scholarly journals Atypical mycobacterial flexor tenosynovitis presenting as carpal tunnel syndrome: Presentation of three cases and review

2002 ◽  
Vol 10 (04) ◽  
Author(s):  
JP Brutus ◽  
A Nikolis
2000 ◽  
Vol 25 (3) ◽  
pp. 308-310 ◽  
Author(s):  
A. WADA ◽  
S. NOMURA ◽  
F. IHARA

We present a case in which Mycobacterium kansasii flexor tenosynovitis caused the development of carpal tunnel syndrome. The diagnosis was made from synovial tissue specimens taken at the time of operation.


Hand Surgery ◽  
1997 ◽  
Vol 02 (02) ◽  
pp. 117-122
Author(s):  
Takuro Wada ◽  
Yasuhiko Minagi ◽  
Takashi Kirita ◽  
Seiichi Ishii

We carried out a survey of hand abnormalities in 65 Japanese adult diabetics (non-insulin-dependent diabetes mellitus) and compared the result with that of 65 non-diabetic adults as controls. A significantly higher incidence of Dupuytren's contracture, limited joint mobility, carpal tunnel syndrome, and flexor tenosynovitis was found in the diabetic group. In the diabetic group, Dupuytren's contracture was noted in 10 patients (15%), limited joint mobility in 34 patients (52%), carpal tunnel syndrome in 21 patients (32%), and flexor tenosynovitis in 10 patients (15%). Dupuytren's contracture in the diabetic patients involved the fourth and fifth rays. In diabetic carpal tunnel syndrome, as compared to the idiopathic form, men were more frequently affected than women. Carpal tunnel syndrome showed a positive correlation with duration of the diabetes and use of insulin during the last twelve months prior to the examination. All of 10 diabetic patients with flexor tenosynovitis also had limited joint mobility. Six of these 10 patients also had carpal tunnel syndrome.


2017 ◽  
Vol 56 (11) ◽  
pp. 1439-1442
Author(s):  
Hirofumi Yoshida ◽  
Haruki Imura ◽  
Taiga Goto ◽  
Takeharu Nakamata ◽  
Mohamud R. Daya ◽  
...  

2002 ◽  
Vol 10 (4) ◽  
pp. 167-170
Author(s):  
Jp Brutus ◽  
A Nikolis ◽  
Y Baeten ◽  
N Chahidi ◽  
L Kinnen ◽  
...  

Three patients with carpal tunnel syndrome secondary to atypical mycobacteria flexor tenosynovitis are presented. Aggressive surgical debridement combined with long term antitubercular pharmacotherapy resulted in a good outcome, but the lag time to diagnosis and course of disease were long. Diagnosis of these infections requires a high suspicion index, adequate surgical biopsy and appropriate cultures. Atypical mycobacteria infections must be considered in the differential diagnosis in any patient with evolving chronic tenosynovitis, even if the patient has no history of immunosuppression, and especially if environmental risk factors are present.


2003 ◽  
Vol 8 (4) ◽  
pp. 4-5
Author(s):  
Christopher R. Brigham ◽  
James B. Talmage

Abstract Permanent impairment cannot be assessed until the patient is at maximum medical improvement (MMI), but the proper time to test following carpal tunnel release often is not clear. The AMA Guides to the Evaluation of Permanent Impairment (AMA Guides) states: “Factors affecting nerve recovery in compression lesions include nerve fiber pathology, level of injury, duration of injury, and status of end organs,” but age is not prognostic. The AMA Guides clarifies: “High axonotmesis lesions may take 1 to 2 years for maximum recovery, whereas even lesions at the wrist may take 6 to 9 months for maximal recovery of nerve function.” The authors review 3 studies that followed patients’ long-term recovery of hand function after open carpal tunnel release surgery and found that estimates of MMI ranged from 25 weeks to 24 months (for “significant improvement”) to 18 to 24 months. The authors suggest that if the early results of surgery suggest a patient's improvement in the activities of daily living (ADL) and an examination shows few or no symptoms, the result can be assessed early. If major symptoms and ADL problems persist, the examiner should wait at least 6 to 12 months, until symptoms appear to stop improving. A patient with carpal tunnel syndrome who declines a release can be rated for impairment, and, as appropriate, the physician may wish to make a written note of this in the medical evaluation report.


2007 ◽  
Vol 12 (6) ◽  
pp. 5-8 ◽  
Author(s):  
J. Mark Melhorn

Abstract Medical evidence is drawn from observation, is multifactorial, and relies on the laws of probability rather than a single cause, but, in law, finding causation between a wrongful act and harm is essential to the attribution of legal responsibility. These different perspectives often result in dissatisfaction for litigants, uncertainty for judges, and friction between health care and legal professionals. Carpal tunnel syndrome (CTS) provides an example: Popular notions suggest that CTS results from occupational arm or hand use, but medical factors range from congenital or acquired anatomic structure, age, sex, and body mass index, and perhaps also involving hormonal disorders, diabetes, pregnancy, and others. The law separately considers two separate components of causation: cause in fact (a cause-and-effect relationship exists) and proximate or legal cause (two events are so closely related that liability can be attached to the first event). Workers’ compensation systems are a genuine, no-fault form of insurance, and evaluators should be aware of the relevant thresholds and legal definitions for the jurisdiction in which they provide an opinion. The AMA Guides to the Evaluation of Permanent Impairment contains a large number of specific references and outlines the methodology to evaluate CTS, including both occupational and nonoccupational risk factors and assigning one of four levels of evidence that supports the conclusion.


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