AS-51: Benefits from Carotid Stenting before Open Heart Surgery: A Single-Center Experience with 76 Patients

2009 ◽  
Vol 103 (9) ◽  
pp. 24B-25B
Author(s):  
Qian Yang ◽  
Xiong Jing Jiang ◽  
Yue Jin Yang ◽  
Hai Ying Wu ◽  
Run Ling Gao
Keyword(s):  
Heart Surgery ◽  
Open Heart Surgery ◽  
Carotid Stenting ◽  
Open Heart ◽  
Single Center ◽  
Single Center Experience

Outcome in octogenarians undergoing open heart surgery – a single center experience

2008 ◽  
Vol 56 (S 1) ◽  
Author(s):  
A Pritisanac ◽  
H Gulbins ◽  
A Abugameh ◽  
U Rosendahl ◽  
S Bauer ◽  
...  
Keyword(s):  
Heart Surgery ◽  
Open Heart Surgery ◽  
Open Heart ◽  
Single Center ◽  
Single Center Experience

ICU after heart surgery-back and forth again – a single center experience -

2013 ◽  
Vol 61 (S 01) ◽  
Author(s):  
M Ernst ◽  
A Reinecke ◽  
M vd Brelie ◽  
J Cremer ◽  
N Haake
Keyword(s):  
Heart Surgery ◽  
Single Center ◽  
Single Center Experience

The Hemostatic Mechanism after Open-Heart Surgery

1978 ◽  
Vol 39 (02) ◽  
pp. 474-487 ◽  
Author(s):  
E R Cole ◽  
F Bachmann ◽  
C A Curry ◽  
D Roby
Keyword(s):  
Extracorporeal Circulation ◽  
Heart Surgery ◽  
Polyacrylamide Gel Electrophoresis ◽  
Open Heart Surgery ◽  
Coagulation System ◽  
Open Heart ◽  
Time Activity ◽  
Post Surgery ◽  
Plasma Fractions ◽  
The Mean

SummaryA prospective study in 13 patients undergoing open-heart surgery with extracorporeal circulation revealed a marked decrease of the mean one-stage prothrombin time activity from 88% to 54% (p <0.005) but lesser decreases of factors I, II, V, VII and X. This apparent discrepancy was due to the appearance of an inhibitor of the extrinsic coagulation system, termed PEC (Protein after Extracorporeal Circulation). The mean plasma PEC level rose from 0.05 U/ml pre-surgery to 0.65 U/ml post-surgery (p <0.0005), and was accompanied by the appearance of additional proteins as evidenced by disc polyacrylamide gel electrophoresis of plasma fractions (p <0.0005). The observed increases of PEC, appearance of abnormal protein bands and concomitant increases of LDH and SGOT suggest that the release of an inhibitor of the coagulation system (similar or identical to PIVKA) may be due to hypoxic liver damage during extracorporeal circulation.

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