scholarly journals Acute movement disorder with bilateral basal ganglia lesions in diabetic uremia

2011 ◽  
Vol 14 (3) ◽  
pp. 211 ◽  
Author(s):  
GurusidheshwarM Wali ◽  
RajendraV Mali ◽  
MallikarjunS Khanpet
Keyword(s):  
Basal Ganglia ◽  
Movement Disorder

Charles David Marsden. 15 April 1938 — 29 September 1998

2012 ◽  
Vol 58 ◽  
pp. 203-228 ◽  
Author(s):  
Niall Quinn ◽  
John Rothwell ◽  
Peter Jenner
Keyword(s):  
Basal Ganglia ◽  
Movement Disorder ◽  
Communication Skills ◽  
Movement Disorders ◽  
Normal Function ◽  
Clinical Aspects ◽  
Publication Record ◽  
Research And Teaching ◽  
The Usa ◽  
Movement Disorder Society

David Marsden was the most outstanding UK clinical neuroscientist of his generation, making key discoveries in the neurophysiology, neurochemistry and clinical aspects of diseases of the basal ganglia, and their normal function. His legacies are the establishment, with Stanley Fahn in the USA, of movement disorders as a subspecialty within neurology, of the international Movement Disorder Society, and of the journal Movement Disorders ; his ex-students and fellows around the globe; and his research and teaching output embodied in his extraordinarily prolific publication record of more than 1360 papers, books and chapters, culminating in the posthumous completion and publication in December 2011 of Marsden’s book of movement disorders , a project he had started in 1984. All of these were achieved through the combination of his intellect and drive, his communication skills, and his forceful and charismatic personality.

The complex movement disorder of Kasabach-Merritt syndrome associated with a basal ganglia lesion

2012 ◽  
Vol 27 (4) ◽  
pp. 591-593
Author(s):  
Christos Ganos ◽  
Simone Zittel ◽  
Christian Gerloff ◽  
Alexander Münchau ◽  
Tobias Bäumer
Keyword(s):  
Basal Ganglia ◽  
Movement Disorder ◽  
Complex Movement

scholarly journals Fahr’s Disease Presenting with Dementia at Onset: A Case Report and Literature Review

2014 ◽  
Vol 2014 ◽  
pp. 1-3 ◽  
Author(s):  
Rocco Salvatore Calabrò ◽  
Letteria Spadaro ◽  
Angela Marra ◽  
Placido Bramanti
Keyword(s):  
Case Report ◽  
Basal Ganglia ◽  
Literature Review ◽  
Movement Disorder ◽  
Rare Case ◽  
Centrum Semiovale ◽  
Fahr’S Disease ◽  
Radiological Features ◽  
Fahr's Disease ◽  
Behavioral Abnormalities

Fahr’s disease (FD) is characterized by sporadic or familiar idiopathic calcification of the basal ganglia, dentate nuclei of the cerebellum, and centrum semiovale, mainly presenting with movement disorder, dementia, and behavioral abnormalities. We described a rare case of Fahr’s disease presenting at onset only with behavioral and neuropsychological alterations, whose diagnosis was supposed only after a brain CT, which showed extensive bilateral calcifications in the dentate nuclei of the cerebellum and basal ganglia. Since the onset of Fahr’s disease may be a dysexecutive syndrome with behavioral abnormalities, the clinical and radiological features are really important to do the appropriate diagnosis.

scholarly journals Progressive chorea and dystonia associated with a large arteriovenous malformation

2019 ◽  
Author(s):  
Jeffrey S Raskin ◽  
Mered Parnes ◽  
Sandi Lam
Keyword(s):  
Intellectual Disability ◽  
Basal Ganglia ◽  
Arteriovenous Malformation ◽  
Movement Disorder ◽  
Progressive Movement ◽  
The Right

A 9-year-old male with intellectual disability and epilepsy presenting with a progressive movement disorder characterized by chorea and dystonia primarily affecting the left face, arm, and leg and attributed to an arteriovenous malformation centered within the right thalamus with bilateral extension into the basal ganglia, midbrain and pons is reported.

scholarly journals The Role of the Cerebellum in the Pathophysiology of Parkinson's Disease

2013 ◽  
Vol 40 (3) ◽  
pp. 299-306 ◽  
Author(s):  
Mechelle M. Lewis ◽  
Shawna Galley ◽  
Samantha Johnson ◽  
James Stevenson ◽  
Xuemei Huang ◽  
...  
Keyword(s):  
Parkinson’S Disease ◽  
Parkinson's Disease ◽  
Basal Ganglia ◽  
Substantia Nigra ◽  
Movement Disorder ◽  
Significant Variation ◽  
Compensatory Mechanisms ◽  
Dopaminergic Cells ◽  
Basal Ganglia Disease

Parkinson's disease (PD), the most common neurodegenerative movement disorder, has traditionally been considered a “classic” basal ganglia disease, as the most obvious pathology is seen in the dopaminergic cells in the substantia nigra pars compacta. Nevertheless recent discoveries in anatomical connections linking the basal ganglia and the cerebellum have led to a re-examination of the role of the cerebellum in the pathophysiology of PD. This review summarizes the role of the cerebellum in explaining many curious features of PD: the significant variation in disease progression between individuals; why severity of dopaminergic deficit correlates with many features of PD such as bradykinesia, but not tremor; and why PD subjects with a tremor-predominant presentation tend to have a more benign prognosis. It is clear that the cerebellum participates in compensatory mechanisms associated with the disease and must be considered an essential contributor to the overall pathophysiology of PD.

Sign in / Sign up

Export Citation Format

Share Document