Monitoring of pulmonary arterial diastolic pressure through a right ventricular pressure transducer

1995 ◽  
Vol 1 (2) ◽  
pp. 161-168 ◽  
Author(s):  
Åke Ohlsson ◽  
Tom Bennett ◽  
Rolf Nordlander ◽  
Johan Rydén ◽  
Hans Åström ◽  
...  
1996 ◽  
Vol 2 (1) ◽  
pp. 41-46 ◽  
Author(s):  
Peter P. Chuang ◽  
Robert F. Wilson ◽  
David C. Homans ◽  
Karen Stone ◽  
Torrey Bergman ◽  
...  

1982 ◽  
Vol 53 (4) ◽  
pp. 908-913 ◽  
Author(s):  
J. E. Whinnery ◽  
M. H. Laughlin

Measurements of right ventricular pressure in miniature swine were made at +Gz levels from +1 through +9 Gz. Polyethylene catheters were chronically placed in the cranial vena cava of five 2-yr-old female miniature swine (35–50 kg). The catheters were large enough to allow the introduction of a Millar pressure transducer into the venous system for placement in the right heart. The animals were fitted with an abdominal anti-G suit, restrained in a fiberglass couch, and exposed to the various +Gz levels on a centrifuge while fully conscious and unanesthetized. Right ventricular pressure and heart rate were measured during and for 2 min following 30-s exposures to each level of +Gz stress. The maximum right ventricular systolic pressure observed during +Gz was 200 Torr at +5 Gz with the maximum diastolic pressure being 88 Torr observed at +5 Gz. Mean heart rates were 200–210 beats/min at all levels of +Gz greater than or equal to +3 Gz when the animal remained stable. Mean maximum right ventricular pressures during +Gz stress were observed to increase through +5 Gz (85 Torr) and to decrease at higher levels of +Gz, indicating that through +5 Gz there is at least a partial compensation during acceleration stress. Decompensation in response to the stress began to occur during acceleration above +5 Gz with all animals decompensating during +9 Gz.


2015 ◽  
Vol 21 (10) ◽  
pp. S152
Author(s):  
Takafumi Sakamoto ◽  
Kohtaro Abe ◽  
Kazuya Hosokawa ◽  
Keiji Oi ◽  
Yasushi Mukai ◽  
...  

CHEST Journal ◽  
2007 ◽  
Vol 132 (1) ◽  
pp. 37-43 ◽  
Author(s):  
Mustafa Karamanoglu ◽  
Michael McGoon ◽  
Robert P. Frantz ◽  
Raymond L. Benza ◽  
Robert C. Bourge ◽  
...  

1988 ◽  
Vol 65 (4) ◽  
pp. 1885-1890 ◽  
Author(s):  
B. J. Grant ◽  
L. J. Paradowski ◽  
J. M. Fitzpatrick

We determined the effect of perivascular electromagnetic flow probes (EMF) on pulmonary hemodynamics in acute experiments. In seven dogs placement of the EMF on the main pulmonary artery (MPA) increased pulmonary arterial pulse pressure by 25% (17.8-21.9 cmH2O, P less than 0.005) and mean right ventricular pressure by 12% (23.2-25.9 cmH2O, P less than 0.001) but did not alter heart rate, systemic blood pressure, mean pulmonary arterial pressure, or right ventricular end-diastolic pressure. This response was not abolished by local application of lidocaine to the MPA. In three cats input impedance was calculated from measurements of pressure and flow in the MPA. Impedance was calculated with flow measured using an EMF and ultrasonic volume flow probe (USF), which avoids the constraining effect of the EMF. When flow was measured with an EMF rather than a USF, there was a significant difference in the impedance spectra (P less than 0.001), but it was only apparent in the moduli greater than six harmonics. We conclude that the EMF does affect right ventricular afterload in acute experiments and alters the measured input impedance.


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