Relationship between portal blood flow measured by image-directed doppler ultrasonography and hepatic blood flow measured by indocyanine green constant infusion in patients with cirrhosis

1995 ◽  
Vol 23 (5) ◽  
pp. 297-303 ◽  
Author(s):  
Massimo Bolognesi ◽  
David Sacerdoti ◽  
Carlo Merkel ◽  
Angelo Gatta
Keyword(s):  
Blood Flow ◽  
Indocyanine Green ◽  
Hepatic Blood Flow ◽  
Doppler Ultrasonography ◽  
Portal Blood Flow ◽  
Portal Blood ◽  
Constant Infusion

Portal blood flow, effective hepatic blood flow, and outcome after partial portal decompression

2004 ◽  
Vol 117 (1) ◽  
pp. 64-70 ◽  
Author(s):  
Alexander S Rosemurgy ◽  
Donald P Thometz ◽  
Emanuel E Zervos
Keyword(s):  
Blood Flow ◽  
Hepatic Blood Flow ◽  
Portal Blood Flow ◽  
Portal Blood ◽  
Portal Decompression

Increase in hepatic blood flow during early sepsis is due to increased portal blood flow

1991 ◽  
Vol 261 (6) ◽  
pp. R1507-R1512 ◽  
Author(s):  
P. Wang ◽  
Z. F. Ba ◽  
I. H. Chaudry
Keyword(s):  
Blood Flow ◽  
Cardiac Output ◽  
Hepatic Blood Flow ◽  
Portal Blood Flow ◽  
Portal Blood ◽  
Arterial Blood Flow ◽  
Blood Flows ◽  
Arterial Blood ◽  
Total Hepatic Blood Flow ◽  
Early Sepsis

Although hepatic blood flow increases significantly during early sepsis [as produced by cecal ligation and puncture (CLP)], it is not known whether this is due to the increase in portal or hepatic arterial blood flows. To study this, rats were subjected to CLP, after which they and sham-operated rats received either 3 or 6 ml normal saline/100 g body wt subcutaneously (i.e., all rats received crystalloid therapy). Blood flow in various organs was determined by using a radioactive microsphere technique at 5 and 20 h after CLP or sham operation. Portal blood flow was calculated as the sum of blood flows to the spleen, pancreas, gastrointestinal tract, and mesentery. Total hepatic blood flow was the sum of portal blood flow and hepatic arterial blood flow. A significant increase in portal blood flow and in total hepatic blood flow was observed at 5 h after CLP (i.e., early sepsis), and this was not altered by doubling the volume of crystalloid resuscitation after the induction of sepsis. In contrast, hepatic arterial blood flow during early sepsis was found to be similar to control; however, it was significantly reduced in late sepsis (i.e., 20 h after CLP). Cardiac output was significantly higher than the control in early sepsis. However, even in late sepsis, cardiac output and total hepatic blood flow were not significantly different from controls. These results indicate that the increased total hepatic blood flow during early hyperdynamic sepsis is solely due to the increased portal blood flow.

The Effect of Partial Portal Decompression on Portal Blood Flow and Effective Hepatic Blood Flow in Man: A Prospective Study

1995 ◽  
Vol 59 (6) ◽  
pp. 627-630 ◽  
Author(s):  
Alexander S. Rosemurgy ◽  
Earl W. McAllister ◽  
Constantine V. Godellas ◽  
Sarah E. Goode ◽  
Michael H. Albrink ◽  
...  
Keyword(s):  
Blood Flow ◽  
Prospective Study ◽  
Hepatic Blood Flow ◽  
Portal Blood Flow ◽  
Portal Blood ◽  
Portal Decompression ◽  
A Prospective Study

scholarly journals Evaluation of portal blood flow using transcutaneous and intraoperative Doppler ultrasonography in dairy cows with fatty liver

2011 ◽  
Vol 94 (6) ◽  
pp. 2964-2971 ◽  
Author(s):  
A. Starke ◽  
S. Schmidt ◽  
A. Haudum ◽  
T. Scholbach ◽  
P. Wohlsein ◽  
...  
Keyword(s):  
Blood Flow ◽  
Fatty Liver ◽  
Dairy Cows ◽  
Doppler Ultrasonography ◽  
Portal Blood Flow ◽  
Portal Blood

Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitation

1992 ◽  
Vol 263 (6) ◽  
pp. G895-G900 ◽  
Author(s):  
P. Wang ◽  
Z. F. Ba ◽  
I. H. Chaudry
Keyword(s):  
Blood Flow ◽  
Cardiac Output ◽  
Hepatic Blood Flow ◽  
Portal Blood Flow ◽  
Portal Blood ◽  
Arterial Blood Flow ◽  
Shed Blood ◽  
Hepatic Arterial Blood Flow ◽  
Arterial Blood ◽  
Small Intestinal

Although ATP-MgCl2 administration after hemorrhage and resuscitation restores the decreased hepatic blood flow, it is not known whether this is due to the increase in portal blood flow or hepatic arterial blood flow. To study this, rats underwent a midline laparotomy (i.e., trauma induced) and were bled to and maintained at a mean arterial pressure of 40 mmHg until 40% of maximal shed blood volume was returned in the form of Ringer lactate (RL). The animals were resuscitated with four times the volume of the shed blood with RL, during and after which ATP-MgCl2 (50 mumol/kg body wt) or an equal volume of normal saline was infused intravenously over 95 min. Cardiac output and organ blood flow were determined by 85Sr-labeled microspheres at 90 min after the completion of resuscitation. The results indicate that portal blood flow and total hepatic blood flow decreased significantly after hemorrhage and resuscitation. ATP-MgCl2 treatment, however, restored these parameters to sham values. In contrast, hepatic arterial blood flow did not change significantly after either hemorrhage and resuscitation or ATP-MgCl2 infusion. Moreover, the depressed cardiac output was normalized and coronary blood flow was higher than shams after ATP-MgCl2 treatment. Unlike small intestinal blood flow, blood flows to the stomach, spleen, pancreas, mesentery, and cecum were not markedly affected with ATP-MgCl2 infusion. Thus the restoration of hepatic blood flow with ATP-MgCl2 treatment under such conditions is due to the increased portal blood flow, i.e., solely due to the increased small intestinal blood flow.

scholarly journals Hepatofugal Portal Venous Flow: From Normal to Pathological

2019 ◽  
pp. 1-4
Author(s):  
Anca Dragean ◽  
Emmanuel Coche ◽  
Margaux Collard ◽  
Marin Halut
Keyword(s):  
Blood Flow ◽  
Patient Care ◽  
Doppler Ultrasonography ◽  
Reverse Flow ◽  
Imaging Techniques ◽  
Portal Blood Flow ◽  
Portal Blood ◽  
Portal Venous Flow ◽  
Venous Flow ◽  
Flow Detection

Whether segmental or diffuse, a hepatofugal blood flow is almost always pathological. Over the years, Doppler ultrasonography has retained its position as one of the most accessible and physiological imaging techniques to evaluate the direction of the portal blood flow. Detection of a reverse flow is important as it may change patient care and outcome.

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