Acute Induction of Soft Tissue Calcification with Transient Hyperphosphatemia in the KK Mouse by Modification in Dietary Contents of Calcium, Phosphorus, and Magnesium

1970 ◽  
Vol 100 (4) ◽  
pp. 404-412 ◽  
Author(s):  
Yukihiko Hamuro ◽  
Akio Shino ◽  
Ziro Suzuoki
2021 ◽  
pp. jrheum.201389
Author(s):  
Tal Gazitt ◽  
Joy Feld ◽  
Devy Zisman

Calcinosis or dystrophic soft-tissue calcification occurs in damaged/devitalized tissues in the presence of normal calcium/ phosphorus metabolism.1 It is a known complication of connective tissues diseases, especially juvenile dermatomyositis and systemic sclerosis (SSc), and may be localized or widespread.2


1969 ◽  
Vol 47 (7) ◽  
pp. 597-601 ◽  
Author(s):  
I. Berczi ◽  
A. Somogyi

Experimental soft-tissue calcification was induced in the hind paw of rats injected intravenously with lead acetate and locally with egg white. This mineralization was inhibited by treatment with sodium salicylate given orally. Sodium salicylate did not influence the extravasation of lead in the treated paw, but decreased the lead-induced hypercalcemia and hyperphosphatemia. A parallelism was found between the blood levels of calcium, phosphorus, and lead. A secondary deposition of lead was evident in the heavily calcified paws.


PEDIATRICS ◽  
1986 ◽  
Vol 77 (3) ◽  
pp. 382-385
Author(s):  
Mila Barak ◽  
Sylvia Herschkowitz ◽  
Johanan Montag

Two cases of gross soft tissue calcification following intramuscular administration of vitamin E in two premature babies are described. The drug was administered for prevention of retinopathy of prematurity. The relation between this complication and both the dosage used and the duration of the treatment is discussed. In spite of the extent of the calcifications, a benign course was observed. Other reports about this complication are reviewed.


Sign in / Sign up

Export Citation Format

Share Document