Cognitive Mechanism, Affecting Factors and Its Neural Basis in L2 Syntactic Processing

2015 ◽  
Vol 23 (9) ◽  
pp. 1540
Author(s):  
Wen-Guang HE
Neuroreport ◽  
2007 ◽  
Vol 18 (14) ◽  
pp. 1437-1441 ◽  
Author(s):  
Kenji Ogawa ◽  
Masato Ohba ◽  
Toshio Inui

2021 ◽  
Vol 29 (4) ◽  
pp. 586
Author(s):  
Tingyong FENG ◽  
Xueke WANG ◽  
Ti SU

2010 ◽  
Vol 22 (9) ◽  
pp. 2131-2140 ◽  
Author(s):  
Giosuè Baggio ◽  
Travis Choma ◽  
Michiel van Lambalgen ◽  
Peter Hagoort

Research in psycholinguistics and in the cognitive neuroscience of language has suggested that semantic and syntactic processing are associated with different neurophysiologic correlates, such as the N400 and the P600 in the ERPs. However, only a handful of studies have investigated the neural basis of the syntax–semantics interface, and even fewer experiments have dealt with the cases in which semantic composition can proceed independently of the syntax. Here we looked into one such case—complement coercion—using ERPs. We compared sentences such as, “The journalist wrote the article” with “The journalist began the article.” The second sentence seems to involve a silent semantic element, which is expressed in the first sentence by the head of the verb phrase (VP) “wrote the article.” The second type of construction may therefore require the reader to infer or recover from memory a richer event sense of the VP “began the article,” such as began writing the article, and to integrate that into a semantic representation of the sentence. This operation is referred to as “complement coercion.” Consistently with earlier reading time, eye tracking, and MEG studies, we found traces of such additional computations in the ERPs: Coercion gives rise to a long-lasting negative shift, which differs at least in duration from a standard N400 effect. Issues regarding the nature of the computation involved are discussed in the light of a neurocognitive model of language processing and a formal semantic analysis of coercion.


2015 ◽  
Vol 29 (4) ◽  
pp. 135-146 ◽  
Author(s):  
Miroslaw Wyczesany ◽  
Szczepan J. Grzybowski ◽  
Jan Kaiser

Abstract. In the study, the neural basis of emotional reactivity was investigated. Reactivity was operationalized as the impact of emotional pictures on the self-reported ongoing affective state. It was used to divide the subjects into high- and low-responders groups. Independent sources of brain activity were identified, localized with the DIPFIT method, and clustered across subjects to analyse the visual evoked potentials to affective pictures. Four of the identified clusters revealed effects of reactivity. The earliest two started about 120 ms from the stimulus onset and were located in the occipital lobe and the right temporoparietal junction. Another two with a latency of 200 ms were found in the orbitofrontal and the right dorsolateral cortices. Additionally, differences in pre-stimulus alpha level over the visual cortex were observed between the groups. The attentional modulation of perceptual processes is proposed as an early source of emotional reactivity, which forms an automatic mechanism of affective control. The role of top-down processes in affective appraisal and, finally, the experience of ongoing emotional states is also discussed.


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