Hormone Replacement Therapy in Hypertensive, Postmenopausal Women: Role of the Gynecologist

1994 ◽  
pp. 45-51 ◽  
Author(s):  
Karen Bradshaw
Keyword(s):  
Hormone Replacement Therapy ◽  
Replacement Therapy ◽  
Postmenopausal Women ◽  
Hormone Replacement

Management of Corticosteroid-Induced Osteoporosis

1998 ◽  
Vol 4 (2) ◽  
pp. 52-56 ◽  
Author(s):  
R M Francis
Keyword(s):  
Hormone Replacement Therapy ◽  
Bone Density ◽  
Replacement Therapy ◽  
Postmenopausal Women ◽  
Anabolic Steroids ◽  
Hormone Replacement ◽  
Increased Risk ◽  
Fluoride Salts ◽  
Major Abnormality

The pathogenesis of corticosteroid-induced osteoporosis is different from postmenopausal osteoporosis in that the major abnormality is a reduction in bone formation. Corticosteroids result in a 10–15% overall bone loss leading to an increased risk of fracture. Patients on corticosteroids also appear to fracture at a higher bone density than other postmenopausal women. It may therefore be appropriate to start treatment for osteoporosis in corticosteroid-treated patients at a higher bone density than for other postmenopausal women. The various treatments that have been employed are discussed, including calcium and vitamin D, calcitriol, hormone replacement therapy, bisphosphonates, calcitonin, anabolic steroids and fluoride salts. Bisphosphonates are the most extensively investigated treatments. The use of hormone replacement therapy should also be encouraged. The role of other treatments such as calcitriol, calcitonin and fluoride salts remains uncertain.

Effect of exogenous melatonin on vascular reactivity and nitric oxide in postmenopausal women: role of hormone replacement therapy

2001 ◽  
Vol 54 (2) ◽  
pp. 261-266 ◽  
Author(s):  
Angelo Cagnacci ◽  
Serenella Arangino ◽  
Marco Angiolucci ◽  
Gian Benedetto Melis ◽  
Fabio Facchinetti ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Hormone Replacement Therapy ◽  
Replacement Therapy ◽  
Postmenopausal Women ◽  
Vascular Reactivity ◽  
Hormone Replacement ◽  
Exogenous Melatonin
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