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Ultrastructural Cytopiasmic Changes of Adrenal Cortex During Pregnancy

Proceedings, annual meeting, Electron Microscopy Society of America ◽

10.1017/s0424820100063408 ◽

1969 ◽

Vol 27 ◽

pp. 298-299

Author(s):

T. M. Murad ◽

Karen Israel ◽

Jack C. Geer

Keyword(s):

Adrenal Gland ◽

Steroid Hormones ◽

Adrenal Cortex ◽

Lipid Droplets ◽

Plasma Cholesterol ◽

Adrenal Steroids ◽

Dynamic Changes ◽

Cortical Cells ◽

Acetyl Coenzyme A ◽

Adrenal Cortical Cells

Adrenal steroids are normally synthesized from acetyl coenzyme A via cholesterol. Cholesterol is also shown to enter the adrenal gland and to be localized in the lipid droplets of the adrenal cortical cells. Both pregnenolone and progesterone act as intermediates in the conversion of cholesterol into steroid hormones. During pregnancy an increased level of plasma cholesterol is known to be associated with an increase of the adrenal corticoid and progesterone. The present study is designed to demonstrate whether the adrenal cortical cells show any dynamic changes during pregnancy.

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An adrenocortical oncocytoma with unusual mitochondrial inclusions and cytoplasmic crystals

Proceedings, annual meeting, Electron Microscopy Society of America ◽

10.1017/s0424820100168980 ◽

1994 ◽

Vol 52 ◽

pp. 250-251

Author(s):

W.T. Gunning ◽

G.D. Haselhuhn ◽

E.R. Phillips ◽

S.H. Selman

Keyword(s):

Adrenal Gland ◽

Salivary Glands ◽

Mitotic Activity ◽

Diagnostic Criteria ◽

Vascular Invasion ◽

Benign Neoplasm ◽

Benign Tumors ◽

Nuclear Morphology ◽

Case Presentation ◽

Microscopic Evidence

Within the last few years, adrenal cortical tumors with features concordant with the diagnostic criteria attributed to oncocytomas have been reported. To date, only nine reported cases exist in the literature. This report is the tenth case presentation of a presumptively benign neoplasm of the adrenal gland with a rare differentiation. Oncocytomas are well recognized benign tumors of the thyroid, parathyroid, and salivary glands and of the kidney. Other organs also give rise to these types of tumors, however with less frequency than the former sites. The characteristics generally used to classify a tumor as an oncocytoma include the following criteria: the tumor is 1) usually a solitary circumscribed mass with no gross nor microscopic evidence of metastasis (no tissue nor vascular invasion), 2) fairly bland in terms of mitotic activity and nuclear morphology, and 3) composed of large eosinophillic cells in which the cytoplasm is packed full of mitochondria (Figure 1).

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Successful surgical treatment of a giant right atrial myxoma associated with left sided adrenal gland tumour

The Thoracic and Cardiovascular Surgeon ◽

10.1055/s-0029-1246660 ◽

2010 ◽

Vol 58 (S 01) ◽

Author(s):

W Bedda ◽

U Walle ◽

W Jork ◽

J Paula ◽

N Doll

Keyword(s):

Surgical Treatment ◽

Adrenal Gland ◽

Atrial Myxoma ◽

Right Atrial ◽

Right Atrial Myxoma ◽

Gland Tumour ◽

Adrenal Gland Tumour

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Total minimal invasive access for a pancoast tumor with a metastasis of the adrenal gland

Zentralblatt für Chirurgie - Zeitschrift für Allgemeine Viszeral- Thorax- und Gefäßchirurgie ◽

10.1055/s-0036-1587489 ◽

2016 ◽

Vol 141 (S 01) ◽

Author(s):

G Seebacher ◽

J Tripsky ◽

E Stubenberger

Keyword(s):

Adrenal Gland ◽

Minimal Invasive ◽

Pancoast Tumor

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Contribution of Adrenaline to the Fibrinolytic Activity Evoked by E. Coli Endotoxin in the Rat

Thrombosis and Haemostasis ◽

10.1055/s-0038-1665254 ◽

1983 ◽

Vol 50 (02) ◽

pp. 557-559 ◽

Cited By ~ 3

Author(s):

J F Fracasso ◽

A M Rothschild

Keyword(s):

Fatty Acid ◽

Adrenal Gland ◽

Intravenous Injection ◽

Fibrinolytic Activity ◽

Blocking Agent ◽

E Coli ◽

Adrenergic Blocking

SummaryIntravenous injection of E. coli endotoxin (ETX), of adrenaline (AD) or of carbamylcholine (CBCH), caused fibrinolytic activity (FA), directly detectable on plasminogen-rich fibrin plates, to appear in the plasma of the rat. Adrenodemedul- lation abolished responses to ETX or CBCH, but enhanced those to AD. Rats given ETX exhibited marked hypotension, followed by a compensatory phase of normotension abolished by adrenodemedullation and significantly attenuated by phenoxy- benzamine, an a-adrenergic blocking agent which however failed to block FA caused by either ETX or AD. Aspirin, but not indomethacin, inhibited FA evoked by ETX, AD or CBCH. These results suggest that FA evoked by ETX in the rat is caused by AD released from the adrenal gland and does involve the fatty acid cyclooxygenase system.

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