Fetal response to maternal exercise in pregnancies with uteroplacental insufficiency

2005 ◽  
Vol 193 (3) ◽  
pp. 995-999 ◽  
Author(s):  
Vandana Chaddha ◽  
Michal J. Simchen ◽  
Lisa K. Hornberger ◽  
Victoria M. Allen ◽  
Shafagh Fallah ◽  
...  
Keyword(s):  
Maternal Exercise ◽  
Fetal Response

Fetal response to maternal exercise in pregnancies with uteroplacental vascular insufficiency

2004 ◽  
Vol 191 (6) ◽  
pp. S148
Author(s):  
Vandana Chaddha ◽  
Michel Simchen ◽  
Lisa Hornberger ◽  
Allen Victoria ◽  
Allan Coates ◽  
...  
Keyword(s):  
Vascular Insufficiency ◽  
Maternal Exercise ◽  
Fetal Response

Human maternal and fetal response to graded exercise

1985 ◽  
Vol 58 (5) ◽  
pp. 1719-1722 ◽  
Author(s):  
L. Jovanovic ◽  
A. Kessler ◽  
C. M. Peterson
Keyword(s):  
Serum Insulin ◽  
Cycle Ergometer ◽  
Maternal Serum ◽  
Third Trimester ◽  
Exercise Regimen ◽  
Graded Exercise ◽  
Fetal Movements ◽  
Maternal Exercise ◽  
Lactate Levels ◽  
Fetal Response

Six healthy active women in the third trimester of pregnancy participated in a graded exercise protocol to levels of exertion perceived to be equivalent to that of their usual exercise regimen. Fetal heart rate response (FHR) was documented by ultrasound transducer and confirmed (n = 1) by ultrasonic visualization. Resting maternal O2 consumption was 277 +/- 50 (SD) ml/min and rose to 1,132 +/- 202 ml/min at a mean final exercise intensity of 79 +/- 9 W after 12.8 +/- 1.7 min on a cycle ergometer. There was no significant change in maternal serum insulin, growth hormone, glucose, or pH values. Maternal leukocyte count, hemoglobin, and venous lactate levels rose significantly during the exercise (P less than 0.05). FHR prior to exercise was 142 +/- 4 beats/min and decreased to 84 +/- 34 beats/min during exercise. The decrease in FHR was documented within 1 min of initiating exercise in all cases. During exercise, fetal movements were not accompanied by FHR accelerations. Within 1 min following the cessation of exercise, FHR rose to 143 +/- 8 beats/min and fetal movements were accompanied by FHR accelerations. Since the recovery of FHR occurred immediately after cessation of maternal exercise, this level of maternal exercise does not appear to be harmful to the fetus.

Insulin secretion in fetal and newborn sheep.

1978 ◽  
Vol 235 (5) ◽  
pp. E467 ◽  
Author(s):  
A F Philipps ◽  
B S Carson ◽  
G Meschia ◽  
F C Battaglia
Keyword(s):  
Insulin Secretion ◽  
Plasma Insulin ◽  
Insulin Response ◽  
Induced Response ◽  
Response Curves ◽  
Fasted State ◽  
Early Insulin Response ◽  
Arterial Plasma ◽  
Fetal Response ◽  
Maternal Glucose

The relationships between arterial plasma insulin, glucose, and fructose concentrations during the fed and fasted state were studied in seven fetal lambs and their mothers. A significant correlation between insulin and glucose concentration was noted in all fetal lambs and in their mothers. Fetal sensitivity to glucose, as measured by the slopes of the insulin-response curves, was equal to that of the adult although the fetal response was shifted to the left of the maternal. Glucose infusion in four fetal lambs caused significant insulin elevations but no early insulin response (phase I). Maternal fasting caused no alteration in glucose-induced response in the fetus. Similar glucose infusions in newborn and 1-mo-old lambs demonstrated significant early-phase insulin secretion. Basal insulin to glucose ratios were consistent with an adult pattern as early as 3 days after birth.

scholarly journals Fetal Renal Echogenicity Associated with Maternal Focal Segmental Glomerulosclerosis: The Effect of Transplacental Transmission of Permeability Factor suPAR

2018 ◽  
Vol 7 (10) ◽  
pp. 324 ◽  
Author(s):  
Shirley Shuster ◽  
Ghada Ankawi ◽  
Christoph Licht ◽  
Jochen Reiser ◽  
Xuexiang Wang ◽  
...  
Keyword(s):  
Focal Segmental Glomerulosclerosis ◽  
Transplacental Transmission ◽  
Transplacental Passage ◽  
Segmental Glomerulosclerosis ◽  
Type Plasminogen Activator ◽  
Urokinase Type Plasminogen Activator ◽  
Plasminogen Activator Receptor ◽  
Maternal Fetal Transmission ◽  
Fetal Response ◽  
Echogenic Kidneys

We report a case of a pregnant woman with nephrotic syndrome due to biopsy-proven focal segmental glomerulosclerosis (FSGS) whose fetus developed echogenic kidneys and severe oligohydramnios by 27 weeks of gestation. Maternal treatment with prednisone resulted in normalization of the amniotic fluid indices and resolution of fetal renal echogenicity. The newborn was noted to have transient renal dysfunction and proteinuria, resolving by 6 weeks postpartum. The transplacental passage of permeability factors is postulated to have caused both the fetal and newborn renal presentation, with significantly elevated levels of soluble urokinase-type plasminogen activator receptor (suPAR) noted in the cord blood. This case documents the transplacental maternal-fetal transmission of suPAR, demonstrating the potential for maternal-fetal transmission of deleterious, disease-causing entities, and adds to the differential diagnosis of fetal echogenic kidneys. Further, this is the first documentation of a fetal response to maternal systemic therapy.

LEUKEMIA IN PREGNANCY AND FETAL RESPONSE TO MULTIAGENT CHEMOTHERAPY

2001 ◽  
Vol 97 (5) ◽  
pp. 809-812
Author(s):  
Wendy F. Hansen ◽  
Peter Fretz ◽  
Stephen K. Hunter ◽  
Jerome Yankowitz
Keyword(s):  
Multiagent Chemotherapy ◽  
Fetal Response ◽  
In Pregnancy
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