scholarly journals Research Note: Age-related effects of feeder space availability on welfare of broilers reared to 56 days of age Part 2: Blood physiological variables.

2022 ◽  
pp. 101698
Author(s):  
H.A. Olanrewaju ◽  
J.L. Purswell ◽  
S.D. Collier ◽  
S.L. Branton
2020 ◽  
Vol 34 (S1) ◽  
pp. 1-1
Author(s):  
Hammed A Olanrewaju ◽  
Joseph L Purswell ◽  
Stephanie D Collier ◽  
Scott L Branton

2021 ◽  
Vol 35 (S1) ◽  
Author(s):  
Hammed Olanrewaju ◽  
Purswell Joseph ◽  
Jeffery Evans ◽  
Stephanie Collier ◽  
Scott Branton

2021 ◽  
Vol 8 ◽  
Author(s):  
Eron G. Manusov ◽  
Carolina Gomez De Ziegler ◽  
Vincent P. Diego ◽  
Gerardo Munoz-Monaco ◽  
Sarah Williams-Blangero

Frailty is the age-related decline in well-being. The Frailty index (FI) measures the accumulation of health deficits and reflects biopsychosocial and cultural determinants of well-being. Frailty is measured as a static phenotype or as a Frailty Index comprising a ratio of suffered health deficits and total deficits. We report a Frailty Index calculated from routinely measured clinical variables gathered from residents of two Colonias (neighborhoods) in South Texas. A Colonia is a predominantly Hispanic, economically distressed, unincorporated neighborhood. We analyzed retrospective data from 894 patients that live in two Colonias located on the Texas-Mexico border. We calculated the FI with seven physiological variables, PHQ-9 score, and the 11 domain-specific Duke Profile scores, for a total of 19 possible health deficits. FI against age separately in males (n = 272) and females (n = 622) was regressed. Females had a significantly higher starting frailty, and males had a significantly greater change rate with age. FI against age for Cameron Park Colonia and Indian Hills Colonia was regressed. We calculated a significantly higher starting FI in Indian Hills and a significantly greater change rate in Cameron Park residents. Frailty's contributors are complex, especially in neighborhoods of poverty, immigration, low education level, and high prevalence of chronic disease. We report baseline Frailty Index data from two Colonias in South Texas and the clinical and research implications.


Author(s):  
Jasmina Pluncević Gligoroska ◽  
Maja Jordanova ◽  
Sanja Mančevska ◽  
Elisaveta Tomova

Physical activity (PA) is stimulatory physiologic stress for the human body and regular PA induces significant changes in many physiological, biochemical and anthropological parameters. The aim of this study is to determine the values and the differences among the cardio-physiological, hematological and anthropological parameters for different age and gender subgroups in healthy physically active children and an adolescent population. An anthropometric evaluation (Matiegka protocol), hematological analysis i.e., red blood cells count (RBC), hemoglobin level, hematocrit, and ergometrical testing (Brus protocol) were made. Regarding mass and height until the age of 12 (U12), girls were heavier and taller than boys. The mean values for relative muscle mass in all the groups of boys and girls of different ages are in the optimal range (>50%) and indicate well developed muscle mass. There are no differences between the same age subgroups for this anthropometric parameter between the boys and girls. All the subgroups of girls of different ages showed higher body fat percent than their male siblings. Among the girls, RBC variables did not show a difference in the age subgroups. Regarding gender, all RBC variables were significantly higher among the male groups, except the U12. Cardio-physiological parameters of heart frequency at rest, exercise time duration and maximal oxygen consumption (VO2max), were significantly higher among the boys. In all age-based male subgroups VO2max was higher among the older boys. Conclusion: This study has shown age related changes in anthropological, hematological and ergometrical parameters in a male and female young active population. The girls showed significantly lower levels of cardio-physiological fitness which can be contributed either to gender and lower volume of PA.


Author(s):  
W. Krebs ◽  
I. Krebs

Various inclusion bodies occur in vertebrate retinal photoreceptor cells. Most of them are membrane bound and associated with phagocytosis or they are age related residual bodies. We found an additional inclusion body in foveal cone cells of the baboon (Papio anubis) retina.The eyes of a 15 year old baboon were fixed by immersion in cacodylate buffered glutaraldehyde (2%)/formaldehyde (2%) as described in detail elsewhere . Pieces of retina from various locations, including the fovea, were embedded in epoxy resin such that radial or tangential sections could be cut.Spindle shaped inclusion bodies were found in the cytoplasm of only foveal cones. They were abundant in the inner segments, close to the external limiting membrane (Fig. 1). But they also occurred in the outer fibers, the perikarya, and the inner fibers (Henle’s fibers) of the cone cells. The bodies were between 0.5 and 2 μm long. Their central diameter was 0.2 to 0. 3 μm. They always were oriented parallel to the long axis of the cone cells. In longitudinal sections (Figs. 2,3) they seemed to have a fibrous skeleton that, in cross sections, turned out to consist of plate-like (Fig.4) and tubular profiles (Fig. 5).


2013 ◽  
Vol 55 ◽  
pp. 119-131 ◽  
Author(s):  
Bernadette Carroll ◽  
Graeme Hewitt ◽  
Viktor I. Korolchuk

Autophagy is a process of lysosome-dependent intracellular degradation that participates in the liberation of resources including amino acids and energy to maintain homoeostasis. Autophagy is particularly important in stress conditions such as nutrient starvation and any perturbation in the ability of the cell to activate or regulate autophagy can lead to cellular dysfunction and disease. An area of intense research interest is the role and indeed the fate of autophagy during cellular and organismal ageing. Age-related disorders are associated with increased cellular stress and assault including DNA damage, reduced energy availability, protein aggregation and accumulation of damaged organelles. A reduction in autophagy activity has been observed in a number of ageing models and its up-regulation via pharmacological and genetic methods can alleviate age-related pathologies. In particular, autophagy induction can enhance clearance of toxic intracellular waste associated with neurodegenerative diseases and has been comprehensively demonstrated to improve lifespan in yeast, worms, flies, rodents and primates. The situation, however, has been complicated by the identification that autophagy up-regulation can also occur during ageing. Indeed, in certain situations, reduced autophagosome induction may actually provide benefits to ageing cells. Future studies will undoubtedly improve our understanding of exactly how the multiple signals that are integrated to control appropriate autophagy activity change during ageing, what affect this has on autophagy and to what extent autophagy contributes to age-associated pathologies. Identification of mechanisms that influence a healthy lifespan is of economic, medical and social importance in our ‘ageing’ world.


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