scholarly journals A Case of Renal Cortical Necrosis in a 15-year-old Boy with Acute Kidney Injury

2019 ◽  
Vol 23 (1) ◽  
pp. 53-57
Author(s):  
Mi-ji Lee ◽  
Hyung Eun Yim ◽  
Kee Hwan Yoo
Renal Failure ◽  
2015 ◽  
Vol 37 (7) ◽  
pp. 1185-1189 ◽  
Author(s):  
Jai Prakash ◽  
Pragya Pant ◽  
Anil K. Singh ◽  
Shashidhar Sriniwas ◽  
Vijay P. Singh ◽  
...  

2012 ◽  
Vol 111 (5) ◽  
pp. 2213-2216 ◽  
Author(s):  
Vivek B. Kute ◽  
Aruna V. Vanikar ◽  
Pramod P. Ghuge ◽  
Jitendra G. Goswami ◽  
Mohan P. Patel ◽  
...  

Author(s):  
Duska Dragun ◽  
Björn Hegner

Any kind of acute renal deterioration that occurs in young women may, besides typical pregnancy-related disorders, account for pregnancy-related acute kidney injury (PR-AKI). Incidence of PR-AKI is continuously decreasing, yet still represents a significant cause of fetomaternal morbidity and mortality. Hyperemesis gravidarum causing volume depletion and septic shock with renal cortical necrosis upon septic abortion are major causes of PR-AKI during early pregnancy. Pre-eclampsia and bleeding complications associated with placental abruption or other causes of obstetric haemorrhage are responsible for the majority of cases during late pregnancy (after week 35) and puerperium. Haemolytic uraemic syndrome and thrombotic thrombocytopenic purpura disorders are less common than pre-eclampsia, yet represent a diagnostic and therapeutic challenge due to similar features to severe pre-eclampsia cases.


2019 ◽  
Vol 30 (4) ◽  
pp. 960
Author(s):  
KonapurAnanth Rao ◽  
RanjithK Nair ◽  
D Mukherjee ◽  
Bhaskar Datt ◽  
Sourabh Sharma ◽  
...  

2017 ◽  
Vol 2017 ◽  
pp. 1-3 ◽  
Author(s):  
Ying Mao Gn ◽  
Arvind Ponnusamy ◽  
Vikram Thimma

Renal complications from snakebite result in high mortality and morbidity. Acute kidney injury (AKI) occurs in 5–30% of cases. Renal manifestation could include acute tubular necrosis, cortical necrosis, interstitial nephritis, glomerulonephritis, and vasculitis. We present a case of thrombotic microangiopathy (TMA) resulting in renal cortical necrosis. Renal biopsy showed fibrin thrombi in glomeruli and arterioles with cortical necrosis. Our patient progressed to end-stage renal disease.


Author(s):  
Ashwani Kumar ◽  
Vivek Mohanty ◽  
Nitin Kumar ◽  
Gaurav Kanra ◽  
Anirudhh Mukherjee

Organophosphorus poisoning in the form of insecticides and pesticides acts by inhibition of anticholinesterase activity at the neuromuscular junction leading to overstimulation of the nicotinic and muscarinic receptor. Renal involvement usually manifests as increased urination, urinary incontinence, strangury due to overstimulation of urinary bladder . Acute kidney injury in the form of acute cortical necrosis is an extremely rare complication of OP poisoning. Here we highlight a case 31 year old male who presented to Emergency with Dichlorvos poisoning with anuria due to cortical necrosis requiring hemodialysis with gradually improving renal function thus representing an uncommon complication of Organophosphorus poisoning thus making it as a rare and interesting presentation. Keywords: Organophosphorus poisoning, Acute cortical necrosis, Granular casts


Renal Failure ◽  
2012 ◽  
Vol 35 (1) ◽  
pp. 170-172 ◽  
Author(s):  
Vivek Kumar ◽  
Ritambhra Nada ◽  
Sravan Kumar ◽  
Raja Ramachandran ◽  
Manish Rathi ◽  
...  

2011 ◽  
Vol 2011 ◽  
pp. 1-3 ◽  
Author(s):  
C. Kennedy ◽  
S. Khilji ◽  
A. Dorman ◽  
J. Walshe

Bacterial meningitis is a relatively common infection of the cerebrospinal fluid (CSF) and leptomeninges. The clinical picture evolves rapidly and, if treatment is delayed, can result in a variety of long-term sequelae, including death. Acute kidney injury in the setting of bacterial meningitis usually results from hypotension and volume depletion and resolves with appropriate treatment. Meningococcaemia with profound hypotension, and/or disseminated intravascular coagulopathy (DIC) may very rarely lead to bilateral renal cortical necrosis. In this context, renal recovery is extremely unlikely. We present two cases of meningococcaemia complicated by bilateral renal cortical necrosis and, ultimately, end stage kidney disease. We also present a review of the literature on the subject. The cases outline the importance of early aggressive intervention by a multidisciplinary team.


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