Lack of Association Between Barometric Pressure and Incidence of Colic in Equine Academic Ambulatory Practice

2021 ◽  
Vol 97 ◽  
pp. 103342
Author(s):  
Justine Cianci ◽  
Ashley G. Boyle ◽  
Darko Stefanovski ◽  
Amy S. Biddle
2011 ◽  
Vol 62 (3) ◽  
pp. 355-359
Author(s):  
Kenji Noda ◽  
Satoru Kodama ◽  
Kanako Noda ◽  
Masashi Suzuki

2016 ◽  
Vol 18 (7) ◽  
pp. 37-42
Author(s):  
E.V. Kulchavenya ◽  
◽  
Keyword(s):  

2021 ◽  
Vol 10 (7) ◽  
pp. 437
Author(s):  
Hongxia Qi ◽  
Yunjia Wang ◽  
Jingxue Bi ◽  
Hongji Cao ◽  
Shenglei Xu

Floor positioning is an important aspect of indoor positioning technology, which is closely related to location-based services (LBSs). Currently, floor positioning technologies are mainly based on radio signals and barometric pressure. The former are impacted by the multipath effect, rely on infrastructure support, and are limited by different spatial structures. For the latter, the air pressure changes with the temperature and humidity, the deployment cost of the reference station is high, and different terminal models need to be calibrated in advance. In view of these issues, here, we propose a novel floor positioning method based on human activity recognition (HAR), using smartphone built-in sensor data to classify pedestrian activities. We obtain the degree of the floor change according to the activity category of every step and determine whether the pedestrian completes floor switching through condition and threshold analysis. Then, we combine the previous floor or the high-precision initial floor with the floor change degree to calculate the pedestrians’ real-time floor position. A multi-floor office building was chosen as the experimental site and verified through the process of alternating multiple types of activities. The results show that the pedestrian floor position change recognition and location accuracy of this method were as high as 100%, and that this method has good robustness and high universality. It is more stable than methods based on wireless signals. Compared with one existing HAR-based method and air pressure, the method in this paper allows pedestrians to undertake long-term static or round-trip activities during the process of going up and down the stairs. In addition, the proposed method has good fault tolerance for the misjudgment of pedestrian actions.


2010 ◽  
Vol 299 (5) ◽  
pp. H1439-H1450 ◽  
Author(s):  
Jennifer M. Hughes ◽  
Melissa A. Riddle ◽  
Michael L. Paffett ◽  
Laura V. Gonzalez Bosc ◽  
Benjimen R. Walker

The systemic vasculature exhibits attenuated vasoconstriction following hypobaric chronic hypoxia (CH) that is associated with endothelium-dependent vascular smooth muscle (VSM) cell hyperpolarization. We hypothesized that increased activity of endothelial cell (EC) large-conductance, calcium-activated potassium (BKCa) channels contributes to this response. Gracilis resistance arteries from hypobaric CH (barometric pressure = 380 mmHg for 48 h) rats demonstrated reduced myogenic reactivity and hyperpolarized VSM membrane potential ( Em) compared with controls under normoxic ex vivo conditions. These differences were eliminated by endothelial disruption. In the presence of cyclooxygenase and nitric oxide synthase inhibition, combined intraluminal administration of the intermediate and small-conductance, calcium-activated K+ channel blockers TRAM-34 and apamin was without effect on myogenic responsiveness and VSM Em in both groups; however, these variables were normalized in CH arteries by intraluminal administration of the BKCa inhibitor iberiotoxin (IBTX). Basal EC Em was hyperpolarized in arteries from CH rats compared with controls and was restored by IBTX, but not by TRAM-34/apamin. K+ channel blockers were without effect on EC basal Em in controls. Similarly, IBTX blocked acetylcholine-induced dilation in arteries from CH rats, but was without effect in controls, whereas TRAM-34/apamin eliminated dilation in controls. Acetylcholine-induced EC hyperpolarization and calcium responses were inhibited by IBTX in CH arteries and by TRAM-34/apamin in controls. Patch-clamp experiments on freshly isolated ECs demonstrated greater K+ current in cells from CH rats that was normalized by IBTX. IBTX was without effect on K+ current in controls. We conclude that hypobaric CH induces increased endothelial BKCa channel activity that contributes to reduced myogenic responsiveness and EC and VSM cell hyperpolarization.


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