Echinococcus vogeli

2016 ◽  
pp. 817-817
Keyword(s):  
2014 ◽  
Vol 89 (4) ◽  
pp. 480-486 ◽  
Author(s):  
F. Almeida ◽  
F. Oliveira ◽  
R. Neves ◽  
N. Siqueira ◽  
R. Rodrigues-Silva ◽  
...  

AbstractPolycystic echinococcosis, caused by the larval stage (metacestode) of the small-sized tapeworm, Echinococcus vogeli, is an emerging parasitic zoonosis of great public health concern in the humid tropical rainforests of South and Central America. Because morphological and morphometric characteristics of the metacestode are not well known, hydatid cysts from the liver and the mesentery were examined from patients following surgical procedures. Whole mounts of protoscoleces with rostellar hooks were examined under light and confocal laser scanning microscopy. Measurements were made of both large and small hooks, including the total area, total length, total width, blade area, blade length, blade width, handle area, handle length and handle width. The results confirmed the 1:1 arrangement of hooks in the rostellar pad and indicated, for the first time, that the morphometry of large and small rostellar hooks varies depending upon the site of infection. Light and confocal microscopy images displayed clusters of calcareous corpuscles in the protoscoleces. In conclusion, morphological features of large and small rostellar hooks of E. vogeli are adapted to a varied environment within the vertebrate host and such morphological changes in calcareous corpuscles occur at different stages in the maturation of metacestodes.


1992 ◽  
Vol 86 (2) ◽  
pp. 151-156 ◽  
Author(s):  
U. G. Meneghelli ◽  
A. L. C. Martinelli ◽  
A. D. Bellucci ◽  
M. G. Villanova ◽  
M. A. S. Llorach Velludo ◽  
...  

2012 ◽  
Vol 42 (13-14) ◽  
pp. 1115-1118 ◽  
Author(s):  
Guilherme B. Santos ◽  
Manoel do C.P. Soares ◽  
Elisabete M. de F. Brito ◽  
André L. Rodrigues ◽  
Nilton G. Siqueira ◽  
...  

Author(s):  
J. Eckert ◽  
P. Deplazes ◽  
P. Kern

In this chapter three forms of echinococcosis in humans are described that are caused by a larval stage (metacestode) of Echinococcus multilocularis Leuckart, 1863, Echinococcus oligarthrus (Diesing, 1863) or Echinococcus vogeli Rausch and Bernstein, 1972. E. multilocularis is the causative agent of alveolar echinococcosis (AE). In the human host the metacestode of E. multilocularis behaves like a malignant tumour, characterized by infiltrative proliferation and the potential to induce serious disease. The liver is nearly exclusively the primary site of metacestode development, but metastases may by formed in adjacent and distant organs. Typically AE exhibits a chronic progressive clinical course, which finally leads to death in up to 90% of untreated patients within 10 years after diagnosis. An undefined proportion of cases are abortive with inactivation of the parasite. Evidence has accumulated in recent years that anti-parasitic therapy with benzimidazoles (albendazole or mebendazole) over many years or lifelong, if necessary combined with interventional procedures, can inhibit disease progression and improve or stabilse the patient’s clinical condition. Radical surgery in an early stage of the infection combined with anti-parasitic therapy for two years may lead to cure. The introduction of benzimidazole therapy of AE (1977), combined with improved diagnostic and surgical procedures, has resulted in significantly increased life-expectancies of adequately treated AE patients. In highly endemic areas ultrasound population screening (partially combinated with antibody detection) has been successfully used for early detection of AE cases. Countrywide annual AE incidence rates are mostly low at approximately < 0.1 to 2.0 per 100,000 inhabitants, but they can be much higher locally. Furthermore, there are indications of emerging case numbers in some areas of Europe and Asia. In spite of relatively low case numbers, AE is a significant disease due to its severity and high costs of treatment (median costs of approximately 145,800 per case).


2017 ◽  
Vol 10 (1) ◽  
Author(s):  
Leandro Batista das Neves ◽  
Paulo Eduardo Ferlini Teixeira ◽  
Sidnei Silva ◽  
Fernanda Bittencourt de Oliveira ◽  
Daniel Daipert Garcia ◽  
...  

2016 ◽  
Vol 3 (4) ◽  
pp. 173-175 ◽  
Author(s):  
David Botero ◽  
Marcos Restrepo ◽  
Alejandro Restrepo
Keyword(s):  

2002 ◽  
Vol 97 (1) ◽  
pp. 123-126 ◽  
Author(s):  
Rosângela Rodrigues-Silva ◽  
José Resende V Peixoto ◽  
Regina Maria Figueiredo de Oliveira ◽  
Roberto Magalhães Pinto ◽  
Delir Corrêa Gomes

1987 ◽  
Vol 20 (3) ◽  
pp. 181-186 ◽  
Author(s):  
Marcelo Simão Ferreira ◽  
Ademir Rocha ◽  
Elmar Gonzaga Gonçalves ◽  
Ademar Margonari Carvalho ◽  
Sérgio de Andrade Nishioka ◽  
...  

Relata-se um caso de hidatidosepolicistica, em homem de 22 anos, clinicamente manifestada por dor no hipocôndrio direito, icterícia obstrutiva, hepatoesplenomegalia, perda de peso e, em estágio final, ascite. O diagnóstico foi após laparotomia exploradora, com biópsia hepática e peritoneal. O paciente evoluiu para o óbito, a despeito de ter sido tratado com mebendazol na dose de 1200 mg/dia, durante um ano. A necrópsia, constatou-se hidatidose do fígado, omento maior e peritônio diafragmático. Não se conseguiu identificar, à microscopia óptica, a espécie de Echinococcus envolvida; com base nos dados epidemiológicos e morfológicos disponíveis, o parasita em apreço poderia ser o Echinococcus vogeli ou o E. oligarthrus. Este parece ser o quarto caso de hidatidose policistica na literatura nacional e é, com muita probabilidade, autóctone de Minas Gerais.


1998 ◽  
Vol 59 (5) ◽  
pp. 787-790 ◽  
Author(s):  
D Basset ◽  
R Gordon ◽  
C Girou ◽  
C Hoang ◽  
A D'Alessandro ◽  
...  
Keyword(s):  

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