The relationship of weight suppression and dietary restraint to binge eating in bulimia nervosa

2007 ◽  
Vol 40 (7) ◽  
pp. 640-644 ◽  
Author(s):  
Michael R. Lowe ◽  
J. Graham Thomas ◽  
Debra L. Safer ◽  
Meghan L. Butryn
Keyword(s):  
Bulimia Nervosa ◽  
Binge Eating ◽  
Dietary Restraint ◽  
Weight Suppression ◽  
Relationship Of ◽  
The Relationship

The relationship between binge eating disorder and non-purging bulimia nervosa

2005 ◽  
Vol 10 (1) ◽  
pp. 8-12 ◽  
Author(s):  
Carla E. Ramacciotti ◽  
E. Coli ◽  
R. Paoli ◽  
G. Gabriellini ◽  
F. Schulte ◽  
...  
Keyword(s):  
Eating Disorder ◽  
Bulimia Nervosa ◽  
Binge Eating ◽  
Binge Eating Disorder ◽  
The Relationship

scholarly journals Weight suppression increases odds for future onset of anorexia nervosa, bulimia nervosa, and purging disorder, but not binge eating disorder

2020 ◽  
Vol 112 (4) ◽  
pp. 941-947 ◽  
Author(s):  
Eric Stice ◽  
Paul Rohde ◽  
Heather Shaw ◽  
Chris Desjardins
Keyword(s):  
Risk Factors ◽  
Eating Disorders ◽  
Anorexia Nervosa ◽  
Eating Disorder ◽  
Bulimia Nervosa ◽  
Binge Eating ◽  
Binge Eating Disorder ◽  
Purging Disorder ◽  
Weight Suppression ◽  
Current Weight

ABSTRACT Background Eating disorders affect 13% of females and contribute to functional impairment and mortality, but few studies have identified risk factors that prospectively correlate with future onset of anorexia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and purging disorder (PD). Identifying risk factors specific to each eating disorder is critical for advancing etiologic knowledge and designing effective prevention programs. Objectives This study examined whether weight suppression (the difference between a person's highest past weight at their adult height and their current weight) correlates with future onset of AN, BN, BED, and PD. Methods Data from 1165 young women with body image concerns (mean ± SD age: 21.9 ± 6.4 y) who completed annual diagnostic interviews over a 3-y follow-up period were examined. Logistic regression models evaluated the relation of baseline weight suppression to onset risk of each eating disorder controlling for age, dietary restraint, and intervention condition. Results Elevated weight suppression predicted future onset of AN (OR: 1.36; 95% CI: 1.03, 1.80), BN (OR: 1.34; 95% CI: 1.11, 1.62), PD (OR: 1.46; 95% CI: 1.23, 1.74), and any eating disorder (OR: 1.32; 95% CI: 1.12, 1.56), but not BED (OR: 1.10; 95% CI: 0.89, 1.37). Highest past weight correlated with future onset of BN and PD but not onset of AN, BED, or any eating disorder, and baseline current weight was inversely related to future AN onset only, implying that women with the largest difference between their highest past weight and current weight are at greatest risk of eating disorders. Conclusions The results provide novel evidence that weight suppression correlates with future onset of eating disorders characterized by dietary restriction or compensatory weight control behaviors and suggest weight-suppressed women constitute an important risk group to target with selective prevention programs. These trials were registered at clinicaltrials.gov as NCT01126918 and NCT01949649.

Genetic epidemiology of binging and vomiting

1998 ◽  
Vol 173 (1) ◽  
pp. 75-79 ◽  
Author(s):  
Patrick F. Sullivan ◽  
Cynthia M. Bulik ◽  
Kenneth S. Kendler
Keyword(s):  
Environmental Factors ◽  
Bulimia Nervosa ◽  
Complex Traits ◽  
Population Based ◽  
History Of ◽  
Best Fitting ◽  
Sources Of Variation ◽  
Using Data ◽  
Relationship Of ◽  
The Relationship

BackgroundBulimia nervosa is typically defined as the combination of the behaviours of binging and vomiting. We sought to clarify the relationship of these behaviours from a genetic epidemiological perspective.MethodUsing data on the lifetime history of binging and vomiting from a personally interviewed population-based sample of female twins (n = 1897), we applied bivariate twin modelling to estimate the sources of variation for these traits.ResultsThe association between having ever binged (23.6%) and having ever induced vomiting (4.8%) was very strong (odds ratio=8.78, P < 0.0001). The best-fitting model indicated that lifetime binging and vomiting were both heritable (46% and 72%) and influenced by individual-specific environmental factors (54% and 28%). The overlap between the genetic (ra = 0.74) and individual-specific environmental factors (re = 0.48) for the two traits was substantial. No violations of the equal environment assumption were evident.ConclusionsIncluding binging and vomiting under the rubric of bulimia nervosa appears to be appropriate. Our data are consistent with the identification of binging and vomiting as complex traits resulting from the interplay of multiple genes and individual-specific environmental influences. In contrast to ‘environmentalist’ theories, our results suggest that genetic influences may be of particular relevance to the aetiology of binging and vomiting.

scholarly journals Early response to antidepressant treatment in bulimia nervosa

2009 ◽  
Vol 40 (6) ◽  
pp. 999-1005 ◽  
Author(s):  
R. Sysko ◽  
N. Sha ◽  
Y. Wang ◽  
N. Duan ◽  
B. T. Walsh
Keyword(s):  
Bulimia Nervosa ◽  
Binge Eating ◽  
Operating Characteristic ◽  
Antidepressant Treatment ◽  
Antidepressant Medication ◽  
Roc Curves ◽  
Early Response ◽  
Symptom Change ◽  
The Us ◽  
The Relationship

BackgroundBulimia nervosa (BN) is a serious psychiatric disorder characterized by frequent episodes of binge eating and inappropriate compensatory behavior. Numerous trials have found that antidepressant medications are efficacious for the treatment of BN. Early response to antidepressant treatment, in the first few weeks after medication is initiated, may provide clinically useful information about an individual's likelihood of ultimately benefitting or not responding to such treatment. The purpose of this study was to examine the relationship between initial and later response to fluoxetine, the only antidepressant medication approved by the US Food and Drug Administration (FDA) for the treatment of BN, with the goal of developing guidelines to aid clinicians in deciding when to alter the course of treatment.MethodData from the two largest medication trials conducted in BN (n=785) were used. Receiver operating characteristic (ROC) curves were constructed to assess whether symptom change during the first several weeks of treatment was associated with eventual non-response to fluoxetine at the end of the trial.ResultsEventual non-responders to fluoxetine could be reliably identified by the third week of treatment.ConclusionsPatients with BN who fail to report a ⩾60% decrease in the frequency of binge eating or vomiting at week 3 are unlikely to respond to fluoxetine. As no reliable relationships between pretreatment characteristics and eventual response to pharmacotherapy have been identified for BN, early response is one of the only available indicators to guide clinical management.

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