The Add Health Data in Criminology

2021 ◽  
pp. 1-7
Author(s):  
Marissa Landeis ◽  
John H. Boman ◽  
Thomas J. Mowen
Keyword(s):  
Author(s):  
Amaia Iratzoqui

The current article examined the overlap of domestic violence across the life course, connecting childhood abuse and adolescent dating victimization to adult intimate partner victimization, and the connection between these behaviors and adult domestic violence perpetration against partners and children. Using three waves of Add Health data, the study found that childhood and adolescent domestic victimization were directly and indirectly linked to adult intimate partner victimization and that domestic violence perpetration also played a role. These findings indicate that offending must be accounted for in tracking patterns of victimization over the life course and that the overlap must more directly be reconciled in current criminal justice policy.


2009 ◽  
Vol 31 (3) ◽  
pp. 381-406 ◽  
Author(s):  
Tsui-Sui Annie Kao ◽  
Carol Loveland-Cherry ◽  
Barbara Guthrie

Maternal influences on adolescents’ sexual initiation have been examined over two time points in 433 Asian American—Pacific Islander (AAPI) adolescents in a secondary analysis of the Add Health data set using structural equation modeling. A longitudinal model built on a preliminary qualitative study is used to examine the fit between data and model. Maternal perceived connectedness and adolescents’ perceived maternal sexual expectations in sexual timing are found to be protective for delaying AAPI adolescents’ sexual initiation. Unexpectedly, mothers’ perceptions of sexual discussion are not protective for delaying adolescents’ sexual initiation. The results suggest interventions should focus on promoting AAPI mothers’ perceptions of connectedness with their adolescents and adolescents’ perceptions of their mothers’ sexual expectations (as measured by adolescents’ perceived maternal disapproval attitudes and affects) in delaying sexual initiation.


2017 ◽  
Vol 44 (6) ◽  
pp. 815-842 ◽  
Author(s):  
Turgut Ozkan ◽  
John L. Worrall

Previous tests of the maturity gap thesis incorporated one-dimensional autonomy-based measures of social maturity. We present the first test of the maturity gap thesis to incorporate psychosocial measures (i.e., temperance, responsibility, and perspective). Four delinquency and substance abuse variables from Wave 2 of the Add Health data were regressed on the following variables developed from Wave 1: relative pubertal development, relative psychosocial maturity, personal autonomy, and interactions among each ( n = 4,530 participants younger than 18 and 294 older than 18). Personal autonomy lost significance when psychosocial maturity, personal autonomy, and pubertal development were included in the same model. However, psychosocial maturity did not clearly interact with either personal autonomy or pubertal development. Psychosocial maturity was associated with deviant behaviors for both males and females, above and below age 18.


2013 ◽  
Vol 54 (1) ◽  
pp. 109-127 ◽  
Author(s):  
Dalton Conley ◽  
Emily Rauscher

Numerous studies report gene-environment interactions, suggesting that specific alleles have different effects on social outcomes depending on environment. In all these studies, however, environmental conditions are potentially endogenous to unmeasured genetic characteristics. That is, it could be that the observed interaction effects actually reflect underlying genetic tendencies that lead individuals into certain environments. What is critical to move this literature forward is random environmental variation that we know is not correlated with innate characteristics of subjects. We exploit a natural experiment that randomizes a particular stressor—birth weight discordance within twin pairs—to address this challenge and ask: Do random differences in early environment (prenatal nutrition) moderate genetic effects on depression, delinquency, or GPA? Using Add Health data, the only consistently significant allele–birth weight interaction we reveal works in the opposite direction of Caspi et al.’s classic finding regarding the interaction of maltreatment with genetic variation in the serotonin transporter promoter. Less robust interactions found for DRD2 and MAOA are consistent with this pattern that reverses prior findings. These results do not necessarily overturn existing research but support our methodological point that gene-environment research must address endogeneity.


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