The role of ischemic neurodegeneration of the nodose ganglia on cardiac arrest after subarachnoid hemorrhage: An experimental study

2011 ◽  
Vol 230 (1) ◽  
pp. 90-95 ◽  
Author(s):  
Mehmet D. Aydin ◽  
Ayhan Kanat ◽  
Adem Yilmaz ◽  
Murteza Cakir ◽  
Mucahit Emet ◽  
...  
2010 ◽  
Vol 152 (12) ◽  
pp. 2167-2172 ◽  
Author(s):  
Ayhan Kanat ◽  
Adem Yilmaz ◽  
Mehmet D. Aydin ◽  
Murat Musluman ◽  
Sare Altas ◽  
...  

2011 ◽  
Vol 165 (2) ◽  
pp. 163-167 ◽  
Author(s):  
Selim Kayaci ◽  
Ayhan Kanat ◽  
Mehmet Dumlu Aydin ◽  
Ahmet Murat Musluman ◽  
Mete Eseoglu ◽  
...  

2020 ◽  
Vol 52 (1) ◽  
pp. 61-66
Author(s):  
Mehmet Dumlu Aydin ◽  
Mahmut Acikel ◽  
Nazan Aydin ◽  
Muhammed Enes Aydin ◽  
Ali Ahiskalioglu ◽  
...  

Author(s):  
Osman Tanriverdi ◽  
Mehmet Dumlu Aydin ◽  
Mehmet Resid Onen ◽  
Ilhan Yilmaz ◽  
Mustafa Kilic ◽  
...  

2013 ◽  
Vol 2013 ◽  
pp. 1-7 ◽  
Author(s):  
Nikolaus Plesnila

Subarachnoid hemorrhage (SAH) is the subtype of stroke with one of the highest mortality rates and the least well-understood pathophysiologies. One of the very early events which may occur after SAH is a significant decrease of cerebral perfusion pressure (CPP) caused by the excessive increase of intracranial pressure during the initial bleeding. A severely decreased CPP results in global cerebral ischemia, an event also occurring after cardiac arrest. The aim of the current paper is to review the pathophysiological events occurring in experimental models of SAH and global cerebral ischemia and to evaluate the contribution and the importance of global cerebral ischemia for the pathophysiology of SAH.


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