scholarly journals Congenital Bundle of His Tachycardia

2020 ◽  
Author(s):  
Keyword(s):  
Bundle Of His

Wilhelm His, Jr. and the Bundle of His

1949 ◽  
Vol IV (2) ◽  
pp. 170-187 ◽  
Author(s):  
T. H. BAST ◽  
WESTON D. GARDNER
Keyword(s):  
Bundle Of His

Drugs and pacemakers for complete heart block

1967 ◽  
Vol 5 (5) ◽  
pp. 19-20
Keyword(s):  
Myocardial Infarction ◽  
Heart Failure ◽  
Heart Rate ◽  
Heart Disease ◽  
Ischaemic Heart Disease ◽  
Heart Block ◽  
Complete Heart Block ◽  
Bundle Of His

Complete heart block can occur in ischaemic heart disease, and can acutely complicate myocardial infarction. Most other cases are associated with fibrosis of the bundle of His of unknown cause, or are congenital. In some patients with chronic heart block, especially the congenital type, adequate output is maintained. In other patients chronic or intermittent heart block may cause Stokes-Adams attacks, or heart failure may not respond to digitalis and diuretics until the heart rate is increased. These require treatment by drugs or, when this fails, by use of anartifical pacemaker.

The effect of destruction of the bundle of His or the atrioventricular node on cyclopropane–adrenaline cardiac arrhythmias

1970 ◽  
Vol 48 (4) ◽  
pp. 207-215 ◽  
Author(s):  
Betty I. Sasyniuk ◽  
Peter E. Dresel
Keyword(s):  
Coronary Artery ◽  
Cardiac Arrhythmias ◽  
Atrioventricular Node ◽  
Circumflex Coronary Artery ◽  
Selective Destruction ◽  
Anesthetized Dogs ◽  
Bundle Of His

Destruction of the bundle of His abolished bigeminal rhythms produced by adrenaline in thiopentone–cyclopropane anesthetized dogs. Bigeminal rhythms were still elicited, however, after selective destruction of the A–V node. Destruction of the bundle of His did not prevent multifocal arrhythmias but did increase the dose of adrenaline required to induce them. Multifocal arrhythmias produced after destruction of the bundle of His were abolished by injections of acetylcholine into the left anterior descending, but not the circumflex coronary artery, results opposite to those obtained in normal hearts.

Cardiomyocyte apoptosis in cocaine-induced myocarditis with involvement of bundle of His and left bundle branch

2006 ◽  
Vol 112 (1) ◽  
pp. 116-118 ◽  
Author(s):  
Gianluca Iacobellis ◽  
Walter Kemp
Keyword(s):  
Cardiomyocyte Apoptosis ◽  
Bundle Of His

CONGENITAL HEART BLOCK DUE TO A LESION IN THE CONDUCTION SYSTEM

PEDIATRICS ◽  
1970 ◽  
Vol 45 (4) ◽  
pp. 640-650
Author(s):  
Cirilo Sotelo-Avila ◽  
Harvey S. Rosenberg ◽  
Dan G. McNamara
Keyword(s):  
Heart Block ◽  
Congenital Heart ◽  
Complete Heart Block ◽  
Fibrous Tissue ◽  
Postmortem Examination ◽  
Conduction System ◽  
Cardiac Conduction ◽  
Fibrous Body ◽  
Congenital Complete Heart Block ◽  
Bundle Of His

The cardiac conduction system of an infant with congenital complete heart block was studied at postmortem examination by subserial sections of the interatrial and interventricular septa. A localized area of necrosis in the central fibrous body interrupted the penetrating portion of the bundle of His. The cardiac structure was intact, with no abnormality other than the lesions in the fibrous tissue. The lesion is interpreted as the result of an acquired inflammation late in intrauterine life, with damage localized to the cardiac central fibrous body.

Re-evaluation of the structure of the atrioventricular node and its connections with the atrium

EP Europace ◽  
2020 ◽  
Vol 22 (5) ◽  
pp. 821-830 ◽  
Author(s):  
Robert H Anderson ◽  
Damian Sanchez-Quintana ◽  
Shumpei Mori ◽  
Jose Angel Cabrera ◽  
Eduardo Back Sternick
Keyword(s):  
Atrioventricular Node ◽  
Atrial Septum ◽  
Atrioventricular Conduction ◽  
Marked Variation ◽  
Compelling Evidence ◽  
Serial Sections ◽  
Atrial Myocardium ◽  
Cavotricuspid Isthmus ◽  
Fibrous Body ◽  
Bundle Of His

Abstract Aims The anatomic substrates for atrioventricular nodal re-entry remain enigmatic, but require knowledge of the normal arrangement of the inputs and exist from the atrioventricular node. This knowledge is crucial to understand the phenomenon of atrioventricular nodal re-entry. Methods and results We studied 20 human hearts with serial sections covering the entirety of the triangle of Koch and the cavotricuspid isthmus. We determined the location of the atrioventricular conduction axis and the connections between the specialized cardiomyocytes of the conduction axis and the adjacent working atrial myocardium. The atrioventricular node was found at the apex of the triangle of Koch, with entry of the conduction axis to the central fibrous body providing the criterion for distinction of the bundle of His. We found marked variation in the inferior extensions of the node, the shape of the node, the presence or absence of a connecting bridge within the myocardium of the cavotricuspid isthmus, the connections between the compact node and the myocardium of the atrial septum, the presence of transitional cardiomyocytes, and the ‘last’ connection between the working atrial myocardium and the conduction axis before it became the bundle of His. Conclusion The observed variations of the inferior extensions, combined with the arrangement of the ‘last’ connections between the atrial myocardium and the conduction axis prior to its insulation as the bundle of His, provide compelling evidence to support the concept for atrioventricular nodal re-entry as advanced by Katritsis and Becker.

Mechanism of atrioventricular nodal facilitation in the rabbit heart: role of the distal AV node

1997 ◽  
Vol 272 (6) ◽  
pp. H2815-H2825 ◽  
Author(s):  
G. J. Fahy ◽  
I. Efimov ◽  
Y. Cheng ◽  
G. A. Kidwell ◽  
D. Van Wagoner ◽  
...  
Keyword(s):  
Action Potential ◽  
Action Potential Duration ◽  
Cellular Response ◽  
Rabbit Heart ◽  
Av Node ◽  
Microelectrode Recordings ◽  
Bundle Of His ◽  
Action Potential Shortening

We investigated whether atrioventricular (AV) nodal facilitation is the result of distal AV nodal action potential shortening. Atrial and bundle of His (H) electrograms and microelectrode recordings from proximal and distal AV nodal cells were analyzed in eight superfused rabbit AV node preparations in response to two pacing protocols. In the facilitation protocol, an atrial extrastimulus (A3) was preceded by an atrial impulse (A2) introduced 300, 200, 150, or 125 ms after 30 basic beats (A1). The preexcitation protocol differed from the facilitation protocol by the addition of a premature His depolarization (h2) such that the H1-h2 interval was shorter than the H1-H2 interval. Conduction curves (A3-H3 vs. H2-A3, h2-A3, and A2-A3 intervals) were constructed. Facilitation was demonstrated in all preparations when H2-A3 was used (P = 0.02) but not in the A2-A3 format. Compared with facilitation at the same A1-A2 intervals, preexcitation, despite shortening the distal cellular action potential duration, resulted in longer A3-H3 delays (P = 0.002), shorter A2-A3 intervals, and depression of the proximal nodal cellular response. Thus facilitation does not result from altered distal AV nodal characteristics and instead is a manifestation of an uncontrolled pacing protocol-dependent modulation of proximal AV nodal function.

Noninvasive methods for recording bundle of his activity

1974 ◽  
Vol 33 (3) ◽  
pp. 444-445 ◽  
Author(s):  
Anthony N. Damato
Keyword(s):  
Noninvasive Methods ◽  
Bundle Of His

Propagation of impulses through the atrioventricular node

1959 ◽  
Vol 197 (6) ◽  
pp. 1171-1174 ◽  
Author(s):  
Jesús Alanís ◽  
Enrique López ◽  
Juan J. Mandoki ◽  
Guillermo Pilar
Keyword(s):  
Action Potential ◽  
Atrioventricular Node ◽  
The Other ◽  
Temporal Course ◽  
Bundle Of His ◽  
Experimental Findings

Records taken from the bundle of His and A-V node region of isolated and perfused dogs' hearts showed an action potential (N potential), which appeared after the auricular response and preceding the electrogram of the bundle of His (H potential). The H potential may disappear without affecting the N potential and the latter may be abolished while the auricular electrogram is still present. These observations permit the interpretation that the N potential is generated independently. The temporal course and the time of occurrence of the N potential, indicate that it represents the activity of the A-V node. The N potential divides the A-H interval into A-N and N-H latencies. Progressively increasing frequencies of activation of the auricle lengthened first the N-H latency until the H potential disappeared. Higher frequencies lengthened the A-N latency until the N potential also disappeared. In order to explain the experimental findings, it is suggested that there are two functional discontinuities, one located between the A-V node and the bundle of His, and the other between the atrium and the A-V node; the first is the most vulnerable for propagation.

scholarly journals Comparison of sinoventricular conduction in children and adults using bundle of His electrograms.

Heart ◽  
1973 ◽  
Vol 35 (5) ◽  
pp. 507-515 ◽  
Author(s):  
S Bekheit ◽  
P Morton ◽  
J G Murtagh ◽  
E Fletcher
Keyword(s):  
Bundle Of His
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