Die Affective Neuroscience Personality Scales: eine Orientierung bei der Erforschung der molekularen Grundlagen von interindividuellen Differenzen in Emotionalität

2017 ◽  
pp. 7-11
Author(s):  
Christian Montag
Keyword(s):  
Affective Neuroscience ◽  
Affective Neuroscience Personality Scales

Affective Neuroscience Personality Scales—Short Version

2012 ◽  
Author(s):  
Jean-Baptiste Pingault ◽  
Bruno Falissard ◽  
Sylvana Côté ◽  
Sylvie Berthoz
Keyword(s):  
Short Version ◽  
Affective Neuroscience ◽  
Affective Neuroscience Personality Scales

scholarly journals A Serbian version of the ANPS and its link to the five-factor model of personality

2019 ◽  
Vol 1 (1) ◽  
pp. 303-316 ◽  
Author(s):  
Christian Montag ◽  
Kenneth L. Davis ◽  
Ljiljana B. Lazarevic ◽  
Goran Knezevic
Keyword(s):  
Factor Model ◽  
Five Factor Model ◽  
Self Report ◽  
Affective Neuroscience ◽  
Serbian Version ◽  
Affective Neuroscience Personality Scales ◽  
Different Cultures ◽  
First Time ◽  
Emotional Systems ◽  
Report Measure

AbstractThis short communication presents a Serbian version of the Affective Neuroscience Personality Scales (ANPS). The ANPS is a self-report measure assessing individual differences in primary emotional systems as derived from Jaak Panksepp’s Affective Neuroscience Theory. As a recent work by Montag & Panksepp (2017a) confirmed the original demonstration of strong associations between primary emotions and the Five-Factor Model of Personality (Davis et al., 2003) across different cultures (USA, Germany, China), we replicated these findings in a Serbian sample. Moreover, following the idea of a recent commentary of Di Domencio & Ryan (2017) on Montag & Panksepp’s (2017a), we present for the first time detailed associations between Five-Factor Model facets as assessed with the NEO-PI-R and primary emotions.

Les échelles neuroaffectives de personnalité : développement de l’outil et premières données cliniques

2014 ◽  
Vol 29 (S3) ◽  
pp. 559-560
Author(s):  
S. Berthoz
Keyword(s):  
Affective Neuroscience ◽  
Santé Physique ◽  
De Se ◽  
Régulation Émotionnelle ◽  
Affective Neuroscience Personality Scales ◽  
Troubles Psychiatriques

L’idée que certaines dimensions de la personnalité et du fonctionnement affectif prédisposent à l’émergence de comportements à risque pour la santé physique et/ou soient des facteurs de vulnérabilité pour différents troubles psychiatriques n’est pas nouvelle. Dans la lignée de ce courant, plusieurs instruments auto-rapportés de l’évaluation de la personnalité et/ou du fonctionnement socio-affectif ont été développés. Toutefois, la capacité de ces instruments à rendre compte des mécanismes neurobiologiques sous-jacents reste limitée, et le fait qu’ils constituent des reflets pertinents de mécanismes génétiques et cérébraux spécifiques est de plus en plus discuté [1]. Les échelles de personnalité développées par J. Panksepp et al. pallieraient cette limite. Plutôt que de se baser sur un lexique descriptif du ressenti émotionnel, leur construction repose sur les avancées dans le domaine des neurosciences affectives et l’identification des circuits cérébraux et neuromédiateurs impliqués dans la réaction et la régulation émotionnelle [2]. Ces « échelles neuroaffectives de personnalité » (Affective Neuroscience Personality Scales [ANPS]) permettraient l’évaluation d’endophénotypes émotionnels. Les ANPS rendraient compte du fonctionnement et de l’équilibre entre 6 systèmes émotionnels primaires (3 positifs : la maternance, la jovialité, l’exploration ; 3 négatifs : la peur, la colère et la tristesse). Leur élaboration, validation en langue française ainsi que les données de la littérature émergente en faveur de leur utilisation seront présentées. Les pistes proposées pour en améliorer les qualités psychométriques seront discutées ([3,4]).

Functional connectivity in the resting brain as biological correlate of the Affective Neuroscience Personality Scales

NeuroImage ◽  
2017 ◽  
Vol 147 ◽  
pp. 423-431 ◽  
Author(s):  
Nadja Deris ◽  
Christian Montag ◽  
Martin Reuter ◽  
Bernd Weber ◽  
Sebastian Markett
Keyword(s):  
Functional Connectivity ◽  
Affective Neuroscience ◽  
Affective Neuroscience Personality Scales

A brief form of the Affective Neuroscience Personality Scales.

2013 ◽  
Vol 25 (3) ◽  
pp. 826-843 ◽  
Author(s):  
Frederick S. Barrett ◽  
Richard W. Robins ◽  
Petr Janata
Keyword(s):  
Affective Neuroscience ◽  
Affective Neuroscience Personality Scales

Cholecystokinin B receptor gene polymorphism (rs2941026) is associated with anxious personality and suicidal thoughts in a longitudinal study

2021 ◽  
pp. 1-33
Author(s):  
Aneth Lvovs ◽  
Denis Matrov ◽  
Triin Kurrikoff ◽  
Toomas Veidebaum ◽  
Jaanus Harro
Keyword(s):  
Longitudinal Study ◽  
Gene Polymorphism ◽  
Trait Anxiety ◽  
State Anxiety ◽  
Receptor Gene ◽  
Affective Neuroscience ◽  
Suicidal Thoughts ◽  
Receptor Gene Polymorphism ◽  
Trait And State Anxiety ◽  
Affective Neuroscience Personality Scales

Abstract Objective: Cholecystokinin is a neuropeptide with a role in the neurobiology of adaptive behaviour that is implicated in anxiety disorders, while the underlying mechanisms currently remain insufficiently explained. The rs2941026 variation in the cholecystokinin B receptor gene has previously been associated with trait anxiety. Our aim was to investigate associations between the CCKB receptor gene polymorphism rs2941026 with anxiety, personality, depressiveness, and suicidality in a longitudinal study of late adolescence and early adulthood. Methods: We used reports on trait and state anxiety, depressiveness and suicidal thoughts, as well as Affective Neuroscience Personality Scales, from the two birth cohorts of the Estonian Children Personality, Behaviour and Health Study. We measured associations between the CCKBR gene rs2941026 and anxiety-related phenotypes both longitudinally and cross-sectionally at ages 15, 18, 25 and 33. Results: Homozygosity for both alleles of the CCKBR rs2941026 was associated with higher trait and state anxiety in the longitudinal analysis. Cross-sectional comparisons were statistically significant at ages 18 and 25 for trait anxiety and at ages 25 and 33 for state anxiety. Higher depressiveness and suicidal thoughts were associated with the A/A genotype at age 18. Additionally, homozygosity for the A-allele was related to higher FEAR and SADNESS in the Affective Neuroscience Personality Scales. The genotype effects were more apparent in females, who displayed higher levels of negative affect overall. Conclusions: CCKBR genotype is persistently associated with negative affect in adolescence and young adulthood. The association of the CCKBR rs2941026 genotype with anxiety-related phenotypes is more pronounced in females.

Personality and biological markers of creativity

2005 ◽  
Vol 19 (2) ◽  
pp. 83-95 ◽  
Author(s):  
M. Reuter ◽  
J. Panksepp ◽  
N. Schnabel ◽  
N. Kellerhoff ◽  
P. Kempel ◽  
...  
Keyword(s):  
Test Scores ◽  
Sensation Seeking ◽  
Fluid Intelligence ◽  
Biological Marker ◽  
Intelligence Test ◽  
Affective Neuroscience ◽  
Crystallized Intelligence ◽  
Main Effect ◽  
Affective Neuroscience Personality Scales ◽  
And Biological Markers

The aim of the present study was to test (i) Eysenck's theory that psychoticism (P) should be related to creativity, (ii) whether testosterone (T), due to its association with P claimed in the literature, can be identified as a biological marker of creativity, and (iii) whether the SEEK dimension of the Affective Neuroscience Personality Scales (ANPS) was also related to creativity and to testosterone due to its relationship to Sensation Seeking. In a sample of N = 48 male and female subjects, test scores on figural, verbal, and numeric creativity were compared between high and low P‐scorers as well as between high and low SEEK‐scorers. Effects were controlled for fluid intelligence as measured by Cattell's CFT‐3 and crystallized intelligence as assessed by the Structure‐of‐Intelligence‐Test (Intelligenz‐Struktur‐Test, I‐S‐T 2000 R). Neither a main effect of P or T nor an interaction effect P×T on creativity could be obtained. Instead, SEEK was related to all components of creativity and explained more than 15% of the variance of total creativity. Moreover, significant differences in SEEK could be explained by differences in T, independently of gender. Furthermore, 39% of the variance of SEEK could be explained by the two uncorrelated indicators testosterone and creativity. Copyright © 2004 John Wiley & Sons, Ltd.

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