Fluid Shifts and Loss

2017 ◽  
pp. 57-97
Author(s):  
William Thornton ◽  
Frederick Bonato
Keyword(s):  
Fluid Shifts

Infusion of very hypertonic saline to bled rats: Membrane potentials and fluid shifts

1985 ◽  
Vol 38 (2) ◽  
pp. 180-186 ◽  
Author(s):  
Shin-Ichi Nakayama ◽  
George C. Kramer ◽  
Richard C. Carlsen ◽  
James W. Holcroft
Keyword(s):  
Hypertonic Saline ◽  
Membrane Potentials ◽  
Fluid Shifts

EXTRACELLULAR FLUID SHIFTS IN ENDOTOXIN SHOCK

1967 ◽  
Vol 7 (Sup 6) ◽  
pp. 884-888 ◽  
Author(s):  
BENJAMIN F. RUSH ◽  
ROBERT SPARKS
Keyword(s):  
Extracellular Fluid ◽  
Endotoxin Shock ◽  
Fluid Shifts

scholarly journals Anaesthetic concerns for large intracranial cyst excision: expect the unexpected!

2012 ◽  
Vol 2 (2) ◽  
pp. 50 ◽  
Author(s):  
Uma Hariharan Hariharan ◽  
Rakesh Garg ◽  
Alka Gupta ◽  
Seema Wasnik ◽  
Mridula Pawar
Keyword(s):  
Blood Loss ◽  
Hydatid Cyst ◽  
Cystic Lesion ◽  
Neurosurgical Procedures ◽  
Venous Cannulation ◽  
Cyst Excision ◽  
Massive Blood Loss ◽  
Fluid Shifts ◽  
The Brain ◽  
Space Occupying Lesion

Neurosurgical procedures in infants poses various challenges such as difficulty in venous cannulation, securing invasive vascular lines, difficult airway, controlling intra-cranial tension, managing large fluid shifts to positioning- related issues and temperature maintenance. We hereby present an case of a large intracranial space occupying lesion, suspected to be a hydatid cyst, which later turned out to be an infected ventricular cyst and intraoperative problems. A thorough preparation prior to operation of infected cystic lesion of the brain is required including anticipation of massive blood loss and its management. A slow decompression of the large cystic lesion should be done.

Fluid Shifts After Burn Injury

1975 ◽  
Vol 68 (7) ◽  
pp. 887-893 ◽  
Author(s):  
CARL JELENKO III
Keyword(s):  
Burn Injury ◽  
Fluid Shifts

scholarly journals Assessment of Human Hemodynamics under Hyper- and Microgravity: Results of two Aachen University Parabolic Flight Experiments

10.14311/958 ◽  
2007 ◽  
Vol 47 (4-5) ◽  
Author(s):  
N. Blanik ◽  
M. Hülsbusch ◽  
M. Herzog ◽  
C. R. Blazek
Keyword(s):  
Parabolic Flight ◽  
Lower Extremities ◽  
Joint Project ◽  
Fluid Shifts ◽  
Optoelectronic Sensor ◽  
German Aerospace ◽  
Micro Gravity

Astronauts complain about fluid shifts from their lower extremities to their head caused by weightlessness during their flight into space. For a study of this phenomenon, RWTH Aachen University and Charité University Berlin participated in a joint project on two parabolic flight campaigns of the German Aerospace Centre (DLR) in September 2005 and June 2006. During these campaigns, the characteristics of the rapid fluid shifts during hyper- and micro gravity were measured by a combination of PPG and PPGI optoelectronic sensor concepts. 

scholarly journals Fluid Shifts With Acute Exposure To Normobaric And Hypobaric Hypoxia

2021 ◽  
Vol 53 (8S) ◽  
pp. 353-353
Author(s):  
Larry Robins ◽  
Brent Ruby ◽  
Walter Hailes ◽  
Christopher Collins ◽  
Dustin Slivka
Keyword(s):  
Hypobaric Hypoxia ◽  
Acute Exposure ◽  
Fluid Shifts

Hemodynamic Changes and Fluid Shifts After Large-Volume Fluid Infiltration

2010 ◽  
Vol 64 (1) ◽  
pp. 83-88 ◽  
Author(s):  
Selahattin Ozmen ◽  
Krzysztof Kusza ◽  
Betul G. Ulusal ◽  
Landon Pryor ◽  
Maria Siemionow ◽  
...  
Keyword(s):  
Large Volume ◽  
Hemodynamic Changes ◽  
Fluid Infiltration ◽  
Fluid Shifts

Endothelium-dependent vasodilation of cerebral arteries is altered with simulated microgravity through nitric oxide synthase and EDHF mechanisms

2006 ◽  
Vol 101 (1) ◽  
pp. 348-353 ◽  
Author(s):  
Rhonda D. Prisby ◽  
M. Keith Wilkerson ◽  
Elke M. Sokoya ◽  
Robert M. Bryan ◽  
Emily Wilson ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Methyl Ester ◽  
Nitric Oxide Synthase ◽  
Cerebral Perfusion ◽  
Simulated Microgravity ◽  
Cerebral Arteries ◽  
Caveolin 1 ◽  
Fluid Shifts ◽  
Oxide Synthase

Cephalic elevations in arterial pressure associated with microgravity and prolonged bed rest alter cerebrovascular autoregulation in humans. Using the head-down tail-suspended (HDT) rat to chronically induce headward fluid shifts and elevate cerebral artery pressure, previous work has likewise shown cerebral perfusion to be diminished. The purpose of this study was to test the hypothesis that 2 wk of HDT reduces cerebral artery vasodilation. To test this hypothesis, dose-response relations for endothelium-dependent (2-methylthioadenosine triphosphate and bradykinin) and endothelium-independent (nitroprusside) vasodilation were determined in vitro in middle cerebral arteries (MCAs) from HDT and control rats. All in vitro measurements were done in the presence and absence of the nitric oxide synthase inhibitor NG-nitro-l-arginine methyl ester (10−5 M) and cyclooxygenase inhibitor indomethacin (10−5 M). MCA caveolin-1 protein content was measured by immunoblot analysis. Endothelium-dependent vasodilation to 2-methylthioadenosine triphosphate and bradykinin were both lower in MCAs from HDT rats. These lower vasodilator responses were abolished with NG-nitro-l-arginine methyl ester but were unaffected by indomethacin. In addition, HDT was associated with lower levels of MCA caveolin-1 protein. Endothelium-independent vasodilation was not altered by HDT. These results indicate that chronic cephalic fluid shifts diminish endothelium-dependent vasodilation through alterations in the endothelial nitric oxide synthase signaling mechanism. Such decrements in endothelium-dependent vasodilation of cerebral arteries could contribute to the elevations in cerebral vascular resistance and reductions in cerebral perfusion that occur after conditions of simulated microgravity in HDT rats.

Continuous venovenous hemodialysis

1994 ◽  
Vol 3 (2) ◽  
pp. 92-99 ◽  
Author(s):  
BL Strohschein ◽  
DM Caruso ◽  
KA Greene
Keyword(s):  
Renal Failure ◽  
Acute Renal Failure ◽  
Hemodynamic Instability ◽  
Cerebellar Hemorrhage ◽  
Renal Replacement Therapies ◽  
Surgical Evacuation ◽  
Fluid Shifts ◽  
Continuous Venovenous Hemodialysis ◽  
Dialysis Disequilibrium Syndrome ◽  
Continuous Hemodialysis

Hemodialysis and peritoneal dialysis are the main renal replacement therapies for patients with acute renal failure. These patients are often unable to tolerate drastic fluid shifts and other complications of conventional dialysis. Continuous hemodialysis, however, provides protection from the hemodynamic consequences and osmotic stressors of conventional dialysis and is rapidly becoming the treatment of choice for critically ill patients. We present a case in which a patient with a spontaneous cerebellar hemorrhage developed acute renal failure. Surgical evacuation was not an option. Clinical management included the use of continuous venovenous hemodialysis, which is described in the setting of a patient with a posterior fossa mass. The risks of anticoagulation, hemodynamic instability, and development of dialysis disequilibrium syndrome are discussed.

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