scholarly journals AO Spine classification of upper cervical injuries

2018 ◽  
Author(s):  
Francis Deng ◽  
Craig Hacking
2013 ◽  
Vol 37 (2) ◽  
pp. 139-151 ◽  
Author(s):  
Andrei F. Joaquim ◽  
Enrico Ghizoni ◽  
Helder Tedeschi ◽  
Brandon Lawrence ◽  
Darrel S. Brodke ◽  
...  

2001 ◽  
Vol 8 (1) ◽  
pp. 33-37 ◽  
Author(s):  
T.E. COSAN ◽  
E. TEL ◽  
A. ARSLANTAS ◽  
M. VURAL ◽  
A.I. GUNER

2015 ◽  
Vol 6 (1) ◽  
pp. 16 ◽  
Author(s):  
AndreiF Joaquim ◽  
AlpeshA Patel ◽  
Helder Tedeschi ◽  
AlexanderR Vaccaro ◽  
AlexandreR. D. Yacoub ◽  
...  

1984 ◽  
Vol 60 (4) ◽  
pp. 700-706 ◽  
Author(s):  
Stephen A. Hill ◽  
Carole A. Miller ◽  
Edward J. Kosnik ◽  
William E. Hunt

✓ This review of pediatric neck injuries includes patients admitted to Children's Hospital of Columbus, Ohio, during the period 1969 to 1979. The 122 patients with neck injuries constituted 1.4% of the total neurosurgical admissions during this time. Forty-eight patients had cervical strains; 74 had involvement of the spinal column; and 27 had neurological deficits. The injuries reached their peak incidence during the summer months, with motor-vehicle accidents accounting for 31%, diving injuries and falls from a height 20% each, football injuries 8%, other sports 11%, and miscellaneous 10%. There is a clear division of patients into a group aged 8 years or less with exclusively upper cervical injuries, and an older group with pancervical injuries. In the younger children, the injuries involved soft tissue (subluxation was seen more frequently than fracture), and tended to occur through subchondral growth plates, with a more reliable union than similar bone injuries. In the older children, the pattern and etiology of injury are the same as in adults. The entire cervical axis is at risk, and there is a tendency to fracture bone rather than cartilaginous structures.


2020 ◽  
pp. 34-39
Author(s):  
V.F. Makeev ◽  
U.D. Telyshevska ◽  
O.D. Telyshevska ◽  
M.Yu. Mykhailevych

Temporomandibular joint disease (TMJ) is one of the most pressing problems of modern dentistry, on the one hand, the frequency of pathology of the temporomandibular joint, and on the other hand - the complexity of diagnosis. In the medical specialty "dentistry" there is no section where there would be as many debatable and unresolved issues as in the diagnosis and treatment of diseases of the temporomandibular joints. Aim of the research. Based on the analysis of sources of scientific and medical information to determine the role and place of "Costen's syndrome" in the pathology of the temporomandibular joints. Results and discussion The term TMJ dysfunction has up to 20 synonyms: dysfunction, muscle imbalance, myofascial pain syndrome, musculoskeletal dysfunction, occlusal-articulation syndrome, cranio-mandibular TMJ dysfunction, neuromuscular and articular dysfunction. Finally, in the International Classification of Diseases (ICD-10), pain dysfunction of the temporomandibular joint has taken its place under the code K0760 with the additional name "Costen's syndrome", which is given in parentheses under the same code. Thus, such a diagnosis as "Costen's syndrome" is not excluded in the International Classification of Diseases. The first clinical symptoms and signs of TMJ were systematized in 1934 by the American otorhinolaryngologist J. Costen and included in the special literature called "Costen's syndrome". This syndrome includes: pain in the joint, which often radiates to the neck, ear, temple, nape; clicking, crunching, squeaking sound during movements of the lower jaw; trismus; hearing loss; dull pain inside and outside the ears, noise, congestion in the ears; pain and burning of the tongue; dizziness, headache on the side of the affected joint, facial pain on the type of trigeminal neuralgia. The author emphasized the great importance of pain and even singled out "mandibular neuralgia." The criteria proposed by McNeill (McNeill C.) in 1997 are somewhat different from those described in ICD-10: pain in the masticatory muscles, TMJ, or in the ear area, which is aggravated by chewing; asymmetric movements of the lower jaw; pain that does not subside for at least 3 months. The definition of the International Headache Society is similar in content. Anatomical and topographic study of the corpse material suggested the presence of a structural connection between the TMJ and the middle ear. According to some data, in 68% of cases the wedge-shaped mandibular ligament reaches the scaly-tympanic fissure and the middle ear, and in 8% of cases it is attached to the hammer. In addition, several ways of spreading inflammatory mediators from the affected TMJ to the middle and inner ear, which causes otological symptoms, have been described. It should be noted that there are certain prerequisites for the mutual influence of the structures of the cervical apparatus, middle and inner ear and upper cervical region at different levels: embryological, anatomical and physiological. At the embryological level. It is confirmed that from the first gill arch develops the upper jaw, hammer and anvil, Meckel's cartilage of the lower jaw, masticatory muscles, the muscle that tenses the eardrum, the muscle that tenses the soft palate, the anterior abdomen of the digastric muscle, glands, as well as the maxillary artery and trigeminal nerve, the branches of which innervate most of these structures. At the anatomical level. Nerve, muscle, joint and soft tissue structures of this region are located close enough and have a direct impact on each other. The location of the stony-tympanic cleft in the medial parts of the temporomandibular fossa is important for the development of pain dysfunction. At the physiological level. A child who begins to hold the head, the functional activity of the extensors and flexors of the neck gradually increases synchronously with the muscles of the floor of the mouth and masticatory muscles, combining their activity around the virtual axis of the paired temporomandibular joint. In addition, the location of the caudal spinal nucleus of the trigeminal nerve, which is involved in the innervation of the structures of the ear, temporomandibular joint and masticatory muscles at the level of the cervical segments C1-C3 creates the possibility of switching afferent impulses from the trigeminal nerve to the upper cervical system. Innervate the outer ear, neck muscles and skin of the neck and head. Also important are the internuclear connections in the brainstem, which switch signals between the vestibular and trigeminal nuclei. That is why the approach to the treatment of this pathology should be only comprehensive, including clinical assessment of the disease not only by a dentist or maxillofacial surgeon, but also a neurologist, otorhinolaryngologist, chiropractor, psychotherapist with appropriate diagnostic methods and joint management of the patient.


1979 ◽  
Vol 24 (1) ◽  
pp. 10787J ◽  
Author(s):  
A. D. Hooper

Neurosurgery ◽  
2007 ◽  
Vol 61 (5) ◽  
pp. 995-1015 ◽  
Author(s):  
Dachling Pang ◽  
William R. Nemzek ◽  
John Zovickian

Abstract OBJECTIVE The diagnosis of atlanto-occipital dislocation (AOD) remains problematic as a result of a lack of reliable radiodiagnostic criteria. In Part 1 of the AOD series, we showed that the normal occiput–C1 joint in children has an extremely narrow joint gap (condyle–C1 interval [CCI]) with great left-right symmetry. In Part 2, we used a CCI of 4 mm or greater measured on reformatted computed tomographic (CT) scans as the indicator for AOD and tested the diagnostic sensitivity and specificity of CCI against published criteria. The clinical manifestation, neuroimaging findings, management, and outcome of our series of patients with AOD are also reported. METHOD For diagnostic sensitivity, we applied the CCI criterion on 16 patients who fulfilled one or more accepted radiodiagnostic criteria of AOD and who showed clinical and imaging hallmarks of the syndrome. All 16 patients had plain cervical spine x-rays, head CT scans, axial cervical spine CT scans with reconstruction, and magnetic resonance imaging scans. The diagnostic yield and false-negative rate of CCI were compared with those of four published “standard” tests, namely Wholey's dens-basion interval, Powers' ratio, Harris' basion-axis interval, and Sun's interspinous ratio. The diagnostic value of “nonstandard” indicators such as cervicomedullary deficits, tectorial membrane and other ligamentous damage, perimedullary subarachnoid hemorrhage, and extra-axial blood at C1−C2 were also assessed. For diagnostic specificity, we applied CCI and the “standard” and “nonstandard” tests on 10 patients from five classes of non-AOD upper cervical injuries. The false-positive diagnostic rates for AOD of all respective tests were documented. RESULTS The CCI criterion was positive in all 16 patients with AOD with a diagnostic sensitivity of 100%. Fourteen patients had bilateral AOD with disruption and widening of both OC1 joints. Two patients had unilateral AOD with only one joint wider than 4 mm. The abnormal CCI varied from 5 to 34 mm. Eight patients showed blatant left-right joint asymmetry in either CCI or anatomic conformation. The diagnostic sensitivities for the “standard” tests are as follows: Wholey's, 50%; Powers', 37.5%; Harris', 31%; and Sun's, 25%, with false-negative rates of 50, 62.5, 69, and 75%, respectively. The sensitivities for the “nonstandard” indicators are: tectorial membrane damage, 71%; perimedullary blood, 63%; and C1−C2 extra-axial blood, 75%, with false-negative rates of 29, 37, and 25%, respectively. Fifteen patients with AOD had occiput-cervical fusion. There were one early and two delayed deaths (19% mortality); two patients (12%) had complete or severe residual high quadriplegia, but 11 children (69%) enjoyed excellent neurological recovery. CCI was normal in all 10 patients with non-AOD upper cervical injuries with a diagnostic specificity of 100%. The false-positive rates for the four “standard” tests were: Sun's, 60%; Harris', 50%; Wholey's, 30%; and Powers', 10%; for the “nonstandard” indicator, the rates were: cervicomedullary deficits, 70%; tectorial membrane damage, 40%; C1−C2 extra-axial blood, 40%; and perimedullary blood, 30%. CONCLUSION The CCI criterion has the highest diagnostic sensitivity and specificity for AOD among all other radiodiagnostic criteria and indicators. CCI is easily computed from reconstructed CT scans, has almost no logistical or technical distortions, can capture occiput–C1 joint dislocation in all three planes, and is unaffected by congenital anomalies or maturation changes of adjacent structures. Because CCI is the only test that directly measures the integrity of the actual joint injured in AOD and a widened CCI cannot be concealed by postinjury changes in the head and neck relationship, it surpasses others that use changeable landmarks.


1995 ◽  
Vol 9 (4) ◽  
pp. 223-233
Author(s):  
C.B Huckell ◽  
S.M Tooke ◽  
J.P Kostuik

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