Cutaneous leishmaniasis: surgical management of a case with unusual clinical and histological features

1978 ◽  
Vol 114 (9) ◽  
pp. 1354-1355 ◽  
Author(s):  
S. Barsky
2019 ◽  
Vol 9 (4) ◽  
Author(s):  
Ziad Sleiman ◽  
Loubna Abboud ◽  
Elie Mehanna ◽  
Ramzi Mahmoud ◽  
Elie Yaacoub ◽  
...  

Neural tumors localized in jaw bone are relatively rare. This article presents a case of intraosseous neurofibroma of the mandible in a 37-year-old female patient. A review of clinical, radiographic, histological features and surgical management of the patient are discussed along with a review of the literature.


2012 ◽  
Vol 54 (3) ◽  
pp. 165-170 ◽  
Author(s):  
Erika Ivett Sosa-Bibiano ◽  
Nicole Raymonde Van Wynsberghe ◽  
Silvia Beatriz Canto-Lara ◽  
Fernando Jose Andrade-Narvaez

There is not an experimental model of localized cutaneous leishmaniasis (LCL) caused by Leishmania (Leishmania) mexicana. The aim of the present study was to characterize the clinical and histological features of Peromyscus yucatanicus experimentally infected with L. (L.) mexicana. A total of 54 P. yucatanicus (groups of 18) were inoculated with 1x10(6) promastigotes of L. (L.) mexicana in the base of the tail. They were euthanized at three and six months post experimental infection. The control group was inoculated with RPMI-1640. The predominant clinical sign observed was a single ulcerated lesion in 27.77% (5/18) and in 11.11% (2/18) P. yucatanicus at three and six months respectively. The histological pattern described as chronic granulomatous inflammation with or without necrosis was found in 7/7 (100%) biopsies of euthanized P. yucatanicus at three (n = 5) and six (n = 2) months, respectively. These results resembled clinical and histological features caused by L. (L.) mexicana in humans, and support the possibility to employ P. yucatanicus as a novel experimental model to study LCL caused by this parasite.


2019 ◽  
Vol 4 (5) ◽  
pp. 857-869
Author(s):  
Oksana A. Jackson ◽  
Alison E. Kaye

Purpose The purpose of this tutorial was to describe the surgical management of palate-related abnormalities associated with 22q11.2 deletion syndrome. Craniofacial differences in 22q11.2 deletion syndrome may include overt or occult clefting of the palate and/or lip along with oropharyngeal variances that may lead to velopharyngeal dysfunction. This chapter will describe these circumstances, including incidence, diagnosis, and indications for surgical intervention. Speech assessment and imaging of the velopharyngeal system will be discussed as it relates to preoperative evaluation and surgical decision making. Important for patients with 22q11.2 deletion syndrome is appropriate preoperative screening to assess for internal carotid artery positioning, cervical spine abnormalities, and obstructive sleep apnea. Timing of surgery as well as different techniques, common complications, and outcomes will also be discussed. Conclusion Management of velopharyngeal dysfunction in patients with 22q11.2 deletion syndrome is challenging and requires thoughtful preoperative assessment and planning as well as a careful surgical technique.


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