Genetic and Environmental Influences on Gambling and Substance Use in Early Adolescence

2014 ◽  
Vol 44 (4) ◽  
pp. 347-355 ◽  
Author(s):  
Frank Vitaro ◽  
Amy C. Hartl ◽  
Mara Brendgen ◽  
Brett Laursen ◽  
Ginette Dionne ◽  
...  
2011 ◽  
Vol 41 (9) ◽  
pp. 1907-1916 ◽  
Author(s):  
J. H. Baker ◽  
H. H. Maes ◽  
H. Larsson ◽  
P. Lichtenstein ◽  
K. S. Kendler

BackgroundGenetic and environmental factors are important in the etiology of substance use. However, little is known about the stability of these factors across development. We aimed to answer three crucial questions about this etiology that have never been addressed in a single study: (1) Is there a general vulnerability to substance consumption from early adolescence to young adulthood? (2) If so, do the genetic and environmental influences on this vulnerability change across development? (3) Do these developmental processes differ in males and females?MethodSubjects included 1480 twin pairs from the Swedish Twin Study of Child and Adolescent Development who have been followed since 1994. Prospective, self-reported regular smoking, alcohol intoxication and illicit drug use were assessed at ages 13–14, 16–17 and 19–20 years. Structural modeling was performed with the program Mx.ResultsAn underlying common factor accounted for the association between smoking, alcohol and illicit drug consumption for the three age groups. Common genetic and shared environmental effects showed substantial continuity. In general, as participants aged, the influence of the shared environment decreased, and genetic effects became more substance specific in their effect.ConclusionsThe current report answers three important questions in the etiology of substance use. The genetic and environmental risk for substance consumption is partly mediated through a common factor and is partly substance specific. Developmentally, evidence was strongest for stability of common genetic effects, with less evidence for genetic innovation. These processes seem to be the same in males and females.


2021 ◽  
Vol 51 (4) ◽  
pp. 170-174
Author(s):  
Jacquelyn L. Meyers ◽  
Jessica E. Salvatore

2018 ◽  
Vol 72 (6) ◽  
pp. 465-470 ◽  
Author(s):  
Daniel R Hale ◽  
Russell M Viner

BackgroundEducation is recognised as a strong determinant of health. Yet there is increasing concern that health in adolescence may also influence educational attainments and future life chances. We examined associations between health in early adolescence and subsequent academic and employment outcomes, exploring potential mediators of these relationships to inform intervention strategies.MethodsWe used data from the Longitudinal Study of Young People in England. Adolescent health was measured at waves 1 and 2. Outcomes included educational attainment at age 16 years and being NEET (not in education, employment or training) at age 19 years. Associations were adjusted for ethnicity, area-level deprivation and early adolescent academic attainment. Where significant associations were identified, we examined the role of hypothesised mediators including attendance and truancy, classroom behaviour, substance use and psychological distress.ResultsHealth conditions in early adolescence predicted poor subsequent education and employment outcomes (ORs ranged from 1.25 to 1.72) with the exception of long-term chronic conditions and NEET status, which were unassociated. The most consistent mediating variable was social exclusion. School behaviour, truancy and substance use were significant mediators for mental health. Long-term absences mediated associations between mental health and physical health and later outcomes.ConclusionsHealth is a key component of academic and vocational achievement. Investment in health is a way of improving life chances. The identification of key mediators such as social exclusion and truancy indicate areas where screening for health conditions and provision of targeted support could improve educational, employment and health outcomes.


2019 ◽  
Vol 29 (Supplement_4) ◽  
Author(s):  
M Bacikova-Sleskova ◽  
L Hricová ◽  
O Kalina ◽  
J Benka

Abstract Background Previous research has shown that one of the strongest predictor of smoking and alcohol consumption in early adolescence is perceived parental approval of such behaviour. The aim of the present study is to explore which parenting practices predict the probability of perceiving parental disapproval of smoking and alcohol consumption in adolescence. Methods A representative sample of 1133 early adolescents (mean age = 12.9, SD 0.77) filled in questionnaires including sociodemographic characteristics (3 items), perceived parental risk behaviour (alcohol consumption at least once a week, drunkenness once a month and daily smoking); substance specific conversations with parents (1 item); both emotional and controlling aspects of parenting (6 items), and perceived parental disapproval of adolescents’ smoking and alcohol consumption as dependent variables. Logistic regression was used to analyse the data. Results The most important predictor of perceived parental disapproval of alcohol use was behavioural control (setting rules) (OR 2.34), followed by parental non-drinking (OR 1.81) and alcohol specific communication (OR 1.28). Regarding smoking disapproval, the significant predictors were parental non-smoking (OR 1.92); behavioural control (OR 1.79); time spent with a child (OR 1.44) and lower psychological control (guild induction, pressuring, manipulation) (OR 0.69). Conclusions Decreasing adolescents’ substance use via increasing their perceived parental disapproval of such behaviour seems to be substance specific. The most salient strategy both for smoking and drinking seems to be the use of behavioural control characterized by clear rules setting and lack of parental own substance use. Key messages Decreasing adolescents’ substance use via increasing their perceived parental disapproval of such behaviour seems to be substance specific. The most salient strategy to increase perceived parental disapproval of smoking and alcohol consumption seems to be the use of behavioural control characterized by clear rules setting.


2015 ◽  
Vol 146 ◽  
pp. e262 ◽  
Author(s):  
Karen J. Derefinko ◽  
Richard J. Charnigo ◽  
Richard Milich ◽  
Donald R. Lynam

2017 ◽  
Vol 30 (4) ◽  
pp. 1355-1369 ◽  
Author(s):  
Chung Jung Mun ◽  
Thomas J. Dishion ◽  
Jenn-Yun Tein ◽  
Roy Otten

AbstractThis study examined the mediated effect of early adolescence familial context on early adulthood problematic substance use through effortful control in late adolescence. The sample consisted of a community sample of 311 adolescents and their families comprising the control group within a randomized trial intervention. Parental monitoring and parent–child relationship quality (P-C RQ) were measured annually from ages 11 to 13. Effortful control was measured by self-reports and parent and teacher reports at ages 16 to 17. Self-reports of problematic tobacco, alcohol, and marijuana use were measured at ages 18 to 19, 21 to 22, 23 to 24, and 26 to 27. Structural equation modeling was employed to test hypothesized models. Only P-C RQ was found to be significantly associated with adolescent effortful control. As expected, higher levels of adolescent effortful control were associated with lower problematic substance use through early adulthood, controlling for previous substance use levels. Mediation analyses showed that effortful control significantly mediated the relationship between P-C RQ and problematic substance use. Higher relationship quality between youth and parents in early adolescence is associated with higher effortful control, which in turn relates to a lower level of problematic substance use in early adulthood.


2011 ◽  
Vol 14 (2) ◽  
pp. 119-128 ◽  
Author(s):  
Inge L.C. van Soelen ◽  
Rachel M. Brouwer ◽  
Marieke van Leeuwen ◽  
René S. Kahn ◽  
Hilleke E. Hulshoff Pol ◽  
...  

The longitudinal stability of IQ is well-documented as is its increasing heritability with age. In a longitudinal twin study, we addressed the question to what extent heritability and stability differ for full scale (FSIQ), verbal (VIQ), and performance IQ (PIQ) in childhood (age 9–11 years), and early adolescence (age 12–14 years). Genetic and environmental influences and correlations over time were evaluated in an extended twin design, including Dutch twins and their siblings. Intelligence was measured by the Wechsler Intelligence Scale for children — Third version (WISC III). Heritability in childhood was 34% for FSIQ, 37% for VIQ, and 64% for PIQ, and increased up to 65%, 51%, and 72% in early adolescence. The influence of common environment decreased between childhood and early adolescence from explaining 43% of the phenotypic variance for FSIQ to 18% and from 42% for VIQ to 26%. For PIQ common environmental influences did not play a role, either in childhood or in early adolescence. The stability in FSIQ and VIQ across the 3-year interval (rp) was .72 for both measures and was explained by genetic and common environmental correlations across time (FSIQ, rg= .96, rc= 1.0; VIQ, rg=.78, rc= 1.0). Stability of PIQ (rp=.56) was lower and was explained by genetic influences (rg= .90). These results confirm the robust findings of increased heritability of general cognitive abilities during the transition from childhood to adolescence. Interestingly, results for PIQ differ from those for FSIQ and VIQ, in that no significant contribution of environment shared by siblings from the same family was detected.


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