scholarly journals Therapeutical Options in the Management of Carotid Dissection

2017 ◽  
Vol 66 (2) ◽  
pp. 675-676
Author(s):  
O. Martinelli ◽  
S. Venosi ◽  
J. BenHamida ◽  
A. Malaj ◽  
C. Belli ◽  
...  
2017 ◽  
Vol 41 ◽  
pp. 69-76 ◽  
Author(s):  
Ombretta Martinelli ◽  
Salvatore Venosi ◽  
Jamila BenHamida ◽  
Alban Malaj ◽  
Cristina Belli ◽  
...  

2021 ◽  
pp. 100353
Author(s):  
Walter R. Duarte-Celada ◽  
Dongkwan Jin ◽  
Gabriel Neves ◽  
Thomas Windisch

2003 ◽  
Vol 9 (3) ◽  
pp. 305-310 ◽  
Author(s):  
J. Sedat ◽  
M. Dib ◽  
J. Szapiro ◽  
P. Paquis

The stenting of carotid dissection has been described for the prevention of cerebral ischemia in patients who remain symptomatic despite therapeutic anticoagulation, in those who present contraindications for anticoagulation therapy, or who present a local or extensive stenosis, with an associated pseudoaneurysm. We here report a case associating a high clinical grade aneurysmal rupture with a bilateral extracranial carotid dissection. Because of the haemodynamic risk due to the acute bilateral stenosis-induced dissection and the occurrence of a vasospasm, the carotid dissections were treated with self-expendable stents.


2018 ◽  
Vol 40 ◽  
pp. 21-25 ◽  
Author(s):  
Viviana Manuel ◽  
Ryan Gouveia e Melo ◽  
Ruy Fernandes e Fernandes ◽  
Ana Rita Santos ◽  
Luís Silvestre ◽  
...  

2003 ◽  
Vol 113 (3) ◽  
pp. 537-540 ◽  
Author(s):  
Tiemo Wessels ◽  
R. Sparing ◽  
C. Neuschaefer-Rube ◽  
C. Kl??tzsch

2012 ◽  
Vol 2 (1) ◽  
pp. 20 ◽  
Author(s):  
Farah Adel ◽  
Jose M Ramia ◽  
Roberto De la Plaza ◽  
Jose Quilñones ◽  
Vladimir Arteaga ◽  
...  

Complications of dropped gallstones after laparoscopic cholecystectomy are infrequent but retroperitoneal abscess is extremely rare. We present a new case, discuss causes, diagnostic methods, preventive measures and therapeutical options.


2021 ◽  
Vol 12 ◽  
Author(s):  
Marie J. Pietrowski ◽  
Amr Ahmed Gabr ◽  
Stanislav Kozlov ◽  
David Blum ◽  
Annett Halle ◽  
...  

Purinergic signaling regulates neuronal and glial cell functions in the healthy CNS. In neurodegenerative diseases, purinergic signaling becomes dysregulated and can affect disease-associated phenotypes of glial cells. In this review, we discuss how cell-specific expression patterns of purinergic signaling components change in neurodegeneration and how dysregulated glial purinergic signaling and crosstalk may contribute to disease pathophysiology, thus bearing promising potential for the development of new therapeutical options for neurodegenerative diseases.


1998 ◽  
Vol 65 (5) ◽  
pp. 803a-803a
Author(s):  
R. E BARTT
Keyword(s):  

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