scholarly journals An unrecognized combined congenital heart defect - Bicuspid aortic valve and ventricular septal defect as a cause of acute heart failure in adulthood

Cor et Vasa ◽  
2018 ◽  
Vol 60 (5) ◽  
pp. e512-e517 ◽  
Author(s):  
Kristýna Bayerová ◽  
Gabriela Dostálová ◽  
Zuzana Hlubocká ◽  
Tomáš Paleček ◽  
Jaroslav Hlubocký ◽  
...  
Keyword(s):  
Heart Failure ◽  
Aortic Valve ◽  
Ventricular Septal Defect ◽  
Acute Heart Failure ◽  
Bicuspid Aortic Valve ◽  
Congenital Heart Defect ◽  
Congenital Heart ◽  
Septal Defect ◽  
Heart Defect

scholarly journals Atrial Fibrillation-Induced Cardiac Shock: First Manifestation of a Congenitally Corrected Transposition of the Great Arteries in a 45-Year-Old Man

2012 ◽  
Vol 2012 ◽  
pp. 1-4
Author(s):  
M. Graf ◽  
M. Zaczkiewicz ◽  
J. Torzewski ◽  
O. Zimmermann
Keyword(s):  
Heart Failure ◽  
Atrial Fibrillation ◽  
Acute Heart Failure ◽  
Congenital Heart Defect ◽  
Congenital Heart ◽  
Prior History ◽  
Heart Defect ◽  
Cardiac Shock ◽  
Congenitally Corrected Transposition ◽  
Corrected Transposition

Background.The congenitally corrected transposition of the great arteries (L-TGA) is a very rare congenital heart defect, which often remains undetected for several decades of life.Case Presentation.We report on a 45-year-old man without prior history of heart disease, presenting with cardiac shock related to a first episode of tachycardic atrial fibrillation. The diagnostic work-up identified a L-TGA as the underlying cause for acute heart failure.Discussion.L-TGA is a very rare congenital heart defect, which is characterized by an atrioventricular as well as a ventriculoarterial discordance. By this means, the physiological sequence of pulmonary and systemic circulation is still maintained. On the basis of an ongoing strain of the right ventricle, which has to carry the burden of the systemic blood pressure, after more than four decades without symptoms, acute heart failure was triggered by a tachycardic atrial fibrillation.

Disturbance in Heart Rate during Exercise Following Lntracardiac Repair: Contribution of Cardiopulmonary Bypass

1992 ◽  
Vol 4 (3) ◽  
pp. 270-280 ◽  
Author(s):  
Hélène Perrault ◽  
André Davignon ◽  
Gail Grief ◽  
Anne Fournier ◽  
Claude Chartrand
Keyword(s):  
Heart Rate ◽  
Cardiopulmonary Bypass ◽  
Ventricular Septal Defect ◽  
Congenital Heart Defect ◽  
Congenital Heart ◽  
Septal Defect ◽  
Control Group ◽  
Common Denominator ◽  
Chronotropic Response ◽  
Heart Defect

A disturbance in the chronotropic response to exercise has well documented following surgical repair of a congenital heart defect. Observations of a similar dysfunction following correction of simple atrial as well as ventricular septal defect suggest that its occurrence might be linked to a common denominator in the surgical procedures. Since cardiopulmonary bypass (CPB) is used for all initacardiac surgeries, the contribution of this factor to the abnormal response was examined. Three groups of patients von; evaluated: VSDop: operated on for a ventricular septal defect, CPB required; VSDnon-op: spontaneous closure of a congenital 73D, no surgery; and PDAoperated on for a patent ductus arteriosus, no CPB required. They were compared io a control group of healthy adolescents (C). The main, finding was that of lower submax final and maximal heart rates in VCDop disc in any other group. These results suggest that placement of cannulae resulting from-CPB could alter the control of heart rate during exercise and contribute to the abnormal chronotropic response following intracardiac repair of a congenital heart defect.

scholarly journals Significance of Atrial and Brain Natriuretic Peptide Measurements in Fetuses With Heart Failure

2021 ◽  
Vol 12 ◽  
Author(s):  
Takekazu Miyoshi ◽  
Hiroshi Hosoda ◽  
Naoto Minamino
Keyword(s):  
Heart Failure ◽  
Brain Natriuretic Peptide ◽  
Amniotic Fluid ◽  
Congenital Heart Defect ◽  
Umbilical Cord Blood ◽  
Congenital Heart ◽  
Fetal Heart ◽  
Diagnostic Biomarker ◽  
Heart Defect ◽  
Fetal Heart Failure

Fetal heart failure is mainly caused by congenital heart defect and arrhythmia. It is difficult to appropriately diagnose the severity of fetal heart failure simply by ultrasonography because the development of a fetal heart in fetoplacental circulation and how well the fetal myocardium can adapt to postnatal cardiopulmonary circulation are challenging to assess. In adult cardiology, natriuretic peptides (NPs) are the most useful biomarker of heart failure; however, studies investigating NP levels in the fetuses and amniotic fluid are quite limited. Furthermore, little is known about their production and metabolism. This review summarized the most relevant findings on NP levels in the umbilical cord blood and amniotic fluid. The findings can then extend their use as a diagnostic biomarker of heart failure in fetuses with congenital heart defect and/or arrhythmia.

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