scholarly journals Spinal Stenosis Caused by Calcinosis in a Patient With Systemic Sclerosis

2021 ◽  
pp. jrheum.201389
Author(s):  
Tal Gazitt ◽  
Joy Feld ◽  
Devy Zisman
Keyword(s):  
Systemic Sclerosis ◽  
Soft Tissue ◽  
Spinal Stenosis ◽  
Juvenile Dermatomyositis ◽  
Phosphorus Metabolism ◽  
Soft Tissue Calcification ◽  
Connective Tissues ◽  
Tissue Calcification ◽  
Calcium Phosphorus ◽  
Normal Calcium

Calcinosis or dystrophic soft-tissue calcification occurs in damaged/devitalized tissues in the presence of normal calcium/ phosphorus metabolism.1 It is a known complication of connective tissues diseases, especially juvenile dermatomyositis and systemic sclerosis (SSc), and may be localized or widespread.2

scholarly journals Calcinosis cutis in limited cutaneous systemic sclerosis

QJM ◽  
2021 ◽  
Author(s):  
Sumantro Mondal ◽  
Debanjali Sinha ◽  
Alakendu Ghosh
Keyword(s):  
Systemic Sclerosis ◽  
Soft Tissue ◽  
Clinical Image ◽  
Soft Tissue Calcification ◽  
Radiographic Image ◽  
Calcinosis Cutis ◽  
Tissue Calcification ◽  
Rheumatological Diseases

Abstract Soft tissue calcification is seen in some rheumatological diseases, including systemic sclerosis. We herein present a clinical image of calcinosis cutis of finger pulps and its characteristic radiographic image in a patient with limited cutaneous systemic sclerosis.

Widespread soft tissue calcification in systemic sclerosis, polymyositis, and polyarthritis

The Lancet ◽  
2021 ◽  
Vol 397 (10272) ◽  
pp. 409
Author(s):  
Ole Hudowenz ◽  
Philipp Klemm ◽  
Uwe Lange ◽  
Ulf Mueller-Ladner
Keyword(s):  
Systemic Sclerosis ◽  
Soft Tissue ◽  
Soft Tissue Calcification ◽  
Tissue Calcification

Biochemical studies on the anticalcinotic effect of sodium salicylate

1969 ◽  
Vol 47 (7) ◽  
pp. 597-601 ◽  
Author(s):  
I. Berczi ◽  
A. Somogyi
Keyword(s):  
Soft Tissue ◽  
Lead Acetate ◽  
Sodium Salicylate ◽  
Egg White ◽  
Blood Levels ◽  
Soft Tissue Calcification ◽  
Tissue Calcification ◽  
Biochemical Studies ◽  
Calcium Phosphorus

Experimental soft-tissue calcification was induced in the hind paw of rats injected intravenously with lead acetate and locally with egg white. This mineralization was inhibited by treatment with sodium salicylate given orally. Sodium salicylate did not influence the extravasation of lead in the treated paw, but decreased the lead-induced hypercalcemia and hyperphosphatemia. A parallelism was found between the blood levels of calcium, phosphorus, and lead. A secondary deposition of lead was evident in the heavily calcified paws.

Sign in / Sign up

Export Citation Format

Share Document