scholarly journals Painful tic convulsif caused by posterior fossa arteriovenous malformation

2019 ◽  
Vol 2 (3) ◽  
pp. 136
Author(s):  
Niraj Kumar ◽  
Sweety Kumari ◽  
SarajK Singh ◽  
Deepak Kumar
Keyword(s):  
Arteriovenous Malformation ◽  
Posterior Fossa ◽  
Painful Tic Convulsif

Arteriovenous malformations in the posterior fossa

1977 ◽  
Vol 47 (1) ◽  
pp. 50-56 ◽  
Author(s):  
Hiroshi Matsumura ◽  
Yasumasa Makita ◽  
Kuniyuki Someda ◽  
Akinori Kondo
Keyword(s):  
Arteriovenous Malformation ◽  
Brain Stem ◽  
Posterior Fossa ◽  
Arteriovenous Malformations ◽  
Cerebellopontine Angle ◽  
Anterior Part ◽  
Content Type ◽  
The Brain ◽  
Fine Print

✓ We have operated on 12 of 14 cases of arteriovenous malformation (AVM) in the posterior fossa since 1968, with one death. The lesions were in the cerebellum in 10 cases (three anteromedial, one central, three lateral, and three posteromedial), and in the cerebellopontine angle in two; in two cases the lesions were directly related to the brain stem. The AVM's in the anterior part of the cerebellum were operated on through a transtentorial occipital approach.

Posterior fossa subdural hematoma due to ruptured arteriovenous malformation

2000 ◽  
Vol 8 (6) ◽  
pp. 1-4 ◽  
Author(s):  
Narendra N. Datta ◽  
Kwong Y. Chan ◽  
John C. K. Kwok ◽  
Christopher Y. F. Poon
Keyword(s):  
Arteriovenous Malformation ◽  
Posterior Fossa ◽  
Subdural Hematoma ◽  
Subarachnoid Space ◽  
Arteriovenous Malformations ◽  
Acute Subdural Hematoma ◽  
Rare Entity ◽  
Neurosurgical Department ◽  
Usual Location

Patients with cerebellar arteriovenous malformations (AVM) commonly present to the neurosurgical department after having suffered hemorrhages. The subarachnoid space is the usual location for these often repeating episodes of bleedings. In addition, these patients can present with parenchymal hemorrhage. Acute subdural hematoma caused by a ruptured cerebellar AVM is a rare entity and is not generally recognized. The authors present a case of acute posterior fossa subdural hematoma resulting from a ruptured cerebellar AVM.

Successful evacuation of a pontine hematoma secondary to rupture of a pathologically diagnosed “cryptic” vascular malformation

1973 ◽  
Vol 39 (1) ◽  
pp. 104-108 ◽  
Author(s):  
Ben B. Scott ◽  
Joachim F. Seeger ◽  
Richard C. Schneider
Keyword(s):  
Arteriovenous Malformation ◽  
Brain Stem ◽  
Posterior Fossa ◽  
Vascular Malformation ◽  
Nerve Palsy ◽  
Neurological Deficits ◽  
Content Type ◽  
Brain Stem Lesion ◽  
Seventh Nerve ◽  
Fine Print

✓ A posterior fossa exploration was performed on a child thought initially to have an inoperable brain stem lesion. A pontine hematoma was discovered and evacuated. The pathological specimen was designated as a “cryptic” arteriovenous malformation. All preoperative neurological deficits disappeared except for a minimal left seventh nerve palsy.

Arteriovenous malformation in the posterior fossa supplied by the external carotid circulation

1974 ◽  
Vol 41 (4) ◽  
pp. 502-507 ◽  
Author(s):  
Carl J. Graf ◽  
Arnold H. Menezes
Keyword(s):  
Carotid Artery ◽  
Arteriovenous Malformation ◽  
Posterior Fossa ◽  
Blood Supply ◽  
External Carotid Artery ◽  
External Carotid ◽  
Content Type ◽  
Carotid Circulation ◽  
Fine Print

✓ The authors report a case in which the blood supply of a posterior fossa arteriovenous malformation was derived entirely from the external carotid artery.

Spontaneous regression of posterior fossa dural arteriovenous malformation

1979 ◽  
Vol 51 (5) ◽  
pp. 715-717 ◽  
Author(s):  
Shunro Endo ◽  
Keiji Koshu ◽  
Jiro Suzuki
Keyword(s):  
Arteriovenous Malformation ◽  
Posterior Fossa ◽  
Spontaneous Regression ◽  
Surgical Intervention ◽  
Clinical Symptoms ◽  
Dural Arteriovenous Malformation ◽  
Content Type ◽  
Homonymous Hemianopsia ◽  
Bleeding Episodes ◽  
Fine Print

✓ The authors report a case of infratentorial arteriovenous malformation that regressed spontaneously within 25 months without bleeding episodes or surgical intervention. The patient's clinical symptoms, left-sided tinnitus, cranial bruit, and right homonymous hemianopsia, disappeared completely over the same period.

Arteriovenous Malformation Associated with Multiple Aneurysms in the Posterior Fossa: A Case Report with a Review of the Literature

Neurosurgery ◽  
1987 ◽  
Vol 21 (3) ◽  
pp. 387-391 ◽  
Author(s):  
J. Noterman ◽  
P. Georges ◽  
J. Brotchi
Keyword(s):  
Case Report ◽  
Subarachnoid Hemorrhage ◽  
Arteriovenous Malformation ◽  
Posterior Fossa ◽  
The Other ◽  
Review Of The Literature ◽  
Multiple Aneurysms ◽  
One Stage ◽  
Cerebellar Artery

Abstract The association of two aneurysms on the distal posteroinferior cerebellar artery with an arteriovenous malformation fed by the same artery is reported. A one-stage operation was performed. The other cases in the literature are analyzed with particular regard to the origin of the subarachnoid hemorrhage and the location of the aneurysms.

scholarly journals Neurotological examination can conclude as arteriovenous malformation of the posterior fossa.

1985 ◽  
Vol 78 (8) ◽  
pp. 1541-1547
Author(s):  
Kazuhiro Teramoto ◽  
Koki Nakaigawa ◽  
Kyoko Ohtsu ◽  
Eiji Sakata
Keyword(s):  
Arteriovenous Malformation ◽  
Posterior Fossa

Arteriovenous fistula around the ventriculoperitoneal shunt system in a patient with a dural arteriovenous malformation of the posterior fossa

1995 ◽  
Vol 82 (2) ◽  
pp. 288-290 ◽  
Author(s):  
Shin-ichi Yoshimura ◽  
Nobuo Hashimoto ◽  
Kiyoshi Kazekawa ◽  
Atsushi Obata ◽  
Chikao Yutani ◽  
...  
Keyword(s):  
Arteriovenous Malformation ◽  
Posterior Fossa ◽  
Ventriculoperitoneal Shunt ◽  
Rare Complication ◽  
Therapeutic Embolization ◽  
Dural Arteriovenous Malformation ◽  
Shunt System ◽  
Content Type ◽  
Underlying Mechanisms ◽  
Dural Avm

✓ A dural arteriovenous malformation (AVM) of the posterior fossa can produce persistent tinnitus, convulsions, and dementia. Successful therapeutic embolization may result in a complete cure, but in some cases, patients do not respond to the treatment. The authors report a patient with a dural AVM of the posterior fossa that did not respond to repeated intravascular treatments, but resulted in an arteriovenous shunt in the scalp around the ventriculoperitoneal shunt system. Although several hypotheses have been proposed on the pathogenesis of dural AVMs, the underlying mechanisms remain unknown. The rare complication we encountered may shed some light on the pathogenesis of dural AVMs.

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