scholarly journals Expression, Localization, and Effect of High Salt Intake on Electroneutral Na+/HCO3– Cotransporter NBCn2 in Rat Small Intestine: Implication in Intestinal NaCl Absorption

2019 ◽  
Vol 10 ◽  
Author(s):  
Jin-Lin Wang ◽  
Lei Zhao ◽  
Jun Zhu ◽  
Deng-Ke Wang ◽  
Mei-Juan Ren ◽  
...  
Keyword(s):  
Small Intestine ◽  
Salt Intake ◽  
High Salt ◽  
Rat Small Intestine ◽  
Nacl Absorption ◽  
High Salt Intake

scholarly journals High Salt Intake and Stroke: Meta-analysis of the Epidemiologic Evidence

2012 ◽  
Vol 18 (8) ◽  
pp. 691-701 ◽  
Author(s):  
Xiu-Yang Li ◽  
Xian-Lei Cai ◽  
Ping-Da Bian ◽  
Liu-Ru Hu
Keyword(s):  
Salt Intake ◽  
Meta Analysis ◽  
High Salt ◽  
Epidemiologic Evidence ◽  
High Salt Intake

scholarly journals Hypoxia-Inducible Factor Prolyl-Hydroxylase 2 Senses High-Salt Intake to Increase Hypoxia Inducible Factor 1α Levels in the Renal Medulla

Hypertension ◽  
2010 ◽  
Vol 55 (5) ◽  
pp. 1129-1136 ◽  
Author(s):  
Zhengchao Wang ◽  
Qing Zhu ◽  
Min Xia ◽  
Pin-Lan Li ◽  
Shante J. Hinton ◽  
...  
Keyword(s):  
Salt Intake ◽  
Hypoxia Inducible Factor ◽  
Renal Medulla ◽  
Prolyl Hydroxylase ◽  
High Salt ◽  
Hypoxia Inducible Factor 1Α ◽  
High Salt Intake

scholarly journals Differential responses to salt supplementation in adult male and female rat adrenal glands following intrauterine growth restriction

2011 ◽  
Vol 209 (1) ◽  
pp. 85-94 ◽  
Author(s):  
Karine Bibeau ◽  
Mélissa Otis ◽  
Jean St-Louis ◽  
Nicole Gallo-Payet ◽  
Michèle Brochu
Keyword(s):  
Protein Expression ◽  
Adrenal Glands ◽  
Salt Intake ◽  
High Salt ◽  
Receptor Subtypes ◽  
Mrna Levels ◽  
Female Rat ◽  
Angiotensin Ii Receptor ◽  
Intrauterine Growth ◽  
High Salt Intake

In low sodium-induced intrauterine growth restricted (IUGR) rat, foetal adrenal steroidogenesis as well as the adult renin–angiotensin–aldosterone system (RAAS) is altered. The aim of the present study was to determine the expression of cytochrome P450 aldosterone synthase (P450aldo) and of angiotensin II receptor subtypes 1 (AT1R) and 2 (AT2R) in adult adrenal glands and whether this expression could be influenced by IUGR and by high-salt intake in a sex-specific manner. After 6 weeks of 0.9% NaCl supplementation, plasma renin activity, P450aldo expression and serum aldosterone levels were decreased in all groups. In males, IUGR induced an increase in AT1R, AT2R, and P450aldo levels, without changes in morphological appearance of the zona glomerulosa (ZG). By contrast, in females, IUGR had no effect on the expression of AT1R, but increased AT2R mRNA while decreasing protein expression of AT2R and P450aldo. In males, salt intake in IUGR rats reduced both AT1R mRNA and protein, while for AT2R, mRNA levels decreased whereas protein expression increased. In females, salt intake reduced ZG size in IUGR but had no affect on AT1R or AT2R expression in either group. These results indicate that, in response to IUGR and subsequently to salt intake, P450aldo, AT1R, and AT2R levels are differentially expressed in males and females. However, despite these adrenal changes, adult IUGR rats display adequate physiological and adrenal responses to high-salt intake, via RAAS inhibition, thus suggesting that extra-adrenal factors likely compensate for ZG alterations induced by IUGR.

scholarly journals High fat and/or high salt intake during pregnancy alters maternal meta-inflammation and offspring growth and metabolic profiles

2014 ◽  
Vol 2 (8) ◽  
pp. e12110 ◽  
Author(s):  
Clare M. Reynolds ◽  
Mark H. Vickers ◽  
Claudia J. Harrison ◽  
Stephanie A. Segovia ◽  
Clint Gray
Keyword(s):  
Salt Intake ◽  
High Salt ◽  
Metabolic Profiles ◽  
High Fat ◽  
High Salt Intake ◽  
Offspring Growth

Assessment of angiotensin receptor blockers for the hypertensive patients with high salt intake estimated with spot urine

2016 ◽  
Vol 10 (4) ◽  
pp. e32-e33
Author(s):  
Makiko Ogiku ◽  
Daisuke Matsumoto ◽  
Jyunpei Koike ◽  
Yusuke Tanaka ◽  
Nao Ohira ◽  
...  
Keyword(s):  
Salt Intake ◽  
Angiotensin Receptor Blockers ◽  
High Salt ◽  
Angiotensin Receptor ◽  
Hypertensive Patients ◽  
Spot Urine ◽  
High Salt Intake ◽  
Receptor Blockers

scholarly journals High salt intake: detrimental not only for blood pressure, but also for bone health?

Endocrine ◽  
2015 ◽  
Vol 49 (3) ◽  
pp. 580-582 ◽  
Author(s):  
Thomas Remer
Keyword(s):  
Blood Pressure ◽  
Bone Health ◽  
Salt Intake ◽  
High Salt ◽  
High Salt Intake

High Salt Intake Blunts Plasma Catecholamine and Renin Responses to Exercise

1984 ◽  
Vol 6 ◽  
pp. S95 ◽  
Author(s):  
L. H. Block ◽  
B. E. Lütold ◽  
P. Bolli ◽  
W. Kiowski ◽  
F. R. Bühler
Keyword(s):  
Salt Intake ◽  
High Salt ◽  
Plasma Catecholamine ◽  
High Salt Intake

Effect of high salt intake on sodium, potassium-dependent adenosine triphosphatase activity in the erythrocytes of normotensive men

1988 ◽  
Vol 75 (2) ◽  
pp. 167-170 ◽  
Author(s):  
Antonio P. Quintanilla ◽  
Maria I. Weffer ◽  
Haengil Koh ◽  
Mohammed Rahman ◽  
Agostino Molteni ◽  
...  
Keyword(s):  
Atpase Activity ◽  
Inorganic Phosphate ◽  
Adenosine Triphosphatase ◽  
Salt Intake ◽  
Control Period ◽  
Plasma Renin ◽  
Normal Diet ◽  
High Salt ◽  
Sodium Potassium ◽  
High Salt Intake

1. We measured ouabain-insensitive adenosine triphosphatase (ATPase), sodium, potassium-dependent adenosine triphosphatase (Na+,K+-ATPase) and intracellular Na+ and K+ in the erythrocytes of 19 healthy volunteers, before and after supplementation of their normal diet with 6.0–8.9 g of salt (102–137 mmol of NaCl) per day, for 5 days. 2. The subjects had a small but significant gain in weight. Mean plasma renin activity decreased from 1.57 to 0.73 pmol of angiotensin I h−1 ml−1 and plasma aldosterone from 0.46 to 0.24 nmol/l. 3. Total ATPase activity fell from 197.9 nmol of inorganic phosphate h−1 mg−1 during the control period to 173.5 during the high-salt period (P < 0.0125). Na+,K+-ATPase activity fell from 162.2 to 141.4 nmol of inorganic phosphate h−1 mg−1 (P < 0.05). Intracellular Na + and intracellular K+ did not change. 4. These results are consistent with the hypothesis that salt-induced volume expansion causes the release of a factor inhibitory to the Na+ pump.

Increased depressor response to N-arachidonoyl-dopamine during high salt intake: role of the TRPV1 receptor

2007 ◽  
Vol 25 (12) ◽  
pp. 2426-2433 ◽  
Author(s):  
Youping Wang ◽  
Donna H Wang
Keyword(s):  
Salt Intake ◽  
High Salt ◽  
Trpv1 Receptor ◽  
Depressor Response ◽  
High Salt Intake
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