxide. The patient’s high-output heart failure and persistent pulmonary hypertension were stabilized with veno-arterial extra-corporeal membrane oxygenation (VA-ECMO) using central cannulation. Further transarterial staged embolization of the VGAM was undertaken on VA-ECMO support. CONCLUSION: There may be a role of VA-ECMO using central cannulation to optimize management of high output cardiac failure and persistent pulmonary hypertension in neonatal VGAM patients who fail medical management to facilitate staged transarterial embolization of the VGAM.


Neurosurgery ◽  
1988 ◽  
Vol 22 (5) ◽  
pp. 908-910 ◽  
Author(s):  
Jane Matjasko ◽  
Walker Robinson ◽  
Daniel Eudaily

Abstract A 12-day-old infant in intractable cardiac failure due to a vein of Galen malformation was treated successfully with serial ligation of the majority of the vessels feeding the malformation. Despite some residual vascular supply to the malformation, the congestive heart failure has disappeared and growth and development have been normal over a 3-year follow-up period.


2009 ◽  
Vol 19 (5) ◽  
pp. 530-533 ◽  
Author(s):  
Sigrun R. Hofmann ◽  
Matthias Weise ◽  
Katharina I. Nitzsche

AbstractCongenital arteriovenous malformations are rare causes of congestive cardiac failure in neonates. The most common sites are in the head and liver, but other sites include the thorax, the abdomen and the limbs. The onset of failure is usually not in the immediate neonatal period, but later on in life, albeit that lesions such as the arteriovenous malformation of the vein of Galen, and other arteriovenous malformations in different locations which produce high flow can present early. We describe here the first case, to the best of our knowledge, of prenatal detection of an intrathoracic arteriovenous malformation producing neonatal cardiac failure, which was successfully treated by surgery postnatally.


2018 ◽  
Vol 71 (6) ◽  
pp. 896-903
Author(s):  
Amtul Aala ◽  
Sairah Sharif ◽  
Leslie Parikh ◽  
Paul C. Gordon ◽  
Susie L. Hu

PEDIATRICS ◽  
1972 ◽  
Vol 49 (4) ◽  
pp. 563-573
Author(s):  
Robert H. McLean ◽  
James H. Moller ◽  
Warren J. Warwick ◽  
Leon Satran ◽  
Russell V. Lucas

Multinodular hemangiomatosis of the liver (MHL) is characterized pathologically by multiple hemangiomas, primarily in the liver but also in other organs. The hepatic hemangiomas act as small arteriovenous fistulae and their combined effect may result in a massive peripheral arteriovenous shunt and high output congestive cardiac failure. Patients with MHL may be recognized clinically by the classical triad of congestive cardiac failure, hepatomegaly, and cutaneous hemangiomas. All but 2 of 31 cases of MHL herein reviewed had the onset of symptoms prior to 6 months of age. High output cardiac failure were severe and resulted in a 70% mortality. Hepatomegaly was massive and out of proportion to the degree of cardiac failure. Therapy includes intensive treatment of congestive cardiac failure and judicious transfusion in anemic patients. Irradiation of the liver and corticosteroids have been utilized in attempts to speed evolution of the hepatic arteriovenous fistulae. The data are not sufficient to establish the efficacy of either of these treatment modalities.


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