Bone Disease in Renal Transplantation and Pleotropic Effects of Vitamin D Therapy

2010 ◽  
Vol 42 (7) ◽  
pp. 2518-2526 ◽  
Author(s):  
M.M. Sikgenc ◽  
S. Paydas ◽  
M. Balal ◽  
E. Demir ◽  
C. Kurt ◽  
...  
2013 ◽  
Author(s):  
Adodra Annika ◽  
Kouklinos Andreas ◽  
Julies Priscilla ◽  
Shaw Mathew ◽  
Jacobs Benjamin

2007 ◽  
Vol 23 (2) ◽  
pp. 450-458 ◽  
Author(s):  
U. Kunzendorf ◽  
B. K. Kramer ◽  
W. Arns ◽  
J. Braun ◽  
J. Grossmann ◽  
...  

Nephron ◽  
1978 ◽  
Vol 22 (1-3) ◽  
pp. 239-248 ◽  
Author(s):  
Michael N. Gottlieb ◽  
Michael K. Stephens ◽  
Edmund G. Lowrie ◽  
Harry J. Griffiths ◽  
John Kenzora ◽  
...  

2002 ◽  
Vol 61 ◽  
pp. S143-S148 ◽  
Author(s):  
Eduardo Slatopolsky ◽  
Adriana Dusso ◽  
Alex J. Brown

Bone ◽  
1994 ◽  
Vol 15 (6) ◽  
pp. 727-728 ◽  
Author(s):  
J.A. Kanis ◽  
C. Cooper ◽  
R. Francis ◽  
N. Hamdy ◽  
P. Selby ◽  
...  

PEDIATRICS ◽  
1993 ◽  
Vol 92 (6) ◽  
pp. 862-864
Author(s):  
YASUSHI NAKO ◽  
NAOBUMI FUKUSHIMA ◽  
TAKESHI TOMOMASA ◽  
KANJI NAGASHIMA ◽  
TAKAYOSHI KUROUME

Hypervitaminosis D is one of the causes of severe hypercalcemia in children. Most cases of hypervitaminosis D during childhood are due to an excessive supplementation of vitamin D by physicians or parents.1,2 To prevent metabolic bone disease of prematurity (rickets of prematurity), formulas designed for premature infants ("premature formulas"), which contain more calcium and vitamin D than standard formulas, are given to premature infants in addition to human milk.1 In some cases, separate vitamin D products are also given to these infants, although requirements for vitamin D and calcium intake in the premature infant and the formerly premature infant have not been fully estimated.1


2008 ◽  
Vol 85 (12) ◽  
pp. 1755-1759 ◽  
Author(s):  
Didier Ducloux ◽  
Cécile Courivaud ◽  
Jamal Bamoulid ◽  
Amir Kazory ◽  
Gilles Dumoulin ◽  
...  

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